Ko Sato

693 total citations
12 papers, 518 citations indexed

About

Ko Sato is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Ko Sato has authored 12 papers receiving a total of 518 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Oncology and 4 papers in Genetics. Recurrent topics in Ko Sato's work include DNA Repair Mechanisms (3 papers), BRCA gene mutations in cancer (2 papers) and Microtubule and mitosis dynamics (2 papers). Ko Sato is often cited by papers focused on DNA Repair Mechanisms (3 papers), BRCA gene mutations in cancer (2 papers) and Microtubule and mitosis dynamics (2 papers). Ko Sato collaborates with scholars based in Japan, United Kingdom and Spain. Ko Sato's co-authors include Tomohiko Ohta, Wenwen Wu, Hiroyuki Nishikawa, Mamoru Fukuda, Ryosuke Hayami, Toru Nishikawa, Hirotaka Koizumi, Ashok R. Venkitaraman, Ayaka Koike and Haruki Ogata and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Cancer Research.

In The Last Decade

Ko Sato

11 papers receiving 513 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ko Sato Japan	8	427	157	102	59	54	12	518
Stephanie K. Guest United Kingdom	7	323 0.8×	168 1.1×	44 0.4×	59 1.0×	65 1.2×	7	429
Christine von Klitzing Germany	6	244 0.6×	116 0.7×	40 0.4×	36 0.6×	55 1.0×	7	340
Alexander C. Jordan United States	7	307 0.7×	134 0.9×	38 0.4×	87 1.5×	36 0.7×	10	453
Timothy J. Stanek United States	10	458 1.1×	217 1.4×	34 0.3×	93 1.6×	25 0.5×	13	545
Zhi-Ren Liu United States	9	516 1.2×	159 1.0×	31 0.3×	96 1.6×	32 0.6×	9	641
Neele Drobnitzky United Kingdom	6	421 1.0×	168 1.1×	23 0.2×	58 1.0×	26 0.5×	6	497
Shaoli Lu China	7	522 1.2×	151 1.0×	45 0.4×	50 0.8×	33 0.6×	7	607
Susanne Thieme Germany	5	272 0.6×	105 0.7×	62 0.6×	62 1.1×	37 0.7×	6	363
Silvia Licciulli Italy	8	366 0.9×	127 0.8×	20 0.2×	83 1.4×	60 1.1×	8	470

Countries citing papers authored by Ko Sato

Since Specialization

Citations

This map shows the geographic impact of Ko Sato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ko Sato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ko Sato more than expected).

Fields of papers citing papers by Ko Sato

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ko Sato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ko Sato. The network helps show where Ko Sato may publish in the future.

Co-authorship network of co-authors of Ko Sato

This figure shows the co-authorship network connecting the top 25 collaborators of Ko Sato. A scholar is included among the top collaborators of Ko Sato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ko Sato. Ko Sato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Tanaka, Susumu, Koichiro Nozaki, Satoshi Watanabe, et al.. (2025). Risk of lung injury with immune checkpoint inhibitors after talc pleurodesis: A retrospective study. Lung Cancer. 204. 108590–108590.

Maeda, Ichiro, Miki Yoshiike, Wataru Usuba, et al.. (2021). A new bioinformatics approach identifies overexpression of GRB2 as a poor prognostic biomarker for prostate cancer. Scientific Reports. 11(1). 5696–5696. 5 indexed citations

Okamoto, Naoki, Toshio Kumai, Eri Sato, et al.. (2020). NF‐kB signaling in cardiomyocytes is inhibited by sevoflurane and promoted by propofol. FEBS Open Bio. 10(2). 259–267. 6 indexed citations

Oda, Keiko, Nobuyuki Yanagisawa, Ichiro Maeda, et al.. (2017). A bioinformatics-to-clinic sequential approach to analysis of prostate cancer biomarkers using TCGA datasets and clinical samples: a new method for precision oncology?. Oncotarget. 8(59). 99601–99611. 5 indexed citations

Sato, Ko, Satoshi Watanabe, Satoshi Shoji, et al.. (2016). Nephrotoxicity of cisplatin combination chemotherapy in thoracic malignancy patients with CKD risk factors. BMC Cancer. 16(1). 222–222. 43 indexed citations

Nagasawa, Satoi, Yuko Nakagawa, Ichiro Maeda, et al.. (2015). LSD1 Overexpression Is Associated with Poor Prognosis in Basal-Like Breast Cancer, and Sensitivity to PARP Inhibition. PLoS ONE. 10(2). e0118002–e0118002. 73 indexed citations

Nakagawa, Yuko, Naoki Okamoto, Satoi Nagasawa, et al.. (2015). NF-κB signaling mediates acquired resistance after PARP inhibition. Oncotarget. 6(6). 3825–3839. 34 indexed citations

Izawa, Naoki, Wenwen Wu, Ko Sato, et al.. (2011). HERC2 Interacts with Claspin and Regulates DNA Origin Firing and Replication Fork Progression. Cancer Research. 71(17). 5621–5625. 39 indexed citations

Wu, Wenwen, Ko Sato, Ayaka Koike, et al.. (2010). HERC2 Is an E3 Ligase That Targets BRCA1 for Degradation. Cancer Research. 70(15). 6384–6392. 118 indexed citations

10.

Sato, Ko, Eeson Rajendra, & Tomohiko Ohta. (2008). The UPS: a promising target for breast cancer treatment. BMC Biochemistry. 9(S1). S2–S2. 22 indexed citations

11.

Hayami, Ryosuke, Ko Sato, Wenwen Wu, et al.. (2005). Down-regulation of BRCA1-BARD1 Ubiquitin Ligase by CDK2. Cancer Research. 65(1). 6–10. 53 indexed citations

12.

Sato, Ko, Ryosuke Hayami, Wenwen Wu, et al.. (2004). Nucleophosmin/B23 Is a Candidate Substrate for the BRCA1-BARD1 Ubiquitin Ligase. Journal of Biological Chemistry. 279(30). 30919–30922. 120 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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