Kezhou Xia

460 total citations
23 papers, 350 citations indexed

About

Kezhou Xia is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Kezhou Xia has authored 23 papers receiving a total of 350 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 8 papers in Cancer Research and 5 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Kezhou Xia's work include RNA modifications and cancer (6 papers), Cancer-related molecular mechanisms research (5 papers) and Bone Tissue Engineering Materials (3 papers). Kezhou Xia is often cited by papers focused on RNA modifications and cancer (6 papers), Cancer-related molecular mechanisms research (5 papers) and Bone Tissue Engineering Materials (3 papers). Kezhou Xia collaborates with scholars based in China, United Kingdom and Ethiopia. Kezhou Xia's co-authors include Ling Yu, Di Zheng, Zhun Wei, Weichun Guo, Zhengpei Zhang, Ling Yu, Tian Gao, Wei Li, Yanan Zhao and Honglian Dai and has published in prestigious journals such as Scientific Reports, Biochemical and Biophysical Research Communications and International Journal of Biological Macromolecules.

In The Last Decade

Kezhou Xia

21 papers receiving 349 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kezhou Xia China 12 181 96 88 70 52 23 350
Xinyu Qu China 14 214 1.2× 103 1.1× 111 1.3× 50 0.7× 74 1.4× 25 403
Yali Cheng China 11 159 0.9× 94 1.0× 104 1.2× 54 0.8× 45 0.9× 16 389
Sung Ouk Nam Japan 10 130 0.7× 96 1.0× 79 0.9× 61 0.9× 42 0.8× 15 315
Xudong Fu China 12 154 0.9× 50 0.5× 80 0.9× 50 0.7× 42 0.8× 22 341
Yong Xiao China 12 168 0.9× 101 1.1× 89 1.0× 75 1.1× 26 0.5× 41 392
Xiaoping Zhong China 11 169 0.9× 75 0.8× 75 0.9× 39 0.6× 35 0.7× 26 372
Chengwan Xia China 11 158 0.9× 66 0.7× 133 1.5× 45 0.6× 39 0.8× 23 318
Zhaowei Xu China 15 294 1.6× 99 1.0× 134 1.5× 82 1.2× 91 1.8× 28 547

Countries citing papers authored by Kezhou Xia

Since Specialization
Citations

This map shows the geographic impact of Kezhou Xia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kezhou Xia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kezhou Xia more than expected).

Fields of papers citing papers by Kezhou Xia

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kezhou Xia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kezhou Xia. The network helps show where Kezhou Xia may publish in the future.

Co-authorship network of co-authors of Kezhou Xia

This figure shows the co-authorship network connecting the top 25 collaborators of Kezhou Xia. A scholar is included among the top collaborators of Kezhou Xia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kezhou Xia. Kezhou Xia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Liu, Wenda, et al.. (2025). HMGCL activates autophagy in osteosarcoma through β-HB mediated inhibition of the PI3K/AKT/mTOR signaling pathway. Journal of Translational Medicine. 23(1). 219–219. 3 indexed citations
3.
Yu, Ling, Tian Gao, X. Dong, et al.. (2024). Nanofiber-induced hierarchically-porous magnesium phosphate bone cements accelerate bone regeneration by inhibiting Notch signaling. Bioactive Materials. 37. 459–476. 12 indexed citations
4.
Xia, Kezhou, et al.. (2024). CGREF1 modulates osteosarcoma proliferation by regulating the cell cycle through the Wnt/β-catenin signaling pathway. Molecular Medicine. 30(1). 260–260. 2 indexed citations
5.
Wei, Zhun, et al.. (2024). DUSP3 restrains the progression and stemness property of osteosarcoma through regulating EGFR/STAT3/SOX2 axis. International Journal of Biological Sciences. 21(1). 160–174.
6.
Xia, Kezhou, et al.. (2024). SLC38A5 suppresses ferroptosis through glutamine-mediated activation of the PI3K/AKT/mTOR signaling in osteosarcoma. Journal of Translational Medicine. 22(1). 1004–1004. 7 indexed citations
7.
Xia, Kezhou, et al.. (2023). SERPINH1 enhances the malignancy of osteosarcoma via PI3K-Akt signaling pathway. Translational Oncology. 39. 101802–101802. 9 indexed citations
8.
Wei, Zhun, et al.. (2023). RILP inhibits tumor progression in osteosarcoma via Grb10-mediated inhibition of the PI3K/AKT/mTOR pathway. Molecular Medicine. 29(1). 133–133. 14 indexed citations
9.
10.
Xia, Kezhou, et al.. (2023). TRIM26 inhibited osteosarcoma progression through destabilizing RACK1 and thus inactivation of MEK/ERK signaling. Cell Death and Disease. 14(8). 529–529. 17 indexed citations
11.
Liu, Wenda, et al.. (2023). Construction of a prognostic risk score model based on the ARHGAP family to predict the survival of osteosarcoma. BMC Cancer. 23(1). 1179–1179. 3 indexed citations
12.
Zheng, Di, et al.. (2022). Identification of a novel gene signature with regard to ferroptosis, prognosis prediction, and immune microenvironment in osteosarcoma. Frontiers in Genetics. 13. 944978–944978. 8 indexed citations
13.
Zheng, Di, Ling Yu, Zhun Wei, Kezhou Xia, & Weichun Guo. (2022). N6-Methyladenosine-Related lncRNAs Are Potential Prognostic Biomarkers and Correlated With Tumor Immune Microenvironment in Osteosarcoma. Frontiers in Genetics. 12. 805607–805607. 13 indexed citations
14.
Zheng, Di, Kezhou Xia, Ling Yu, et al.. (2021). A Novel Six Metastasis-Related Prognostic Gene Signature for Patients With Osteosarcoma. Frontiers in Cell and Developmental Biology. 9. 699212–699212. 17 indexed citations
15.
Yu, Ling, et al.. (2020). An injectable bioactive magnesium phosphate cement incorporating carboxymethyl chitosan for bone regeneration. International Journal of Biological Macromolecules. 160. 101–111. 62 indexed citations
16.
Fang, Shuo, et al.. (2020). Enhancing the mechanical properties and cytocompatibility of magnesium potassium phosphate cement by incorporating oxygen-carboxymethyl chitosan. Regenerative Biomaterials. 8(1). rbaa048–rbaa048. 28 indexed citations
17.
Yu, Ling, Kezhou Xia, Tian Gao, et al.. (2019). The Notch Pathway Promotes Osteosarcoma Progression through Activation of Ephrin Reverse Signaling. Molecular Cancer Research. 17(12). 2383–2394. 31 indexed citations
18.
Fang, Shuo, Tao Hai, Kezhou Xia, & Weichun Guo. (2019). Proscillaridin A induces apoptosis and inhibits the metastasis of osteosarcoma in vitro and in vivo. Biochemical and Biophysical Research Communications. 521(4). 880–886. 11 indexed citations
19.
Zhang, Zhengpei, Ling Yu, Guo Dai, et al.. (2017). Telomerase reverse transcriptase promotes chemoresistance by suppressing cisplatin-dependent apoptosis in osteosarcoma cells. Scientific Reports. 7(1). 7070–7070. 31 indexed citations
20.
Yang, Jian, Weichun Guo, Lu Wang, et al.. (2017). Notch signaling is important for epithelial-mesenchymal transition induced by low concentrations of doxorubicin in osteosarcoma cell lines. Oncology Letters. 13(4). 2260–2268. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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