Klaus Hempel

1.0k total citations
64 papers, 827 citations indexed

About

Klaus Hempel is a scholar working on Molecular Biology, Biochemistry and Oncology. According to data from OpenAlex, Klaus Hempel has authored 64 papers receiving a total of 827 indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 10 papers in Biochemistry and 8 papers in Oncology. Recurrent topics in Klaus Hempel's work include Amino Acid Enzymes and Metabolism (9 papers), Chemical Reactions and Isotopes (7 papers) and Chemical Synthesis and Analysis (6 papers). Klaus Hempel is often cited by papers focused on Amino Acid Enzymes and Metabolism (9 papers), Chemical Reactions and Isotopes (7 papers) and Chemical Synthesis and Analysis (6 papers). Klaus Hempel collaborates with scholars based in Germany, United States and Sweden. Klaus Hempel's co-authors include Katja Heinze, Leonhard Birkofer, R. Lorenz, Aaron Breivogel, Günter Thomas, W. Maurer, B. Antonioli, Dina H. Triyoso, Fabian M. Koehler and Brigitte Schultze and has published in prestigious journals such as Environmental Health Perspectives, Chemistry - A European Journal and European Journal of Biochemistry.

In The Last Decade

Klaus Hempel

63 papers receiving 771 citations

Peers

Klaus Hempel
W. R. Cherry United States
Wen‐Ji Dong United States
A. Hardy United Kingdom
James E. Whitaker United States
W. R. Cherry United States
Klaus Hempel
Citations per year, relative to Klaus Hempel Klaus Hempel (= 1×) peers W. R. Cherry

Countries citing papers authored by Klaus Hempel

Since Specialization
Citations

This map shows the geographic impact of Klaus Hempel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Klaus Hempel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Klaus Hempel more than expected).

Fields of papers citing papers by Klaus Hempel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Klaus Hempel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Klaus Hempel. The network helps show where Klaus Hempel may publish in the future.

Co-authorship network of co-authors of Klaus Hempel

This figure shows the co-authorship network connecting the top 25 collaborators of Klaus Hempel. A scholar is included among the top collaborators of Klaus Hempel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Klaus Hempel. Klaus Hempel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Heinze, Katja & Klaus Hempel. (2009). Solid‐Phase Synthesis of Peptide Libraries Combining α‐Amino Acids with Inorganic and Organic Chromophores. Chemistry - A European Journal. 15(6). 1346–1358. 42 indexed citations
2.
Heinze, Katja, et al.. (2008). Solid‐Phase Synthesis of Transition‐Metal Complexes. Chemistry - A European Journal. 14(31). 9468–9480. 36 indexed citations
3.
Grawé, Jan, Johannes Biko, R. Lorenz, et al.. (2005). Evaluation of the reticulocyte micronucleus assay in patients treated with radioiodine for thyroid cancer. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 583(1). 12–25. 36 indexed citations
4.
Biko, Johannes, et al.. (2004). Dose response for T-cell receptor (TCR) mutants in patients repeatedly treated with for thyroid cancer. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 548(1-2). 27–33. 7 indexed citations
5.
Hempel, Klaus, et al.. (2003). High gradient magnetic cell sorting and internal standardisation substantially improve the assay for somatic mutations at the glycophorin A (GPA) locus. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 525(1-2). 29–42. 2 indexed citations
6.
Bassukas, Ioannis D., et al.. (1996). Age dependent selection against HPRT deficient T lymphocytes in the HPRT± heterozygous mouse. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 351(1). 67–77. 24 indexed citations
7.
Wixler, Viktor, et al.. (1994). Isolation and quantification of class 1 MHC gene mutants in mouse T cells by immunoselection with a magnetic cell sorter (MACS). Journal of Immunological Methods. 171(1). 121–130. 5 indexed citations
8.
Lorenz, R., et al.. (1993). Normal and reverse dose-rate effect for the induction of mutants in somatic cells by ionizing radiation. Toxicology Letters. 67(1-3). 353–363. 13 indexed citations
9.
Weber, Frank, Richard Meyermann, & Klaus Hempel. (1991). Experimental Allergic Encephalomyelitis – Prophylactic and Therapeutic Treatment with the Cyclooxygenase Inhibitor Piroxicam (Feldene®). International Archives of Allergy and Immunology. 95(2-3). 136–141. 11 indexed citations
10.
Weber, Frank & Klaus Hempel. (1989). Protection against Experimental Allergic Encephalomyelitis with Complete Freund’s Adjuvant Is Unaffected by Prostaglandin Synthesis Inhibition. International Archives of Allergy and Immunology. 89(2-3). 242–245. 3 indexed citations
11.
Hempel, Klaus, et al.. (1987). [Extracranial extraneural metastasizing brain tumor].. PubMed. 8(1). 56–60. 1 indexed citations
12.
Hempel, Klaus, et al.. (1985). Unresponsiveness to Experimental Allergic Encephalomyelitis in Lewis Rats Pretreated with Complete Freund’s Adjuvant. International Archives of Allergy and Immunology. 76(3). 193–199. 27 indexed citations
13.
Hempel, Klaus, et al.. (1977). Conformational Changes Induced by Ionic Strength and pH in Two Bovine Myelin Basic Proteins. Hoppe-Seyler´s Zeitschrift für physiologische Chemie. 358(2). 1345–1352. 13 indexed citations
14.
Hempel, Klaus, et al.. (1969). Quantitative Analyse der Catecholamin-Biosynthese des Nebennierenmarks in vivo und Ruhesekretion neugebildeter Amine unter besonderer Bercksichtigung des Dopamins@@@Quantitative analysis of catecholamine biosynthesis in adrenal medulla in vivo and resting secretion of synthesized amines with special consideration of dopamine. Naunyn-Schmiedeberg s Archives of Pharmacology. 264(4). 363–388. 5 indexed citations
15.
Hempel, Klaus, et al.. (1969). Fraktionierung und EigenschaftenN-methylierter Lysine. Hoppe-Seyler´s Zeitschrift für physiologische Chemie. 350(2). 966–972. 9 indexed citations
16.
Hempel, Klaus. (1968). Strahlenempfindlichkeit von DNS-Synthese und Mitose im Harding-Passey-Melanom und im D�nndarm der Maus. Radiation and Environmental Biophysics. 4(4). 356–369. 1 indexed citations
18.
Hempel, Klaus, et al.. (1963). Einfluss des dopa-decarboxylase-hemmers alpha-methyl-dopa auf die umwandlung von dopa in melanin und brenzcatechinamine. In vitro untersuchungen an melanom/mausen mit tritiertem dopa.. Naunyn-Schmiedeberg s Archives of Pharmacology. 246(2). 203. 3 indexed citations
19.
Hempel, Klaus, et al.. (1963). Einflu� des Dopa-Decarboxylase-Hemmers ?-Methyl-Dopa auf die Umwandlung von Dopa in Melanin und Brenzcatechinamine. Naunyn-Schmiedeberg s Archives of Pharmacology. 246(2). 203–14. 6 indexed citations
20.
Birkofer, Leonhard & Klaus Hempel. (1960). Eine neue Lysin‐Synthese, besonders geeignet zur Darstellung von tritiummarkiertem Lysin. Chemische Berichte. 93(10). 2282–2284. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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