Kay Seidel

3.8k total citations
50 papers, 2.7k citations indexed

About

Kay Seidel is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Kay Seidel has authored 50 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Cellular and Molecular Neuroscience, 26 papers in Molecular Biology and 24 papers in Neurology. Recurrent topics in Kay Seidel's work include Genetic Neurodegenerative Diseases (31 papers), Mitochondrial Function and Pathology (23 papers) and Neurological disorders and treatments (18 papers). Kay Seidel is often cited by papers focused on Genetic Neurodegenerative Diseases (31 papers), Mitochondrial Function and Pathology (23 papers) and Neurological disorders and treatments (18 papers). Kay Seidel collaborates with scholars based in Germany, Netherlands and United States. Kay Seidel's co-authors include Udo Rüb, Wilfred F.A. den Dunnen, Horst‐Werner Korf, E. R. Brunt, Helmut Heinsen, Georg Auburger, Lüdger Schöls, Thomas Deller, Henry L. Paulson and Mohamed Bouzrou and has published in prestigious journals such as Journal of Biological Chemistry, Brain and Annals of Neurology.

In The Last Decade

Kay Seidel

50 papers receiving 2.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kay Seidel Germany 30 1.7k 1.5k 1.1k 392 350 50 2.7k
N. W. Kowall United States 16 2.2k 1.3× 1.6k 1.1× 909 0.8× 516 1.3× 248 0.7× 25 3.1k
Milena Cannella Italy 29 1.5k 0.9× 1.3k 0.9× 1.1k 1.0× 270 0.7× 177 0.5× 60 2.4k
Philippe Anglade France 16 1.2k 0.7× 766 0.5× 993 0.9× 386 1.0× 567 1.6× 39 2.6k
Cristina Missale Italy 27 1.1k 0.6× 860 0.6× 816 0.7× 399 1.0× 155 0.4× 74 2.4k
Albert Giralt Spain 31 1.6k 0.9× 1.7k 1.1× 672 0.6× 447 1.1× 377 1.1× 73 2.8k
J.-P. G. Vonsattel United States 16 3.2k 1.8× 2.6k 1.8× 2.0k 1.8× 486 1.2× 321 0.9× 22 4.4k
Christiane Penet France 17 1.5k 0.9× 1.7k 1.2× 769 0.7× 763 1.9× 295 0.8× 27 2.9k
Grazia Annesi Italy 27 1.2k 0.7× 1.1k 0.7× 1.2k 1.0× 263 0.7× 259 0.7× 94 2.7k
Umar Yazdani United States 18 884 0.5× 609 0.4× 466 0.4× 514 1.3× 211 0.6× 20 1.9k
Victoria Zhukareva United States 28 880 0.5× 1.0k 0.7× 1.1k 1.0× 1.4k 3.5× 620 1.8× 47 3.0k

Countries citing papers authored by Kay Seidel

Since Specialization
Citations

This map shows the geographic impact of Kay Seidel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kay Seidel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kay Seidel more than expected).

Fields of papers citing papers by Kay Seidel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kay Seidel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kay Seidel. The network helps show where Kay Seidel may publish in the future.

