Kenshi Imoto
- Cellular and Molecular Neuroscience top 5%
- Molecular Biology
- Cognitive Neuroscience top 10%
- Pharmacology
- Cell Biology
- Co-authors
- Bert SakmannFritjof HelmchenHideyo OhshikaShoji SakanoHaruo TakemuraChiu‐Yin KwanKeiko YamadaToshihiko Yamashita
- Topics
- Ion channel regulation and function (6 papers)Neuroscience and Neuropharmacology Research (5 papers)Berberine and alkaloids research (3 papers)
In The Last Decade
Kenshi Imoto
20 papers receiving 646 citations
Peers
Comparison fields: 5 of 78
- Cellular and Molecular Neuroscience 457
- Molecular Biology 319
- Cognitive Neuroscience 235
- Pharmacology 49
- Cell Biology 46
Countries citing papers authored by Kenshi Imoto
This map shows the geographic impact of Kenshi Imoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenshi Imoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenshi Imoto more than expected).
Fields of papers citing papers by Kenshi Imoto
This network shows the impact of papers produced by Kenshi Imoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenshi Imoto. The network helps show where Kenshi Imoto may publish in the future.
Co-authorship network of co-authors of Kenshi Imoto
This figure shows the co-authorship network connecting the top 25 collaborators of Kenshi Imoto. A scholar is included among the top collaborators of Kenshi Imoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenshi Imoto. Kenshi Imoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 16 | |
| 3 | 9 | |
| 4 | [Bone and bone related biochemical examinations. Bone and collagen related metabolites. DPD (total, free)]. | 1 |
| 5 | 7 | |
| 6 | [Adequate treatment with bisphosphonates for senile osteoporosis]. | 1 |
| 7 | 8 | |
| 8 | Involvement of tyrosine kinase in capacitative Ca2+ entry pathway in rat glioma C6 cells. | 4 |
| 9 | 4 | |
| 10 | Inhibitory effects of tetrandrine and hernandezine on Ca2+ mobilization in rat glioma C6 cells. | 17 |
| 11 | Slow calcium-induced calcium release (CICR) in Chinese hamster ovary (CHO) cells expressing skeletal ryanodine receptor (RyR) and chimaeric dihydropyridine receptor (DHPR). | 2 |
| 12 | 31 | |
| 13 | 42 | |
| 14 | 468 | |
| 15 | Lack of voltage-operated calcium channel in rat glioma C6 cells. | 11 |
| 16 | 3 | |
| 17 | 1 | |
| 18 | 14 | |
| 19 | 4 | |
| 20 | Molecular pore structure of voltage-gated sodium and calcium channels. | 18 |
About Kenshi Imoto
Kenshi Imoto is a scholar working on Cellular and Molecular Neuroscience, Orthopedics and Sports Medicine and Electrochemistry, having authored 20 papers that have together received 664 indexed citations. Recurring topics across this work include Ion channel regulation and function (6 papers), Neuroscience and Neuropharmacology Research (5 papers) and Berberine and alkaloids research (3 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (457 citations), Cognitive Neuroscience (235 citations) and Sensory Systems (45 citations). Kenshi Imoto has collaborated with scholars based in Japan, Germany and Spain. Frequent co-authors include Bert Sakmann, Fritjof Helmchen, Hideyo Ohshika, Shoji Sakano, Haruo Takemura, Haruo Takemura, Chiu‐Yin Kwan, Keiko Yamada, Toshihiko Yamashita and Stefan H. Heinemann. Their work appears in journals such as Journal of Biological Chemistry, Biochemical Journal and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.