Kei Murayama

5.3k total citations
169 papers, 2.0k citations indexed

About

Kei Murayama is a scholar working on Molecular Biology, Clinical Biochemistry and Physiology. According to data from OpenAlex, Kei Murayama has authored 169 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 122 papers in Molecular Biology, 100 papers in Clinical Biochemistry and 17 papers in Physiology. Recurrent topics in Kei Murayama's work include Mitochondrial Function and Pathology (106 papers), Metabolism and Genetic Disorders (100 papers) and ATP Synthase and ATPases Research (38 papers). Kei Murayama is often cited by papers focused on Mitochondrial Function and Pathology (106 papers), Metabolism and Genetic Disorders (100 papers) and ATP Synthase and ATPases Research (38 papers). Kei Murayama collaborates with scholars based in Japan, United States and Australia. Kei Murayama's co-authors include Akira Ohtake, Yasushi Okazaki, Masaru Shimura, Yoshihito Kishita, Masakazu Kohda, Masaki Takayanagi, Makiko Tajika, Takuya Fushimi, Keiko Ichimoto and Tomoko Tsuruoka and has published in prestigious journals such as Nucleic Acids Research, SHILAP Revista de lepidopterología and Gastroenterology.

In The Last Decade

Kei Murayama

148 papers receiving 1.9k citations

Peers

Kei Murayama

PHYSI

GENET

PPCH

Manuel Schiff France

N. G. G. M. Abeling Netherlands

Pilar Rodríguez‐Pombo Spain

Akira Ohtake Japan

Felix Distelmaier Germany

Saskia B. Wortmann Netherlands

Lambert van den Heuvel Netherlands

Johanna Uusimaa Finland

Neal Sondheimer United States

Paldeep S. Atwal United States

Manuel Schiff France

Kei Murayama

1.3k ×1.1

870 ×1.0

380 ×2.1

PHYSI

255 ×1.5

GENET

200 ×1.2

PPCH

Citations per year, relative to Kei Murayama Kei Murayama (= 1×) peers Manuel Schiff

Countries citing papers authored by Kei Murayama

Since Specialization

Citations

This map shows the geographic impact of Kei Murayama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kei Murayama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kei Murayama more than expected).

Fields of papers citing papers by Kei Murayama

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kei Murayama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kei Murayama. The network helps show where Kei Murayama may publish in the future.

Co-authorship network of co-authors of Kei Murayama

This figure shows the co-authorship network connecting the top 25 collaborators of Kei Murayama. A scholar is included among the top collaborators of Kei Murayama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kei Murayama. Kei Murayama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Nyuzuki, Hiromi, et al.. (2025). Mitochondrial dysfunction in MED13 variant-associated disease: a case of infantile spasms, cardiomyopathy and hepatomegaly. Human Genome Variation. 12(1). 22–22.

Suzuki, Takayuki, et al.. (2025). Fontaine progeroid syndrome with neonatal mitochondrial disease. Human Genome Variation. 12(1). 26–26.

Iijima, Hiroyuki, Ayumu Sugiura, Masaru Shimura, et al.. (2025). Role of BOLA3 in the mitochondrial Fe-S cluster clarified by metabolomic analysis. Molecular Genetics and Metabolism. 145(2). 109113–109113.

Ichimoto, Keiko, et al.. (2024). Increased ketone levels as a key magnetic resonance spectroscopic findings during acute exacerbation in ECHS1-related Leigh syndrome. Radiology Case Reports. 19(12). 6292–6296. 1 indexed citations

Inoue, Kenji, et al.. (2024). X-Linked Myotubular Myopathy and Mitochondrial Function in Muscle and Liver Samples. Neuropediatrics. 56(1). 51–55.

Imasawa, Toshiyuki, Kei Murayama, Daishi Hirano, & Kandai Nozu. (2024). Comprehensive review of mitochondrial nephropathy—a renal phenotype in mitochondrial disease: causative genes, clinical and pathological features, diagnosis, prognosis, and treatment. Clinical and Experimental Nephrology. 29(1). 39–56. 1 indexed citations

Kishita, Yoshihito, Ayumu Sugiura, Tetsuro Matsuhashi, et al.. (2023). Strategic validation of variants of uncertain significance in ECHS1 genetic testing. Journal of Medical Genetics. 60(10). 1006–1015. 2 indexed citations

Ittiwut, Chupong, Rungnapa Ittiwut, Tetsuro Matsuhashi, et al.. (2023). Genetic, metabolic and clinical delineation of an MRPS23-associated mitochondrial disorder. Scientific Reports. 13(1). 22005–22005. 2 indexed citations

Murayama, Kei, et al.. (2023). Acute Encephalopathy Caused by Inherited Metabolic Diseases. Journal of Clinical Medicine. 12(11). 3797–3797. 3 indexed citations

10.

Okamoto, Tatsuya, Miki Yamamoto, Eri Ogawa, et al.. (2023). Living donor liver transplantation for myocerebrohepatopathy spectrum due to POLG mutations. Pediatric Transplantation. 28(1). e14659–e14659. 1 indexed citations

11.

Sharpe, Alice J., Shuai Nie, Mark Ziemann, et al.. (2022). Loss of mitochondrial fatty acid β‐oxidation protein short‐chain Enoyl‐CoA hydratase disrupts oxidative phosphorylation protein complex stability and function. FEBS Journal. 290(1). 225–246. 11 indexed citations

12.

Hattori, Atsushi, Torayuki Okuyama, Motomichi Kosuga, et al.. (2022). Maternal uniparental disomy of chromosome 7 underlying argininosuccinic aciduria and Silver-Russell syndrome. Human Genome Variation. 9(1). 32–32. 1 indexed citations

13.

Tajika, Makiko, Tetsuro Matsuhashi, Masaru Shimura, et al.. (2022). A Japanese single-center experience of the efficacy and safety of asfotase alfa in pediatric-onset hypophosphatasia. Orphanet Journal of Rare Diseases. 17(1). 78–78. 6 indexed citations

14.

Uchida, Hajime, Seisuke Sakamoto, Seiichi Shimizu, et al.. (2021). Outcomes of liver transplantation for mitochondrial respiratory chain disorder in children. Pediatric Transplantation. 25(8). e14091–e14091. 8 indexed citations

15.

Borna, Nurun Nahar, Yoshihito Kishita, Norio Sakai, et al.. (2020). Leigh Syndrome Due to NDUFV1 Mutations Initially Presenting as LBSL. Genes. 11(11). 1325–1325. 8 indexed citations

16.

Inaba, Yuji, Mitsuo Motobayashi, Yoshiro Amano, et al.. (2018). An infant case of diffuse cerebrospinal lesions and cardiomyopathy caused by a BOLA3 mutation. Brain and Development. 40(6). 484–488. 18 indexed citations

17.

Kang, Min‐Ji, et al.. (2016). Identification of DGUOK and MPV17 Mutations in Patients with Hepatocerebral Mitochondrial DNA Depletion Syndrome. 88.

18.

Matsuura, Gen, et al.. (2013). A case of Alagille syndrome with hepatic-duct obliteration presenting intracranial hemorrhage. 89(5). 181–185.

19.

Murayama, Kei & Akira Ohtake. (2009). Mitochondrial respiratory chain disorder and liver disease in children. The Journal of Toxicological Sciences. 34. 1 indexed citations

20.

Nagasaka, Hironori, Haruki Komatsu, Toshihiro Ohura, et al.. (2004). Nitric oxide synthesis in ornithine transcarbamylase deficiency: Possible involvement of low no synthesis in clinical manifestations of urea cycle defect. The Journal of Pediatrics. 145(2). 259–262. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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