Kiyoko Ikeya

474 total citations
13 papers, 394 citations indexed

About

Kiyoko Ikeya is a scholar working on Molecular Biology, Genetics and Genetics. According to data from OpenAlex, Kiyoko Ikeya has authored 13 papers receiving a total of 394 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 3 papers in Genetics and 2 papers in Genetics. Recurrent topics in Kiyoko Ikeya's work include Muscle Physiology and Disorders (10 papers), RNA modifications and cancer (2 papers) and Genetic Neurodegenerative Diseases (2 papers). Kiyoko Ikeya is often cited by papers focused on Muscle Physiology and Disorders (10 papers), RNA modifications and cancer (2 papers) and Genetic Neurodegenerative Diseases (2 papers). Kiyoko Ikeya collaborates with scholars based in Japan, Pakistan and United States. Kiyoko Ikeya's co-authors include O W McBride, A. Bale, Daniel W. Nebert, Daniel D. Petersen, Cindy E. McKinney, Shioko Kimura, D W Nebert, R. A. Owens, John E. Jones and Anil K. Jaiswal and has published in prestigious journals such as The Journal of Pediatrics, Molecular Endocrinology and Acta Neuropathologica.

In The Last Decade

Kiyoko Ikeya

11 papers receiving 385 citations

Peers

Kiyoko Ikeya
Comparison fields: 5 of 66
  • Molecular Biology 254
  • Pharmacology 139
  • Cancer Research 80
  • Oncology 64
  • Genetics 63
Replace Eric Tien with:
Eric Tien United States
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G Brun France
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Irfan M. Hisamuddin United States
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Citations per field, relative to Kiyoko Ikeya
Kiyoko Ikeya · 1×
Citations per year, relative to Kiyoko Ikeya
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Countries citing papers authored by Kiyoko Ikeya

Since Specialization
Citations

This map shows the geographic impact of Kiyoko Ikeya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kiyoko Ikeya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kiyoko Ikeya more than expected).

Fields of papers citing papers by Kiyoko Ikeya

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kiyoko Ikeya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kiyoko Ikeya. The network helps show where Kiyoko Ikeya may publish in the future.

Co-authorship network of co-authors of Kiyoko Ikeya

This figure shows the co-authorship network connecting the top 25 collaborators of Kiyoko Ikeya. A scholar is included among the top collaborators of Kiyoko Ikeya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kiyoko Ikeya. Kiyoko Ikeya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
# Work Indexed citations
1 22
2
Clinicopathological study of transiently ambulant Fukuyama-type congenital muscular dystrophy cases
2
3 32
4 9
5 1
6 16
7 15
8
[Molecular genetics of Duchenne/Becker muscular dystrophy].
0
9 4
10
[Genetic counselling for Duchenne muscular dystrophy].
0
11
Human CYP1A1 gene: cosegregation of the enzyme inducibility phenotype and an RFLP.
152
12 22
13 119

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026