Co-authorship network of co-authors of Kay Seidel

This figure shows the co-authorship network connecting the top 25 collaborators of Kay Seidel. A scholar is included among the top collaborators of Kay Seidel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kay Seidel. Kay Seidel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Şen, Nesli-Ece, Júlia Canet-Pons, Melanie V. Halbach, et al.. (2019). Generation of an Atxn2-CAG100 knock-in mouse reveals N-acetylaspartate production deficit due to early Nat8l dysregulation. Neurobiology of Disease. 132. 104559–104559. 27 indexed citations
2.
3.
Stratmann, Katharina, Helmut Heinsen, Horst‐Werner Korf, et al.. (2015). Precortical Phase of Alzheimer's Disease (AD)‐Related Tau Cytoskeletal Pathology. Brain Pathology. 26(3). 371–386. 116 indexed citations
4.
Schöls, Lüdger, Matthias Reimold, Kay Seidel, et al.. (2015). No parkinsonism in SCA2 and SCA3 despite severe neurodegeneration of the dopaminergic substantia nigra. Brain. 138(11). 3316–3326. 47 indexed citations
5.
Seidel, Kay, Helmut Heinsen, Georg Auburger, et al.. (2014). The Brainstem Pathologies of Parkinson's Disease and Dementia with Lewy Bodies. Brain Pathology. 25(2). 121–135. 216 indexed citations
6.
Hoche, Franziska, Kay Seidel, Eduardo Barbosa‐Sicard, et al.. (2014). Cognitive Phenotype in Ataxia-Telangiectasia. Pediatric Neurology. 51(3). 297–310. 32 indexed citations
7.
Gispert, Suzana, Kay Seidel, Udo Rüb, et al.. (2014). Potentiation of neurotoxicity in double-mutant mice with Pink1 ablation and A53T-SNCA overexpression. Human Molecular Genetics. 24(4). 1061–1076. 50 indexed citations
8.
Seidel, Kay, et al.. (2012). Brain pathology of spinocerebellar ataxias. Acta Neuropathologica. 124(1). 1–21. 301 indexed citations
9.
Seidel, Kay, Melanie Meister, George J. Dugbartey, et al.. (2011). Cellular protein quality control and the evolution of aggregates in spinocerebellar ataxia type 3 (SCA3). Neuropathology and Applied Neurobiology. 38(6). 548–558. 39 indexed citations
10.
Brunt, E. R., Helmut Heinsen, Kay Seidel, et al.. (2011). Pathoanatomy of Cerebellar Degeneration in Spinocerebellar Ataxia Type 2 (SCA2) and Type 3 (SCA3). The Cerebellum. 11(3). 749–760. 77 indexed citations
11.
Carra, Serena, Alessandra Boncoraglio, Bart Kanon, et al.. (2010). Identification of the Drosophila Ortholog of HSPB8. Journal of Biological Chemistry. 285(48). 37811–37822. 74 indexed citations
12.
Seidel, Kay, et al.. (2009). The p62 antibody reveals various cytoplasmic protein aggregates in spinocerebellar ataxia Type 6. Clinical Neuropathology. 28(9). 344–349. 17 indexed citations
13.
Seidel, Kay, Peter Bauer, Wilfred F.A. den Dunnen, et al.. (2009). Widespread thalamic and cerebellar degeneration in a patient with a complicated hereditary spastic paraplegia (HSP). Annals of Anatomy - Anatomischer Anzeiger. 191(2). 203–211. 9 indexed citations
14.
Kern, A., Kay Seidel, & Helmut Oelschläger. (2009). The Central Vestibular Complex in Dolphins and Humans: Functional Implications of Deiters’ Nucleus. Brain Behavior and Evolution. 73(2). 102–110. 9 indexed citations
15.
Gierga, K., Helenius J. Schelhaas, E. R. Brunt, et al.. (2009). Spinocerebellar ataxia type 6 (SCA6): neurodegeneration goes beyond the known brain predilection sites. Neuropathology and Applied Neurobiology. 35(5). 515–527. 36 indexed citations
16.
Seidel, Kay, et al.. (2008). Review: Familial Parkinson's disease – genetics, clinical phenotype and neuropathology in relation to the common sporadic form of the disease. Neuropathology and Applied Neurobiology. 34(3). 255–271. 96 indexed citations
17.
Rüb, Udo, E. R. Brunt, Kay Seidel, et al.. (2007). Spinocerebellar ataxia type 7 (SCA7): widespread brain damage in an adult‐onset patient with progressive visual impairments in comparison with an adult‐onset patient without visual impairments. Neuropathology and Applied Neurobiology. 34(2). 155–168. 28 indexed citations
18.
Rüb, Udo, E. R. Brunt, Elisabeth Petrasch‐Parwez, et al.. (2006). Degeneration of ingestion‐related brainstem nuclei in spinocerebellar ataxia type 2, 3, 6 and 7. Neuropathology and Applied Neurobiology. 32(6). 635–649. 65 indexed citations
19.
Ateş, Mehmet, et al.. (2003). Intrathecally applied flurbiprofen produces an endocannabinoid‐dependent antinociception in the rat formalin test. European Journal of Neuroscience. 17(3). 597–604. 65 indexed citations
20.
Bürger, Marilise Escobar & Kay Seidel. (1958). [Chemical biomorphosis of the human brain and sciatic nerve; a survey].. PubMed. 12(1). 52–79. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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