Kim L. Powell

2.4k total citations
47 papers, 1.8k citations indexed

About

Kim L. Powell is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Psychiatry and Mental health. According to data from OpenAlex, Kim L. Powell has authored 47 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Cellular and Molecular Neuroscience, 31 papers in Molecular Biology and 19 papers in Psychiatry and Mental health. Recurrent topics in Kim L. Powell's work include Neuroscience and Neuropharmacology Research (30 papers), Ion channel regulation and function (22 papers) and Epilepsy research and treatment (18 papers). Kim L. Powell is often cited by papers focused on Neuroscience and Neuropharmacology Research (30 papers), Ion channel regulation and function (22 papers) and Epilepsy research and treatment (18 papers). Kim L. Powell collaborates with scholars based in Australia, Canada and France. Kim L. Powell's co-authors include Terence J. O’Brien, Christopher A. Reid, David A. Williams, Mastura Monif, Megan L. Smart, Pablo M. Casillas‐Espinosa, Terrance P. Snutch, Nigel C. Jones, Stuart M. Cain and Marian Todaro and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Brain.

In The Last Decade

Kim L. Powell

46 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kim L. Powell Australia 22 915 752 638 285 238 47 1.8k
Maria José da Silva Fernandes Brazil 24 774 0.8× 394 0.5× 479 0.8× 314 1.1× 144 0.6× 51 1.5k
Albert Sattin United States 22 1.0k 1.1× 792 1.1× 202 0.3× 432 1.5× 151 0.6× 61 2.0k
Clemens Allgaier Germany 30 1.2k 1.4× 1.3k 1.7× 213 0.3× 682 2.4× 256 1.1× 71 2.3k
Chris G. Dulla United States 24 865 0.9× 503 0.7× 302 0.5× 90 0.3× 238 1.0× 47 1.5k
Nicholas Rensing United States 24 626 0.7× 971 1.3× 367 0.6× 73 0.3× 170 0.7× 44 2.4k
Maria Kukley Germany 21 1.1k 1.2× 604 0.8× 107 0.2× 210 0.7× 531 2.2× 34 1.9k
Johannes Brockhaus Germany 21 829 0.9× 649 0.9× 73 0.1× 151 0.5× 313 1.3× 34 1.8k
Stéphanie Schorge United Kingdom 29 1.6k 1.8× 1.8k 2.4× 629 1.0× 43 0.2× 123 0.5× 67 3.0k
Raimondo D’Ambrosio United States 25 1.3k 1.4× 869 1.2× 645 1.0× 29 0.1× 328 1.4× 34 2.1k
D M Chuang United States 21 880 1.0× 1.1k 1.4× 380 0.6× 81 0.3× 123 0.5× 37 2.0k

Countries citing papers authored by Kim L. Powell

Since Specialization
Citations

This map shows the geographic impact of Kim L. Powell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kim L. Powell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kim L. Powell more than expected).

Fields of papers citing papers by Kim L. Powell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kim L. Powell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kim L. Powell. The network helps show where Kim L. Powell may publish in the future.

