Lars D. Maerz

564 total citations
10 papers, 383 citations indexed

About

Lars D. Maerz is a scholar working on Molecular Biology, Genetics and Pathology and Forensic Medicine. According to data from OpenAlex, Lars D. Maerz has authored 10 papers receiving a total of 383 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Genetics and 2 papers in Pathology and Forensic Medicine. Recurrent topics in Lars D. Maerz's work include Genetic and Kidney Cyst Diseases (4 papers), Epigenetics and DNA Methylation (3 papers) and DNA Repair Mechanisms (2 papers). Lars D. Maerz is often cited by papers focused on Genetic and Kidney Cyst Diseases (4 papers), Epigenetics and DNA Methylation (3 papers) and DNA Repair Mechanisms (2 papers). Lars D. Maerz collaborates with scholars based in Germany, United Kingdom and United States. Lars D. Maerz's co-authors include Birgit Holzwarth, Stefanie Ruf, Annika Sonntag, Ralf Baumeister, Michael Reth, Sushma‐Nagaraja Grellscheid, Christopher Boehlke, Roland Nitschke, Kathrin Thedieck and Mirja Tamara Prentzell and has published in prestigious journals such as Cell, Nucleic Acids Research and Developmental Cell.

In The Last Decade

Lars D. Maerz

10 papers receiving 379 citations

Peers

Countries citing papers authored by Lars D. Maerz

Since Specialization

Citations

This map shows the geographic impact of Lars D. Maerz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lars D. Maerz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lars D. Maerz more than expected).

Fields of papers citing papers by Lars D. Maerz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lars D. Maerz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lars D. Maerz. The network helps show where Lars D. Maerz may publish in the future.

Co-authorship network of co-authors of Lars D. Maerz

This figure shows the co-authorship network connecting the top 25 collaborators of Lars D. Maerz. A scholar is included among the top collaborators of Lars D. Maerz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lars D. Maerz. Lars D. Maerz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Cilia defects upon loss of WDR4 are linked to proteasomal hyperactivity and ubiquitin shortage 2024 ·Cell Death and Disease ·Martin D. Burkhalter, Tom Stiff, Lars D. Maerz, (unknown), Heike Wiese, (unknown), (unknown), (unknown), (unknown), Cornelia Donow, Marius Costel Alupei, Sebastian Iben, Marco Groth, Sebastian Wiese, Joseph A. Church, Penny A. Jeggo, Melanie Philipp	1
2	Kinase Activity Is Not Required for G Protein–Coupled Receptor Kinase 4 Restraining mTOR Signaling during Cilia and Kidney Development 2023 ·Journal of the American Society of Nephrology ·(unknown), Lars D. Maerz, (unknown), Cornelia Donow, Barbara Moepps, Richard T. Premont, Martin D. Burkhalter, Carsten Hoffmann, Melanie Philipp	3
3	Analysis of cilia dysfunction phenotypes in zebrafish embryos depleted of Origin recognition complex factors 2019 ·European Journal of Human Genetics ·Lars D. Maerz, (unknown), (unknown), Cornelia Donow, Penelope A. Jeggo, Melanie Philipp	13
4	Pharmacological cholesterol depletion disturbs ciliogenesis and ciliary function in developing zebrafish 2019 ·Communications Biology ·Lars D. Maerz, Martin D. Burkhalter, (unknown), Oliver H. Wittekindt, Manfred Frick, Melanie Philipp	29
5	Loss of Peter Pan protein is associated with cell cycle defects and apoptotic events 2019 ·Biochimica et Biophysica Acta (BBA) - Molecular Cell Research ·(unknown), (unknown), (unknown), Lars D. Maerz, Melanie Philipp, Astrid S. Pfister	11
6	Resting cells rely on the DNA helicase component MCM2 to build cilia 2018 ·Nucleic Acids Research ·(unknown), Lars D. Maerz, Karol Szafranski, Marco Groth, Tamara J. Blätte, Cornelia Donow, (unknown), Paul Walther, Penelope A. Jeggo, Martin D. Burkhalter, Melanie Philipp	17
7	Xpg limits the expansion of haematopoietic stem and progenitor cells after ionising radiation 2016 ·Nucleic Acids Research ·(unknown), Anett Illing, F Becker, Lars D. Maerz, Yohei Morita, Melanie Philipp, Martin D. Burkhalter	9
8	TSC1 Activates TGF-β-Smad2/3 Signaling in Growth Arrest and Epithelial-to-Mesenchymal Transition 2015 ·Developmental Cell ·(unknown), Mirja Tamara Prentzell, Birgit Holzwarth, Kathrin Kläsener, Ineke Kuper, Christopher Boehlke, Annika Sonntag, Stefanie Ruf, Lars D. Maerz, Roland Nitschke, Sushma‐Nagaraja Grellscheid, Michael Reth, Gerd Walz, Ralf Baumeister, Elke Neumann‐Haefelin, Kathrin Thedieck	52
9	Inhibition of mTORC1 by Astrin and Stress Granules Prevents Apoptosis in Cancer Cells 2013 ·Cell ·Kathrin Thedieck, Birgit Holzwarth, Mirja Tamara Prentzell, Christopher Boehlke, Kathrin Kläsener, Stefanie Ruf, Annika Sonntag, Lars D. Maerz, Sushma‐Nagaraja Grellscheid, Elisabeth Kremmer, Roland Nitschke, E. Wolfgang Kuehn, Johan W. Jonker, Albert K. Groen, Michael Reth, Michael N. Hall, Ralf Baumeister	240
10	Inhibition of mTORC1 by Astrin and Stress Granules Prevents Apoptosis in Cancer Cells 2013 ·Cell ·Kathrin Thedieck, Birgit Holzwarth, Mirja Tamara Prentzell, Christopher Boehlke, Kathrin Kläsener, Stefanie Ruf, Annika Sonntag, Lars D. Maerz, Sushma‐Nagaraja Grellscheid, Elisabeth Kremmer, Roland Nitschke, E. Wolfgang Kuehn, Johan W. Jonker, Albert K. Groen, Michael Reth, Michael N. Hall, Ralf Baumeister	8

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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