Klazien S. Bosch

554 total citations
8 papers, 456 citations indexed

About

Klazien S. Bosch is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Klazien S. Bosch has authored 8 papers receiving a total of 456 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 2 papers in Genetics and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Klazien S. Bosch's work include Genetic Neurodegenerative Diseases (2 papers), Mitochondrial Function and Pathology (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Klazien S. Bosch is often cited by papers focused on Genetic Neurodegenerative Diseases (2 papers), Mitochondrial Function and Pathology (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Klazien S. Bosch collaborates with scholars based in Netherlands, Italy and Canada. Klazien S. Bosch's co-authors include Ard Jonker, Cornelis J.F. Van Noorden, Wikky Tigchelaar, Nadia A. Atai, Dirk Troost, Fonnet E. Bleeker, W. Peter Vandertop, Simona Lamba, Alberto Bardelli and Eric A. Reits and has published in prestigious journals such as PLoS ONE, Journal of Cell Science and Acta Neuropathologica.

In The Last Decade

Klazien S. Bosch

8 papers receiving 454 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Klazien S. Bosch Netherlands 7 224 213 174 57 57 8 456
Guangrong Lu United States 10 215 1.0× 195 0.9× 120 0.7× 91 1.6× 29 0.5× 21 487
Ondřej Kalita Czechia 12 347 1.5× 190 0.9× 215 1.2× 123 2.2× 36 0.6× 31 595
Lorella Valletta Italy 8 254 1.1× 251 1.2× 99 0.6× 48 0.8× 43 0.8× 9 460
Sebastian Barbus Germany 12 396 1.8× 193 0.9× 262 1.5× 162 2.8× 58 1.0× 16 818
Nico V. Henriquez United Kingdom 8 247 1.1× 111 0.5× 98 0.6× 201 3.5× 68 1.2× 9 491
Eduard B. Dinca United Kingdom 11 251 1.1× 284 1.3× 99 0.6× 111 1.9× 51 0.9× 17 658
Marcel van der Weiden Netherlands 14 252 1.1× 157 0.7× 120 0.7× 133 2.3× 161 2.8× 25 625
Carlos Clara Brazil 14 348 1.6× 274 1.3× 181 1.0× 156 2.7× 42 0.7× 38 619
Huarui Zheng China 8 232 1.0× 117 0.5× 104 0.6× 125 2.2× 29 0.5× 17 501
Nicolas Goffart Belgium 9 141 0.6× 187 0.9× 63 0.4× 100 1.8× 63 1.1× 12 345

Countries citing papers authored by Klazien S. Bosch

Since Specialization
Citations

This map shows the geographic impact of Klazien S. Bosch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klazien S. Bosch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klazien S. Bosch more than expected).

Fields of papers citing papers by Klazien S. Bosch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Klazien S. Bosch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klazien S. Bosch. The network helps show where Klazien S. Bosch may publish in the future.

Co-authorship network of co-authors of Klazien S. Bosch

This figure shows the co-authorship network connecting the top 25 collaborators of Klazien S. Bosch. A scholar is included among the top collaborators of Klazien S. Bosch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Klazien S. Bosch. Klazien S. Bosch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Geest, R. J. van, Jan Willem Leeuwis, Amélie Dendooven, et al.. (2013). Connective Tissue Growth Factor Is Involved in Structural Retinal Vascular Changes in Long-Term Experimental Diabetes. Journal of Histochemistry & Cytochemistry. 62(2). 109–118. 16 indexed citations
2.
Atai, Nadia A., Wikky Tigchelaar, Klazien S. Bosch, et al.. (2011). Differential Activity of NADPH-Producing Dehydrogenases Renders Rodents Unsuitable Models to Study IDH1R132 Mutation Effects in Human Glioblastoma. Journal of Histochemistry & Cytochemistry. 59(5). 489–503. 26 indexed citations
3.
Atai, Nadia A., Manju Bansal, Wikky Tigchelaar, et al.. (2010). Osteopontin is up-regulated and associated with neutrophil and macrophage infiltration in glioblastoma. Immunology. 132(1). 39–48. 81 indexed citations
4.
Bleeker, Fonnet E., Nadia A. Atai, Simona Lamba, et al.. (2010). The prognostic IDH1 R132 mutation is associated with reduced NADP+-dependent IDH activity in glioblastoma. Acta Neuropathologica. 119(4). 487–494. 247 indexed citations
5.
Raspe, Marcel, et al.. (2009). Mimicking proteasomal release of polyglutamine peptides initiates aggregation and toxicity. Journal of Cell Science. 122(18). 3262–3271. 45 indexed citations
6.
Krawczyk, Przemek M., et al.. (2008). Polyglutamine Expansion Accelerates the Dynamics of Ataxin-1 and Does Not Result in Aggregate Formation. PLoS ONE. 3(1). e1503–e1503. 13 indexed citations
7.
Boren, Joan, Antonio Ramos‐Montoya, Klazien S. Bosch, et al.. (2005). In Situ Localization of Transketolase Activity in Epithelial Cells of Different Rat Tissues and Subcellularly in Liver Parenchymal Cells. Journal of Histochemistry & Cytochemistry. 54(2). 191–199. 23 indexed citations
8.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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