Co-authorship network of co-authors of Kim L. Powell

This figure shows the co-authorship network connecting the top 25 collaborators of Kim L. Powell. A scholar is included among the top collaborators of Kim L. Powell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kim L. Powell. Kim L. Powell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Zining, Shobi Sivathamboo, Idrish Ali, et al.. (2025). Cardiac structural and molecular alterations in rodent models of temporal lobe epilepsy. Epilepsia Open. 10(3). 809–821. 2 indexed citations
2.
Mellor, Kimberley M., Upasna Varma, V.L. Benson, et al.. (2024). Myocardial glycophagy flux dysregulation and glycogen accumulation characterize diabetic cardiomyopathy. Journal of Molecular and Cellular Cardiology. 189. 83–89. 8 indexed citations
3.
Jazayeri, Dana, Emma L. Braine, Stuart J. McDonald, et al.. (2020). A rat model of valproate teratogenicity from chronic oral treatment during pregnancy. Epilepsia. 61(6). 1291–1300. 12 indexed citations
4.
Zamani, Akram, Kim L. Powell, Ashleigh L. May, & Bridgette D. Semple. (2020). Validation of reference genes for gene expression analysis following experimental traumatic brain injury in a pediatric mouse model. Brain Research Bulletin. 156. 43–49. 22 indexed citations
5.
Casillas‐Espinosa, Pablo M., Sandy R. Shultz, Emma L. Braine, et al.. (2019). Disease-modifying effects of a novel T-type calcium channel antagonist, Z944, in a model of temporal lobe epilepsy. Progress in Neurobiology. 182. 101677–101677. 28 indexed citations
6.
Powell, Kim L., Valentina Jovanovska, Matthias Klugmann, et al.. (2018). Gene therapy mediated seizure suppression in Genetic Generalised Epilepsy: Neuropeptide Y overexpression in a rat model. Neurobiology of Disease. 113. 23–32. 23 indexed citations
7.
Cain, Stuart M., John R. Tyson, Rebecca W.Y. Ko, et al.. (2018). CaV3.2 drives sustained burst‐firing, which is critical for absence seizure propagation in reticular thalamic neurons. Epilepsia. 59(4). 778–791. 39 indexed citations
8.
Wright, David, Leigh A. Johnston, Kim L. Powell, et al.. (2017). Differences in white matter structure between seizure prone (FAST) and seizure resistant (SLOW) rat strains. Neurobiology of Disease. 104. 33–40. 18 indexed citations
10.
Casillas‐Espinosa, Pablo M., et al.. (2015). Z944, a Novel Selective T-Type Calcium Channel Antagonist Delays the Progression of Seizures in the Amygdala Kindling Model. PLoS ONE. 10(8). e0130012–e0130012. 43 indexed citations
11.
Powell, Kim L., Nigel C. Jones, Thomas Zheng, et al.. (2014). HCN channelopathy and cardiac electrophysiologic dysfunction in genetic and acquired rat epilepsy models. Epilepsia. 55(4). 609–620. 26 indexed citations
13.
Tringham, Elizabeth, Kim L. Powell, Stuart M. Cain, et al.. (2012). T-Type Calcium Channel Blockers That Attenuate Thalamic Burst Firing and Suppress Absence Seizures. Science Translational Medicine. 4(121). 121ra19–121ra19. 162 indexed citations
14.
Morokoff, Andrew, Andrew R. Bjorksten, Giovanna M. D’Abaco, et al.. (2012). Glutamate is associated with a higher risk of seizures in patients with gliomas. Neurology. 79(9). 883–889. 135 indexed citations
15.
Reid, Christopher A., Damian E. Myers, Kim L. Powell, et al.. (2009). Excitotoxic-mediated transcriptional decreases in HCN2 channel function increase network excitability in CA1. Experimental Neurology. 219(1). 249–257. 13 indexed citations
16.
Ng, Caroline L., Kim L. Powell, Mervyn Kyi, et al.. (2008). A CAV3.2 T-TYPE CALCIUM CHANNEL POINT MUTATION HAS SPLICE VARIANT-SPECIFIC EFFECTS ON FUNCTION AND SEGREGATES WITH SEIZURE EXPRESSION IN A POLYGENIC RAT MODEL OF ABSENCE EPILEPSY. Epilepsia. 49. 1 indexed citations
17.
Powell, Kim L., et al.. (2008). Decreases in HCN mRNA expression in the hippocampus after kindling and status epilepticus in adult rats. Epilepsia. 49(10). 1686–1695. 71 indexed citations
18.
Powell, Kim L., Mervyn Kyi, Christopher J. Ng, et al.. (2007). A novel mutation in the rat Ca(v)3.2 T-type Ca2 channel CACNA1H gene increases absence seizure expression. Epilepsia. 48. 1 indexed citations
19.
Leck, Kwong Joo, Selena E. Bartlett, Maree T. Smith, et al.. (2004). Deletion of guanine nucleotide binding protein αz subunit in mice induces a gene dose dependent tolerance to morphine. Neuropharmacology. 46(6). 836–846. 23 indexed citations
20.
Wellby, M.L., Kim L. Powell, Margaret Carman, & Basil S. Hetzel. (1972). Comparative Studies of Diiodotyrosine Deiodinase Activities in Endemic Goiter and Congenital Goiter1. The Journal of Clinical Endocrinology & Metabolism. 35(5). 762–763. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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