Kimberly A. Mace

3.4k total citations
47 papers, 2.6k citations indexed

About

Kimberly A. Mace is a scholar working on Immunology, Molecular Biology and Rehabilitation. According to data from OpenAlex, Kimberly A. Mace has authored 47 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 14 papers in Molecular Biology and 13 papers in Rehabilitation. Recurrent topics in Kimberly A. Mace's work include Immune cells in cancer (14 papers), Wound Healing and Treatments (13 papers) and Mesenchymal stem cell research (7 papers). Kimberly A. Mace is often cited by papers focused on Immune cells in cancer (14 papers), Wound Healing and Treatments (13 papers) and Mesenchymal stem cell research (7 papers). Kimberly A. Mace collaborates with scholars based in United Kingdom, United States and Australia. Kimberly A. Mace's co-authors include Robert A. Duce, Nancy Boudreau, William McGinnis, Matthew J. Hardman, David M. Young, Joseph C. Pearson, Paulo Artaxo, Kate Wicks, N. Kubilay and Neil Tindale and has published in prestigious journals such as Science, Journal of Geophysical Research Atmospheres and Blood.

In The Last Decade

Kimberly A. Mace

46 papers receiving 2.6k citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Kimberly A. Mace 665 617 521 423 319 47 2.6k
Shin‐ichi Nakano 846 1.3× 62 0.1× 79 0.2× 89 0.2× 742 2.3× 231 3.9k
Jeeyoung Kim 449 0.7× 268 0.4× 17 0.0× 142 0.3× 87 0.3× 95 1.8k
Laura S. Leo 1.3k 2.0× 418 0.7× 35 0.1× 66 0.2× 45 0.1× 75 3.3k
L. R. Johnson 922 1.4× 412 0.7× 25 0.0× 110 0.3× 93 0.3× 70 2.3k
Yanwen Qin 807 1.2× 101 0.2× 15 0.0× 300 0.7× 268 0.8× 183 4.3k
Jinhong Zhu 2.7k 4.0× 622 1.0× 84 0.2× 221 0.5× 181 0.6× 180 4.6k
Haifeng Gu 2.1k 3.2× 154 0.2× 28 0.1× 138 0.3× 1.8k 5.6× 249 4.7k
Guillaume Mabilleau 1.2k 1.8× 123 0.2× 66 0.1× 129 0.3× 94 0.3× 106 3.1k
Richard Mackenzie 473 0.7× 34 0.1× 204 0.4× 29 0.1× 63 0.2× 47 1.9k
Hitoshi Yokoyama 1.6k 2.4× 21 0.0× 188 0.4× 326 0.8× 124 0.4× 139 3.5k

Countries citing papers authored by Kimberly A. Mace

Since Specialization
Citations

This map shows the geographic impact of Kimberly A. Mace's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kimberly A. Mace with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kimberly A. Mace more than expected).

Fields of papers citing papers by Kimberly A. Mace

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kimberly A. Mace. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kimberly A. Mace. The network helps show where Kimberly A. Mace may publish in the future.

