Kohei Omachi

682 total citations
24 papers, 359 citations indexed

About

Kohei Omachi is a scholar working on Molecular Biology, Immunology and Allergy and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Kohei Omachi has authored 24 papers receiving a total of 359 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Immunology and Allergy and 6 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Kohei Omachi's work include Cell Adhesion Molecules Research (11 papers), Renal and related cancers (5 papers) and Platelet Disorders and Treatments (4 papers). Kohei Omachi is often cited by papers focused on Cell Adhesion Molecules Research (11 papers), Renal and related cancers (5 papers) and Platelet Disorders and Treatments (4 papers). Kohei Omachi collaborates with scholars based in Japan, United States and Canada. Kohei Omachi's co-authors include Jeffrey H. Miner, Hirofumi Kai, Mary Ann Suico, Tsuyoshi Shuto, Yasuhiro Yoshimura, Yoshiharu Muto, Benjamin D. Humphreys, Ryosuke Fukuda, Tomoaki Koga and Saori Morino‐Koga and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Kohei Omachi

23 papers receiving 356 citations

Peers

Kohei Omachi
Kohei Omachi
Citations per year, relative to Kohei Omachi Kohei Omachi (= 1×) peers Ulrike Hopfer

Countries citing papers authored by Kohei Omachi

Since Specialization
Citations

This map shows the geographic impact of Kohei Omachi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kohei Omachi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kohei Omachi more than expected).

Fields of papers citing papers by Kohei Omachi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kohei Omachi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kohei Omachi. The network helps show where Kohei Omachi may publish in the future.

Co-authorship network of co-authors of Kohei Omachi

This figure shows the co-authorship network connecting the top 25 collaborators of Kohei Omachi. A scholar is included among the top collaborators of Kohei Omachi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kohei Omachi. Kohei Omachi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lin, Meei‐Hua, et al.. (2025). Mouse Alport podocytes are susceptible to AAV9 transduction in vivo. Kidney International. 109(1). 129–138.
2.
Omachi, Kohei, Damian Fermin, Felix Eichinger, et al.. (2024). Association of Genetically Predicted Skipping of COL4A4 Exon 27 with Hematuria and Albuminuria. Journal of the American Society of Nephrology. 36(1). 48–59. 2 indexed citations
3.
Yoshimura, Yasuhiro, Yoshiharu Muto, Nicolas Ledru, et al.. (2023). A single-cell multiomic analysis of kidney organoid differentiation. Proceedings of the National Academy of Sciences. 120(20). e2219699120–e2219699120. 24 indexed citations
4.
Yoshimura, Yasuhiro, Yoshiharu Muto, Kohei Omachi, Jeffrey H. Miner, & Benjamin D. Humphreys. (2023). Elucidating the Proximal Tubule HNF4A Gene Regulatory Network in Human Kidney Organoids. Journal of the American Society of Nephrology. 34(10). 1672–1686. 14 indexed citations
5.
Muto, Yoshiharu, Eryn E. Dixon, Yasuhiro Yoshimura, et al.. (2022). Defining cellular complexity in human autosomal dominant polycystic kidney disease by multimodal single cell analysis. Nature Communications. 13(1). 6497–6497. 52 indexed citations
6.
Tamai, Minori, Shinichi Fujisawa, Thao Nguyen, et al.. (2022). Creation of Philadelphia chromosome by CRISPR/Cas9-mediated double cleavages on BCR and ABL1 genes as a model for initial event in leukemogenesis. Cancer Gene Therapy. 30(1). 38–50. 2 indexed citations
7.
Omachi, Kohei, Hirofumi Kai, Michel Roberge, & Jeffrey H. Miner. (2022). NanoLuc reporters identify COL4A5 nonsense mutations susceptible to drug-induced stop codon readthrough. iScience. 25(3). 103891–103891. 11 indexed citations
8.
Omachi, Kohei & Jeffrey H. Miner. (2022). Comparative analysis of dCas9-VP64 variants and multiplexed guide RNAs mediating CRISPR activation. PLoS ONE. 17(6). e0270008–e0270008. 21 indexed citations
9.
Omachi, Kohei, C. O’Carroll, & Jeffrey H. Miner. (2022). PPARδ Agonism Ameliorates Renal Fibrosis in an Alport Syndrome Mouse Model. Kidney360. 4(3). 341–348. 2 indexed citations
10.
Omachi, Kohei, Sumio Ohtsuki, Shogo Misumi, et al.. (2021). Metformin ameliorates the severity of experimental Alport syndrome. Scientific Reports. 11(1). 7053–7053. 18 indexed citations
11.
Iwata, Hiromitsu, Tsuyoshi Shuto, Kohei Omachi, et al.. (2020). Combined effects of cisplatin and photon or proton irradiation in cultured cells: radiosensitization, patterns of cell death and cell cycle distribution. Journal of Radiation Research. 61(6). 832–841. 11 indexed citations
12.
Yamamura, Tomohiko, Kohei Omachi, Mary Ann Suico, et al.. (2020). Trimerization and Genotype–Phenotype Correlation of COL4A5 Mutants in Alport Syndrome. Kidney International Reports. 5(5). 718–726. 11 indexed citations
13.
Suico, Mary Ann, Kohei Omachi, Tatsuya Kondo, et al.. (2020). Mild electrical stimulation with heat shock attenuates renal pathology in adriamycin-induced nephrotic syndrome mouse model. Scientific Reports. 10(1). 18719–18719. 4 indexed citations
14.
Omachi, Kohei & Jeffrey H. Miner. (2019). Alport Syndrome Therapeutics: Ready for Prime-Time Players. Trends in Pharmacological Sciences. 40(11). 803–806. 12 indexed citations
15.
Omachi, Kohei, et al.. (2018). A Split-Luciferase-Based Trimer Formation Assay as a High-throughput Screening Platform for Therapeutics in Alport Syndrome. Cell chemical biology. 25(5). 634–643.e4. 21 indexed citations
16.
Omachi, Kohei, et al.. (2017). Bromide supplementation exacerbated the renal dysfunction, injury and fibrosis in a mouse model of Alport syndrome. PLoS ONE. 12(9). e0183959–e0183959. 4 indexed citations
17.
Omachi, Kohei, et al.. (2017). Long-term treatment with EGFR inhibitor erlotinib attenuates renal inflammatory cytokines but not nephropathy in Alport syndrome mouse model. Clinical and Experimental Nephrology. 21(6). 952–960. 8 indexed citations
18.
Fukuda, Ryosuke, Mary Ann Suico, Kohei Omachi, et al.. (2015). Podocyte p53 Limits the Severity of Experimental Alport Syndrome. Journal of the American Society of Nephrology. 27(1). 144–157. 23 indexed citations
19.
Koga, Tomoaki, Mary Ann Suico, Eriko Watanabe, et al.. (2015). Endoplasmic Reticulum (ER) Stress Induces Sirtuin 1 (SIRT1) Expression via the PI3K-Akt-GSK3β Signaling Pathway and Promotes Hepatocellular Injury. Journal of Biological Chemistry. 290(51). 30366–30374. 73 indexed citations
20.
Fukuda, Ryosuke, Mary Ann Suico, Kohei Omachi, et al.. (2013). Mild Electrical Stimulation at 0.1-ms Pulse Width Induces p53 Protein Phosphorylation and G2 Arrest in Human Epithelial Cells. Journal of Biological Chemistry. 288(22). 16117–16126. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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