Klaus Starke
- Molecular Biology
- Physiology top 1%
- Cellular and Molecular Neuroscience top 10%
- Endocrine and Autonomic Systems top 5%
- Physiology
- Co-authors
- Ralph BültmannIvar von KügelgenBernd DriessenKatharina KurzAnne‐Ulrike TrendelenburgGerhard J. MolderingsFlorin TulucGeorg Hansmann
- Topics
- Adenosine and Purinergic Signaling (10 papers)Ion channel regulation and function (5 papers)Receptor Mechanisms and Signaling (5 papers)
- Journals
- British Journal of PharmacologyNaunyn-Schmiedeberg s Archives of PharmacologyJournal of Cardiovascular Pharmacology
- Partner nations
- Germany
In The Last Decade
Klaus Starke
18 papers receiving 528 citations
Peers
Comparison fields: 5 of 58
- Molecular Biology 337
- Physiology 312
- Cellular and Molecular Neuroscience 201
- Endocrine and Autonomic Systems 115
- Physiology 115
Countries citing papers authored by Klaus Starke
This map shows the geographic impact of Klaus Starke's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus Starke with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus Starke more than expected).
Fields of papers citing papers by Klaus Starke
This network shows the impact of papers produced by Klaus Starke. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus Starke. The network helps show where Klaus Starke may publish in the future.
Co-authorship network of co-authors of Klaus Starke
This figure shows the co-authorship network connecting the top 25 collaborators of Klaus Starke. A scholar is included among the top collaborators of Klaus Starke based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Klaus Starke. Klaus Starke is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 60 | |
| 3 | 31 | |
| 4 | 20 | |
| 5 | 28 | |
| 6 | 27 | |
| 7 | 46 | |
| 8 | 17 | |
| 9 | 43 | |
| 10 | 21 | |
| 11 | 64 | |
| 12 | 53 | |
| 13 | 10 | |
| 14 | 11 | |
| 15 | 36 | |
| 16 | 9 | |
| 17 | 50 | |
| 18 | 9 |
About Klaus Starke
Klaus Starke is a scholar working on Physiology, Endocrine and Autonomic Systems and Cellular and Molecular Neuroscience, having authored 18 papers that have together received 548 indexed citations. Recurring topics across this work include Adenosine and Purinergic Signaling (10 papers), Ion channel regulation and function (5 papers) and Receptor Mechanisms and Signaling (5 papers). The work is most often cited by research in Physiology (312 citations), Endocrine and Autonomic Systems (115 citations) and Cellular and Molecular Neuroscience (201 citations). Klaus Starke has collaborated with scholars based in Germany. Frequent co-authors include Ralph Bültmann, Ivar von Kügelgen, Bernd Driessen, Katharina Kurz, Anne‐Ulrike Trendelenburg, Gerhard J. Molderings, Florin Tuluc, Georg Hansmann, N. Limberger and Béla Szabó. Their work appears in journals such as British Journal of Pharmacology, Naunyn-Schmiedeberg s Archives of Pharmacology and Journal of Cardiovascular Pharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.