Co-authorship network of co-authors of Kimberly A. Mace

This figure shows the co-authorship network connecting the top 25 collaborators of Kimberly A. Mace. A scholar is included among the top collaborators of Kimberly A. Mace based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kimberly A. Mace. Kimberly A. Mace is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Umehara, Takahiro, Ryoichi Mori, Kimberly A. Mace, et al.. (2022). circRNAs May Be Involved in Dysfunction of Neutrophils of Type 2 Diabetic Mice through Regulation of Specific miRNAs. Biomedicines. 10(12). 3129–3129. 1 indexed citations
2.
Mace, Kimberly A., et al.. (2022). Status and challenges of electrical stimulation use in chronic wound healing. Current Opinion in Biotechnology. 75. 102710–102710. 61 indexed citations
3.
Gherardini, Jennifer, Youhei Uchida, J. Hardman, et al.. (2020). Tissue-resident macrophages can be generated de novo in adult human skin from resident progenitor cells during substance P-mediated neurogenic inflammation ex vivo. PLoS ONE. 15(1). e0227817–e0227817. 17 indexed citations
4.
Bridoux, Laure, Víctor Latorre, Syed Murtuza Baker, et al.. (2020). HOX paralogs selectively convert binding of ubiquitous transcription factors into tissue-specific patterns of enhancer activation. PLoS Genetics. 16(12). e1009162–e1009162. 21 indexed citations
5.
Wilkinson, Holly N., et al.. (2019). Elevated Local Senescence in Diabetic Wound Healing Is Linked to Pathological Repair via CXCR2. Journal of Investigative Dermatology. 139(5). 1171–1181.e6. 93 indexed citations
6.
Wilkinson, Holly N., et al.. (2019). Tissue Iron Promotes Wound Repair via M2 Macrophage Polarization and the Chemokine (C-C Motif) Ligands 17 and 22. American Journal Of Pathology. 189(11). 2196–2208. 58 indexed citations
7.
Burgess, Matthew, et al.. (2019). Cx3CR1 Expression Identifies Distinct Macrophage Populations That Contribute Differentially to Inflammation and Repair. ImmunoHorizons. 3(7). 262–273. 72 indexed citations
8.
Wilkinson, Holly N., et al.. (2019). Reduced Iron in Diabetic Wounds: An Oxidative Stress-Dependent Role for STEAP3 in Extracellular Matrix Deposition and Remodeling. Journal of Investigative Dermatology. 139(11). 2368–2377.e7. 39 indexed citations
9.
McKenzie, Edward A., et al.. (2019). Dysregulation of macrophage development and phenotype in diabetic human macrophages can be rescued by Hoxa3 protein transduction. PLoS ONE. 14(10). e0223980–e0223980. 16 indexed citations
10.
Vara, Dina, Matthew Burgess, Kate Wicks, et al.. (2017). Direct Activation of NADPH Oxidase 2 by 2-Deoxyribose-1-Phosphate Triggers Nuclear Factor Kappa B-Dependent Angiogenesis. Antioxidants and Redox Signaling. 28(2). 110–130. 31 indexed citations
11.
Burgess, Matthew, et al.. (2016). Enforced Expression of Hoxa3 Inhibits Classical and Promotes Alternative Activation of Macrophages In Vitro and In Vivo. The Journal of Immunology. 197(3). 872–884. 22 indexed citations
12.
Mace, Kimberly A. & Kristin M. Braun. (2012). Progenitor cells : methods and protocols. Humana Press eBooks. 3 indexed citations
13.
Mahdipour, Elahe & Kimberly A. Mace. (2012). Analyzing the Angiogenic Potential of Gr-1+CD11b+ Immature Myeloid Cells from Murine Wounds. Methods in molecular biology. 916. 219–229. 4 indexed citations
14.
Tondeur, Yves, et al.. (2012). Environmental release of dioxins from reservoir sources during beach nourishment programs. Chemosphere. 88(3). 358–363. 2 indexed citations
15.
Mahdipour, Elahe & Kimberly A. Mace. (2011). Hox transcription factor regulation of adult bone-marrow-derived cell behaviour during tissue repair and regeneration. Expert Opinion on Biological Therapy. 11(8). 1079–1090. 12 indexed citations
16.
Lê, Oanh, et al.. (2009). Secretion of SDF‐1α by bone marrow‐derived stromal cells enhances skin wound healing of C57BL/6 mice exposed to ionizing radiation. Journal of Cellular and Molecular Medicine. 14(6b). 1594–1604. 24 indexed citations
17.
Yu, Diana, Kimberly A. Mace, Scott L. Hansen, Nancy Boudreau, & David M. Young. (2007). Effects of decreased insulin‐like growth factor‐1 stimulation on hypoxia inducible factor 1‐α protein synthesis and function during cutaneous repair in diabetic mice. Wound Repair and Regeneration. 15(5). 628–635. 22 indexed citations
18.
Mace, Kimberly A., Diana Yu, Keyianoosh Z. Paydar, Nancy Boudreau, & David M. Young. (2007). Sustained expression of Hif‐1α in the diabetic environment promotes angiogenesis and cutaneous wound repair. Wound Repair and Regeneration. 15(5). 636–645. 142 indexed citations
19.
Mace, Kimberly A., Joseph C. Pearson, & William McGinnis. (2005). An Epidermal Barrier Wound Repair Pathway in Drosophila Is Mediated by grainy head. Science. 308(5720). 381–385. 184 indexed citations
20.
Mace, Kimberly A. & Antonio Tugores. (2004). The product of the split ends gene is required for the maintenance of positional information during Drosophiladevelopment. BMC Developmental Biology. 4(1). 15–15. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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