Kazunari Oka
- Neurology top 1%
- Intracranial Aneurysms: Treatment and Complications 9
- Vascular Malformations Diagnosis and Treatment 5
- Rheumatology top 2%
- Moyamoya disease diagnosis and treatment 8
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- Cerebrospinal fluid and hydrocephalus 18
- Psychiatry and Mental health top 5%
- Genetics top 10%
- Glioma Diagnosis and Treatment 4
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- Fetal and Pediatric Neurological Disorders 10
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- Protease and Inhibitor Mechanisms 6
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- Spinal Dysraphism and Malformations 6
- Co-authors
- Masamichi TomonagaMasanori YamashitaKentaro TanakaK TanakaMasaaki YamamotoMargaret BarryAlbert L. RhotonKoichi Ikeda
- Partner nations
- JapanUnited StatesNorway
In The Last Decade
Kazunari Oka
53 papers receiving 1.8k citations
Peers
Comparison fields: 5 of 107
- Neurology 887
- Rheumatology 430
- Cellular and Molecular Neuroscience 426
- Psychiatry and Mental health 294
- Genetics 148
Countries citing papers authored by Kazunari Oka
This map shows the geographic impact of Kazunari Oka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazunari Oka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazunari Oka more than expected).
Fields of papers citing papers by Kazunari Oka
This network shows the impact of papers produced by Kazunari Oka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazunari Oka. The network helps show where Kazunari Oka may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Kazunari Oka, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 5 | |
| 2 | 2016 | 10 | |
| 3 | 2013 | 6 | |
| 4 | 2008 | 17 | |
| 5 | 2007 | 49 | |
| 6 | 2000 | 2 | |
| 7 | 1999 | 53 | |
| 8 | 1999 | 9 | |
| 9 | 1997 | 2 | |
| 10 | 1995 | 21 | |
| 11 | 1994 | 7 | |
| 12 | 1994 | 25 | |
| 13 | 1994 | 13 | |
| 14 | 1994 | 47 | |
| 15 | 1993 | 23 | |
| 16 | 1988 | 18 | |
| 17 | 1985 | 210 | |
| 18 | 1983 | 206 | |
| 19 | 1979 | 72 | |
| 20 | 1978 | 1 |
About Kazunari Oka
Kazunari Oka is a scholar working on Neurology, Cellular and Molecular Neuroscience and Rheumatology, having authored 55 papers that have together received 1.9k indexed citations. Recurring topics across this work include Cerebrospinal fluid and hydrocephalus (18 papers), Fetal and Pediatric Neurological Disorders (10 papers), Intracranial Aneurysms: Treatment and Complications (9 papers), Moyamoya disease diagnosis and treatment (8 papers), Protease and Inhibitor Mechanisms (6 papers), Spinal Dysraphism and Malformations (6 papers), Vascular Malformations Diagnosis and Treatment (5 papers) and Glioma Diagnosis and Treatment (4 papers). The work is most often cited by research in Neurology (887 citations), Rheumatology (430 citations) and Cellular and Molecular Neuroscience (426 citations). Kazunari Oka has collaborated with scholars based in Japan, United States and Norway. Frequent co-authors include Masamichi Tomonaga, Masanori Yamashita, Kentaro Tanaka, K Tanaka, Masaaki Yamamoto, Margaret Barry, Albert L. Rhoton, Koichi Ikeda, Terushige Toyooka and Nobusuke Tsuzuki. Their work appears in journals such as Stroke, Journal of neurosurgery and British Journal of Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.