Keeley L. Mui

1.1k total citations
10 papers, 802 citations indexed

About

Keeley L. Mui is a scholar working on Cell Biology, Molecular Biology and Immunology and Allergy. According to data from OpenAlex, Keeley L. Mui has authored 10 papers receiving a total of 802 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cell Biology, 4 papers in Molecular Biology and 4 papers in Immunology and Allergy. Recurrent topics in Keeley L. Mui's work include Cellular Mechanics and Interactions (6 papers), Cell Adhesion Molecules Research (4 papers) and Ovarian function and disorders (3 papers). Keeley L. Mui is often cited by papers focused on Cellular Mechanics and Interactions (6 papers), Cell Adhesion Molecules Research (4 papers) and Ovarian function and disorders (3 papers). Keeley L. Mui collaborates with scholars based in United States. Keeley L. Mui's co-authors include Richard K. Assoian, Christopher S. Chen, Brian D. Cosgrove, Steven R. Caliari, Robert L. Mauck, Tristan P. Driscoll, Jason A. Burdick, Shu‐Lin Liu, Yongho Bae and Tina Xu and has published in prestigious journals such as Nature Materials, PLoS ONE and Current Biology.

In The Last Decade

Keeley L. Mui

10 papers receiving 795 citations

Peers

Keeley L. Mui
Jacopo Di Russo Germany
Jill S. Harunaga United States
Aron Parekh United States
Naoya Sakamoto Japan
Yang‐Kao Wang Taiwan
Philippe Pittet Switzerland
Danielle J. LaValley United States
Seiichiro Ishihara Japan
Vladimír Vinarský Czechia
Emiko Fukumoto Japan
Jacopo Di Russo Germany
Keeley L. Mui
Citations per year, relative to Keeley L. Mui Keeley L. Mui (= 1×) peers Jacopo Di Russo

Countries citing papers authored by Keeley L. Mui

Since Specialization
Citations

This map shows the geographic impact of Keeley L. Mui's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keeley L. Mui with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keeley L. Mui more than expected).

Fields of papers citing papers by Keeley L. Mui

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keeley L. Mui. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keeley L. Mui. The network helps show where Keeley L. Mui may publish in the future.

Co-authorship network of co-authors of Keeley L. Mui

This figure shows the co-authorship network connecting the top 25 collaborators of Keeley L. Mui. A scholar is included among the top collaborators of Keeley L. Mui based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keeley L. Mui. Keeley L. Mui is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Zhang, Qiao, Keeley L. Mui, Christopher S. O’Bryan, et al.. (2019). Mechanical Stabilization of the Glandular Acinus by Linker of Nucleoskeleton and Cytoskeleton Complex. Current Biology. 29(17). 2826–2839.e4. 21 indexed citations
2.
Mui, Keeley L., Christopher S. Chen, & Richard K. Assoian. (2016). The mechanical regulation of integrin–cadherin crosstalk organizes cells, signaling and forces. Journal of Cell Science. 129(6). 1093–1100. 238 indexed citations
3.
Cosgrove, Brian D., Keeley L. Mui, Tristan P. Driscoll, et al.. (2016). N-cadherin adhesive interactions modulate matrix mechanosensing and fate commitment of mesenchymal stem cells. Nature Materials. 15(12). 1297–1306. 274 indexed citations
4.
Mui, Keeley L., Yongho Bae, Lin Gao, et al.. (2015). N-Cadherin Induction by ECM Stiffness and FAK Overrides the Spreading Requirement for Proliferation of Vascular Smooth Muscle Cells. Cell Reports. 10(9). 1477–1486. 52 indexed citations
5.
Bae, Yongho, et al.. (2015). Apolipoprotein E3 Inhibits Rho to Regulate the Mechanosensitive Expression of Cox2. PLoS ONE. 10(6). e0128974–e0128974. 9 indexed citations
6.
Bae, Yongho, Keeley L. Mui, Shu‐Lin Liu, et al.. (2014). A FAK-Cas-Rac-Lamellipodin Signaling Module Transduces Extracellular Matrix Stiffness into Mechanosensitive Cell Cycling. Science Signaling. 7(330). ra57–ra57. 163 indexed citations
7.
Romero, Iris L., Anirban Mitra, Ilyssa O. Gordon, et al.. (2011). The effects of 17β-estradiol and a selective estrogen receptor modulator, bazedoxifene, on ovarian carcinogenesis. Gynecologic Oncology. 124(1). 134–141. 11 indexed citations
8.
Romero, Iris L., Ilyssa O. Gordon, Sujatha Jagadeeswaran, et al.. (2009). Effects of Oral Contraceptives or a Gonadotropin-Releasing Hormone Agonist on Ovarian Carcinogenesis in Genetically Engineered Mice. Cancer Prevention Research. 2(9). 792–799. 13 indexed citations
9.
Romero, Iris L., Ilyssa O. Gordon, Sujatha Jagadeeswaran, et al.. (2009). Effects of Oral Contraceptives or a Gonadotropin-Releasing Hormone Agonist on Ovarian Carcinogenesis in Genetically Engineered Mice. Obstetrical & Gynecological Survey. 65(1). 35–37. 8 indexed citations
10.
Kim, Helen, et al.. (2007). Regulation of Gonadotropin-Releasing Hormone in Nonhypothalamic Tissues. Seminars in Reproductive Medicine. 25(5). 326–336. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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