Kerstin A. Stegmann
- Immunology top 5%
- Immune Cell Function and Interaction 14
- T-cell and B-cell Immunology 8
- IL-33, ST2, and ILC Pathways 3
- Immunotherapy and Immune Responses 2
- Hepatology top 5%
- Hepatitis C virus research 2
- Epidemiology top 10%
- Cytomegalovirus and herpesvirus research 2
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- CAR-T cell therapy research 2
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- Adenosine and Purinergic Signaling 1
- Co-authors
- Mala K. MainiLaura J. PallettBrian R DavidsonVictoria MaleFrancis P. RobertsonEleanor M. RileyAmir HorowitzNavjyot Hansi
- Cited by
- ImmunologyHepatologyEpidemiology
- Journals
- Scientific Reports (2 papers)Frontiers in Immunology (2 papers)Seminars in Liver Disease (1 paper)
- Partner nations
- United KingdomGermanySweden
In The Last Decade
Kerstin A. Stegmann
17 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 72
- Immunology 842
- Hepatology 247
- Epidemiology 299
- Virology 31
- Oncology 163
Countries citing papers authored by Kerstin A. Stegmann
This map shows the geographic impact of Kerstin A. Stegmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kerstin A. Stegmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kerstin A. Stegmann more than expected).
Fields of papers citing papers by Kerstin A. Stegmann
This network shows the impact of papers produced by Kerstin A. Stegmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kerstin A. Stegmann. The network helps show where Kerstin A. Stegmann may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Kerstin A. Stegmann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 3 | |
| 2 | 2022 | 30 | |
| 3 | 2022 | 2 | |
| 4 | 2020 | 20 | |
| 5 | 2020 | 58 | |
| 6 | 2018 | 77 | |
| 7 | 2017 | 22 | |
| 8 | 2017 | 250 | |
| 9 | 2016 | 187 | |
| 10 | 2016 | 96 | |
| 11 | 2015 | 28 | |
| 12 | 2012 | 37 | |
| 13 | 2012 | 95 | |
| 14 | 2010 | 149 | |
| 15 | 2009 | 13 | |
| 16 | 2009 | 24 | |
| 17 | 2008 | 15 |
About Kerstin A. Stegmann
Kerstin A. Stegmann is a scholar working on Immunology, Hepatology and Physiology, having authored 17 papers that have together received 1.1k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (14 papers), T-cell and B-cell Immunology (8 papers), IL-33, ST2, and ILC Pathways (3 papers), CAR-T cell therapy research (2 papers), Immunotherapy and Immune Responses (2 papers), Hepatitis C virus research (2 papers), Cytomegalovirus and herpesvirus research (2 papers) and Adenosine and Purinergic Signaling (1 paper). The work is most often cited by research in Immunology (842 citations), Hepatology (247 citations) and Epidemiology (299 citations). Kerstin A. Stegmann has collaborated with scholars based in United Kingdom, Germany and Sweden. Frequent co-authors include Mala K. Maini, Laura J. Pallett, Brian R Davidson, Victoria Male, Francis P. Robertson, Eleanor M. Riley, Amir Horowitz, Navjyot Hansi, Upkar S. Gill and Patrick Kennedy. Their work appears in journals such as Scientific Reports, Frontiers in Immunology, Seminars in Liver Disease, Journal of Leukocyte Biology and Cell Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.