Kyle Lewis

1.4k total citations · 1 hit paper
15 papers, 585 citations indexed

About

Kyle Lewis is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Kyle Lewis has authored 15 papers receiving a total of 585 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 4 papers in Cell Biology and 3 papers in Oncology. Recurrent topics in Kyle Lewis's work include Liver physiology and pathology (3 papers), Ubiquitin and proteasome pathways (3 papers) and Liver Disease Diagnosis and Treatment (2 papers). Kyle Lewis is often cited by papers focused on Liver physiology and pathology (3 papers), Ubiquitin and proteasome pathways (3 papers) and Liver Disease Diagnosis and Treatment (2 papers). Kyle Lewis collaborates with scholars based in United States, Canada and Japan. Kyle Lewis's co-authors include Takanori Takebe, Nikolai A. Timchenko, Lubov Timchenko, Polina Iakova, Masaki Kimura, Wendy L. Thompson, Momoko Yoshimoto, Mari Maezawa, Tadahiro Shinozawa and Shodai Togo and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and Molecular and Cellular Biology.

In The Last Decade

Kyle Lewis

15 papers receiving 573 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

High-Fidelity Drug-Induced Liver Injury Screen Using Human Pluripotent Stem Cell–Derived Organoids

2020 250 citations Tadahiro Shinozawa, Masaki Kimura et al. Gastroenterology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kyle Lewis United States	12	270	174	123	104	101	15	585
Xiujuan Chang China	13	209 0.8×	345 2.0×	217 1.8×	26 0.3×	132 1.3×	37	724
Veera Baladandayuthapani United States	13	316 1.2×	89 0.5×	46 0.4×	64 0.6×	160 1.6×	28	780
Chunping Wang China	14	245 0.9×	355 2.0×	211 1.7×	29 0.3×	168 1.7×	34	804
Masaru Koido Japan	12	226 0.8×	51 0.3×	52 0.4×	66 0.6×	46 0.5×	30	523
Huijie Jiang China	13	204 0.8×	132 0.8×	73 0.6×	85 0.8×	83 0.8×	60	636
Chee Leong Cheng Singapore	16	230 0.9×	227 1.3×	361 2.9×	32 0.3×	193 1.9×	36	1.0k
Marie Fukushima Japan	13	132 0.5×	191 1.1×	198 1.6×	19 0.2×	43 0.4×	22	565
Zhengyi Jiang China	11	161 0.6×	66 0.4×	107 0.9×	20 0.2×	42 0.4×	14	387
Joyeeta Dutta‐Moscato United States	8	168 0.6×	66 0.4×	84 0.7×	45 0.4×	26 0.3×	11	431

Countries citing papers authored by Kyle Lewis

Since Specialization

Citations

This map shows the geographic impact of Kyle Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kyle Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kyle Lewis more than expected).

Fields of papers citing papers by Kyle Lewis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kyle Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kyle Lewis. The network helps show where Kyle Lewis may publish in the future.

Co-authorship network of co-authors of Kyle Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of Kyle Lewis. A scholar is included among the top collaborators of Kyle Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kyle Lewis. Kyle Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Lewis, Kyle, Matthew G. Annis, Stephanie Totten, et al.. (2024). p66ShcA promotes malignant breast cancer phenotypes by alleviating energetic and oxidative stress. Redox Biology. 70. 103028–103028. 2 indexed citations

Lewis, Kyle, Momoko Yoshimoto, & Takanori Takebe. (2021). Fetal liver hematopoiesis: from development to delivery. Stem Cell Research & Therapy. 12(1). 139–139. 48 indexed citations

Shinozawa, Tadahiro, Masaki Kimura, Yuqi Cai, et al.. (2020). High-Fidelity Drug-Induced Liver Injury Screen Using Human Pluripotent Stem Cell–Derived Organoids. Gastroenterology. 160(3). 831–846.e10. 250 indexed citations breakdown →

Lewis, Kyle, Alexander Kiepas, Julien Senécal, et al.. (2020). p66ShcA functions as a contextual promoter of breast cancer metastasis. Breast Cancer Research. 22(1). 7–7. 12 indexed citations

Li, Yang & Kyle Lewis. (2020). Erythroid Lineage Cells in the Liver: Novel Immune Regulators and Beyond. Journal of Clinical and Translational Hepatology. 8(2). 177–183. 3 indexed citations

Jiang, Yanjun, Ashley Cast, Leila Valanejad, et al.. (2018). Gankyrin Promotes Tumor-Suppressor Protein Degradation to Drive Hepatocyte Proliferation. Cellular and Molecular Gastroenterology and Hepatology. 6(3). 239–255. 11 indexed citations

Lewis, Kyle, Leila Valanejad, Ashley Cast, et al.. (2017). RNA Binding Protein CUGBP1 Inhibits Liver Cancer in a Phosphorylation-Dependent Manner. Molecular and Cellular Biology. 37(16). 25 indexed citations

Valanejad, Leila, Kyle Lewis, Mary Wright, et al.. (2017). FXR-Gankyrin axis is involved in development of pediatric liver cancer. Carcinogenesis. 38(7). 738–747. 32 indexed citations

Jin, Jingling, Leila Valanejad, Kyle Lewis, et al.. (2016). Activation of CDK4 Triggers Development of Non-alcoholic Fatty Liver Disease. Cell Reports. 16(3). 744–756. 39 indexed citations

10.

Lewis, Kyle, Leila Valanejad, Polina Iakova, et al.. (2015). p300 Regulates Liver Functions by Controlling p53 and C/EBP Family Proteins through Multiple Signaling Pathways. Molecular and Cellular Biology. 35(17). 3005–3016. 19 indexed citations

11.

Timchenko, Nikolai A. & Kyle Lewis. (2014). Elimination of Tumor Suppressor Proteins during Liver Carcinogenesis. 1(1). 27–38. 9 indexed citations

12.

Jin, Jingling, Il‐Hwa Hong, Kyle Lewis, et al.. (2014). Cooperation of C/EBP family proteins and chromatin remodeling proteins is essential for termination of liver regeneration. Hepatology. 61(1). 315–325. 39 indexed citations

13.

Jin, Jingling, Polina Iakova, Yanjun Jiang, et al.. (2013). Transcriptional and Translational Regulation of C/EBPβ-HDAC1 Protein Complexes Controls Different Levels of p53, SIRT1, and PGC1α Proteins at the Early and Late Stages of Liver Cancer. Journal of Biological Chemistry. 288(20). 14451–14462. 37 indexed citations

14.

Hong, Il‐Hwa, Kyle Lewis, Polina Iakova, et al.. (2013). Age-associated Change of C/EBP Family Proteins Causes Severe Liver Injury and Acceleration of Liver Proliferation after CCl4 Treatments. Journal of Biological Chemistry. 289(2). 1106–1118. 29 indexed citations

15.

Helgason, Richard V., Jeffery L. Kennington, & Kyle Lewis. (2001). Cruise Missile Mission Planning: A Heuristic Algorithm for Automatic Path Generation. Journal of Heuristics. 7(5). 473–494. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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