Keizo Inoue

696 total citations
11 papers, 613 citations indexed

About

Keizo Inoue is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Keizo Inoue has authored 11 papers receiving a total of 613 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Cell Biology and 3 papers in Genetics. Recurrent topics in Keizo Inoue's work include Protein Kinase Regulation and GTPase Signaling (7 papers), Cellular transport and secretion (4 papers) and Venomous Animal Envenomation and Studies (3 papers). Keizo Inoue is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (7 papers), Cellular transport and secretion (4 papers) and Venomous Animal Envenomation and Studies (3 papers). Keizo Inoue collaborates with scholars based in Japan. Keizo Inoue's co-authors include Ichiro Kudo, Shuntaro Hara, Makoto Murakami, Terumasa Miyamoto, Kunio Matsuta, Atsushi Matsuzawa, Hideo Nakamura, Masato Umeda, Hiroshi Mori and Yuichi Yokoyama and has published in prestigious journals such as FEBS Letters, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research and Advances in experimental medicine and biology.

In The Last Decade

Keizo Inoue

11 papers receiving 597 citations

Peers

Keizo Inoue
Comparison fields: 5 of 77
  • Molecular Biology 390
  • Genetics 122
  • Physiology 106
  • Immunology 102
  • Pharmacology 90
Replace Abdur Rehman Siddiqi with:
Abdur Rehman Siddiqi Sweden
Akihiko Saiga Japan
Yasunori Yokota Japan
Margarete Goppelt‐Strübe Germany
W. Pignat Switzerland
Sarah Ruuska United States
Terumi Kambe Japan
M.C.M. van Dijk Netherlands
Theresa Conway United States
H S Banga United States
Abdur Rehman Siddiqi Sweden View profile →
Citations per field, relative to Keizo Inoue
Keizo Inoue · 1×
Citations per year, relative to Keizo Inoue
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Countries citing papers authored by Keizo Inoue

Since Specialization
Citations

This map shows the geographic impact of Keizo Inoue's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keizo Inoue with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keizo Inoue more than expected).

Fields of papers citing papers by Keizo Inoue

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keizo Inoue. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keizo Inoue. The network helps show where Keizo Inoue may publish in the future.

Co-authorship network of co-authors of Keizo Inoue

This figure shows the co-authorship network connecting the top 25 collaborators of Keizo Inoue. A scholar is included among the top collaborators of Keizo Inoue based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keizo Inoue. Keizo Inoue is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
# Work Indexed citations
1 2
2 12
3 3
4 11
5 10
6 305
7 7
8 62
9 2
10 50
11 149

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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