Latifa Koussih
- Immunology top 10%
- Physiology
- Molecular Biology
- Cellular and Molecular Neuroscience
- Immunology and Allergy top 10%
- Co-authors
- Abdelilah S. GounniJamila ChakirLianyu ShanAbdelhabib SemlaliÉric JacquesAndrew J. HalaykoHesam MovassaghJingbo Zhang
- Topics
- Axon Guidance and Neuronal Signaling (9 papers)Biomarkers in Disease Mechanisms (6 papers)Asthma and respiratory diseases (4 papers)
- Journals
- SHILAP Revista de lepidopterologíaThe Journal of ImmunologyPLoS ONE
- Partner nations
- CanadaUnited StatesFrance
In The Last Decade
Latifa Koussih
19 papers receiving 433 citations
Peers
Comparison fields: 5 of 64
- Immunology 230
- Physiology 171
- Molecular Biology 92
- Cellular and Molecular Neuroscience 72
- Immunology and Allergy 66
Countries citing papers authored by Latifa Koussih
This map shows the geographic impact of Latifa Koussih's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Latifa Koussih with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Latifa Koussih more than expected).
Fields of papers citing papers by Latifa Koussih
This network shows the impact of papers produced by Latifa Koussih. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Latifa Koussih. The network helps show where Latifa Koussih may publish in the future.
Co-authorship network of co-authors of Latifa Koussih
This figure shows the co-authorship network connecting the top 25 collaborators of Latifa Koussih. A scholar is included among the top collaborators of Latifa Koussih based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Latifa Koussih. Latifa Koussih is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 9 | |
| 4 | 10 | |
| 5 | 1 | |
| 6 | 9 | |
| 7 | 12 | |
| 8 | 6 | |
| 9 | 7 | |
| 10 | 17 | |
| 11 | 15 | |
| 12 | 18 | |
| 13 | 27 | |
| 14 | 25 | |
| 15 | 30 | |
| 16 | 35 | |
| 17 | 29 | |
| 18 | 117 | |
| 19 | 70 | |
| 20 | Rapid publication IL-9 expression by human eosinophils: Regulation by IL-1β and TNF-α | 1 |
About Latifa Koussih
Latifa Koussih is a scholar working on Immunology and Allergy, Cellular and Molecular Neuroscience and Immunology, having authored 20 papers that have together received 440 indexed citations. Recurring topics across this work include Axon Guidance and Neuronal Signaling (9 papers), Biomarkers in Disease Mechanisms (6 papers) and Asthma and respiratory diseases (4 papers). The work is most often cited by research in Immunology and Allergy (66 citations), Immunology (230 citations) and Physiology (171 citations). Latifa Koussih has collaborated with scholars based in Canada, United States and France. Frequent co-authors include Abdelilah S. Gounni, Jamila Chakir, Lianyu Shan, Abdelhabib Semlali, Éric Jacques, Andrew J. Halayko, Hesam Movassagh, Jingbo Zhang, Nicholas C. Nicolaides and Esra Nutku. Their work appears in journals such as SHILAP Revista de lepidopterología, The Journal of Immunology and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.