Klaus Olek

1.5k total citations
20 papers, 1.0k citations indexed

About

Klaus Olek is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Klaus Olek has authored 20 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Hematology and 6 papers in Genetics. Recurrent topics in Klaus Olek's work include Hemophilia Treatment and Research (6 papers), Cancer-related gene regulation (6 papers) and Platelet Disorders and Treatments (3 papers). Klaus Olek is often cited by papers focused on Hemophilia Treatment and Research (6 papers), Cancer-related gene regulation (6 papers) and Platelet Disorders and Treatments (3 papers). Klaus Olek collaborates with scholars based in Germany, United Kingdom and United States. Klaus Olek's co-authors include Michael Ludwig, Michael A. Levine, Peter Forster, Colin Renfrew, Matthew E. Hurles, Thomas Jansen, Johannes Oldenburg, Kathryn M. Stowell, Piet De Vos and George G. Brownlee and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and SHILAP Revista de lepidopterología.

In The Last Decade

Klaus Olek

19 papers receiving 961 citations

Peers

Klaus Olek

MB

GENET

HEMAT

CMN

GENET

R T Bronson United States

Heidi Anderson United States

An Langeveld Netherlands

Andrew Nguyen United States

Peter van Tuinen United States

J. Lesley Brown United States

Katja Langenfeld Germany

Pierre Fouchet France

Panayiotis A. Ioannou Australia

Paul E. Mead United States

R T Bronson United States

Klaus Olek

1.2k ×2.4

MB

242 ×0.6

GENET

40 ×0.1

HEMAT

55 ×0.6

CMN

47 ×0.8

GENET

Citations per year, relative to Klaus Olek Klaus Olek (= 1×) peers R T Bronson

Countries citing papers authored by Klaus Olek

Since Specialization

Citations

This map shows the geographic impact of Klaus Olek's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus Olek with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus Olek more than expected).

Fields of papers citing papers by Klaus Olek

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Klaus Olek. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus Olek. The network helps show where Klaus Olek may publish in the future.

Co-authorship network of co-authors of Klaus Olek

This figure shows the co-authorship network connecting the top 25 collaborators of Klaus Olek. A scholar is included among the top collaborators of Klaus Olek based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Klaus Olek. Klaus Olek is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Hüettel, Bruno, Richard Jäger, Dominik Fischer, et al.. (2025). FOGS: A SNPSTR Marker Database to Combat Wildlife Trafficking and a Cell Culture Bank for Ex‐Situ Conservation. Molecular Ecology Resources. 25(4). e14062–e14062. 1 indexed citations

2.

Misof, Bernhard, et al.. (2024). Joint detection of microsatellites and flanking sequences – SNPSTR markers for Athene noctua to fight illegal wildlife trade. SHILAP Revista de lepidopterología. 5. 100084–100084. 2 indexed citations

3.

Hüettel, Bruno, Richard Reinhardt, Rolf Fimmers, et al.. (2016). Methylation Markers for the Identification of Body Fluids and Tissues from Forensic Trace Evidence. PLoS ONE. 11(2). e0147973–e0147973. 65 indexed citations

4.

Pereira, Joana B., et al.. (2011). MtDNA typing of single-sperm cells isolated by micromanipulation. Forensic Science International Genetics. 6(2). 228–235. 9 indexed citations

5.

Schneider, Peter M., et al.. (2006). Examination of Postmortem Animal Interference to Human Remains Using Cross-Species Multiplex PCR. Forensic Science Medicine and Pathology. 2(2). 95–101. 13 indexed citations

6.

Olek, Klaus. (2006). Maturity-onset diabetes of the young: an update.. PubMed. 52(11-12). 593–8. 8 indexed citations

7.

Jansen, Thomas, Peter Forster, Michael A. Levine, et al.. (2002). Mitochondrial DNA and the origins of the domestic horse. Proceedings of the National Academy of Sciences. 99(16). 10905–10910. 271 indexed citations

8.

Oldenburg, Johannes, Jean‐Maurice Lavergne, Michael Ludwig, et al.. (2001). Somatic Mosaicism in Hemophilia A: A Fairly Common Event. The American Journal of Human Genetics. 69(1). 75–87. 97 indexed citations

9.

Oldenburg, Johannes, Simone Rost, Osman El‐Maarri, et al.. (2000). De novo factor VIII gene intron 22 inversion in a female carrier presents as a somatic mosaicism. Blood. 96(8). 2905–2906. 28 indexed citations

10.

Oldenburg, Johannes, Simone Rost, Osman El‐Maarri, et al.. (2000). De novo factor VIII gene intron 22 inversion in a female carrier presents as a somatic mosaicism. Blood. 96(8). 2905–2906. 30 indexed citations

11.

El‐Maarri, Osman, Alexander Olek, Başak Balaban, et al.. (1998). Methylation Levels at Selected CpG Sites in the Factor VIII and FGFR3 Genes, in Mature Female and Male Germ Cells: Implications for Male-Driven Evolution. The American Journal of Human Genetics. 63(4). 1001–1008. 32 indexed citations

12.

Schneppenheim, Reinhard, S. Krey, Frauke Bergmann, et al.. (1994). Genetic heterogeneity of severe von Willebrand disease type III in the German population. Human Genetics. 94(6). 640–52. 77 indexed citations

13.

Faber, J.-P., Wolfgang Poller, Klaus Olek, et al.. (1993). The molecular basis of α1-antichymotrypsin deficiency in a heterozygote with liver and lung disease. Journal of Hepatology. 18(3). 313–321. 77 indexed citations

14.

Knorr, D, et al.. (1992). Clinical heterogeneity of 21-hydroxylase deficiency of sibs with identical 21-hydroxylase genes. European Journal of Endocrinology. 126(1). 7–9. 8 indexed citations

15.

Crossley, Merlin, Michael Ludwig, Kathryn M. Stowell, et al.. (1992). Recovery from Hemophilia B Leyden: An Androgen-Responsive Element in the Factor IX Promoter. Science. 257(5068). 377–379. 106 indexed citations

16.

Gal, Andreas, A. Artlich, Michael Ludwig, et al.. (1991). Pro-347-Arg mutation of the rhodopsin gene in autosomal dominant retinitis pigmentosa. Genomics. 11(2). 468–470. 51 indexed citations

17.

Driesel, A.J., et al.. (1990). Regional assignment of 41 human DNA fragments on chromosome 7 by means of a somatic cell hybrid panel. Human Genetics. 84(2). 147–50. 8 indexed citations

18.

Higuchi, Miyoko, Corinne Wong, Klaus Olek, et al.. (1990). Characterization of mutations in the factor VIII gene by direct sequencing of amplified genomic DNA. Genomics. 6(1). 65–71. 65 indexed citations

19.

Fujita, Ricardo, P Trouillas, Abdoulaye Seck, et al.. (1989). Confirmation of linkage of Friedreich ataxia to chromosome 9 and identification of a new closely linked marker. Genomics. 4(1). 110–111. 56 indexed citations

20.

Gundermann, Karl‐Josef, et al.. (1983). Comparison of a Charcoal Sorbent Fiber with Commercial Charcoals for Hemoperfusion. Artificial Organs. 7(2). 186–196. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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