Koji Kato

498 citations
16 papers · 346 indexed · h-index 8
Topics
Acute Lymphoblastic Leukemia research (3 papers)Peroxisome Proliferator-Activated Receptors (3 papers)Virus-based gene therapy research (2 papers)
Partner nations
Japan

In The Last Decade

Koji Kato

14 papers receiving 344 citations

Peers

Koji Kato
Comparison fields: 5 of 58
  • Molecular Biology 161
  • Neurology 115
  • Cancer Research 93
  • Genetics 75
  • Oncology 73
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Tony Brooks United Kingdom
Stacy Roberts United States
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Hassan Dastsooz Iran
Dieter Pullirsch Austria
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Sara Montagner Italy
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Citations per field
00.5×5.2×
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Citations per year

Countries citing papers authored by Koji Kato

Since Specialization
Citations

This map shows the geographic impact of Koji Kato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Koji Kato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Koji Kato more than expected).

Fields of papers citing papers by Koji Kato

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Koji Kato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Koji Kato. The network helps show where Koji Kato may publish in the future.

Co-authorship network of co-authors of Koji Kato

This figure shows the co-authorship network connecting the top 25 collaborators of Koji Kato. A scholar is included among the top collaborators of Koji Kato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Koji Kato. Koji Kato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
#WorkIndexed citations
1 0
2 0
3 1
4 18
5 2
6 1
7 45
8 40
9 5
10 60
11 31
12 96
13
Detection of minimal residual disease in patients with childhood common acute lymphoblastic leukemia after autologous bone marrow transplantation with ex vivo purging and systemic IL-2 infusion: unsuccessful prediction of subsequent relapse.
3
14
New E2A/PBX1 fusion transcript in a patient with t(1;19)(q23;p13) acute lymphoblastic leukemia.
18
15 22
16
Production and analysis of HH 10 monoclonal antibodies reactive to immature hematopoietic cells and their use for monitoring acute leukemia cells.
4

About Koji Kato

Koji Kato is a scholar working on Physiology, Clinical Biochemistry and Hematology, having authored 16 papers that have together received 346 indexed citations. Recurring topics across this work include Acute Lymphoblastic Leukemia research (3 papers), Peroxisome Proliferator-Activated Receptors (3 papers) and Virus-based gene therapy research (2 papers). The work is most often cited by research in Neurology (115 citations), Cancer Research (93 citations) and Hematology (49 citations). Koji Kato has collaborated with scholars based in Japan. Frequent co-authors include Keizo Horibe, Shin‐ichiro Numata, Yuji Miyajima, Kazuko Kudo, Kimikazu Matsumoto, Minoru Fukuda, Hiroshi Mori, S Numata, Shinobu Tsuzuki and Shoji Tsuji. Their work appears in journals such as Blood, Cancer and Carcinogenesis.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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