Katsuichi Shuto
- Co-authors
- Nobuhiro NakamizoAkira KarasawaKazuhiro KuboAkio IshiiShunji IchikawaTakio KitazawaH. OBASEKenji Ohmori
- Topics
- Phenothiazines and Benzothiazines Synthesis and Activities (10 papers)Synthesis and biological activity (9 papers)Pharmacological Effects of Natural Compounds (9 papers)
- Journals
- Journal of Medicinal ChemistryBritish Journal of PharmacologyBioorganic & Medicinal Chemistry
- Partner nations
- JapanSingaporeUnited States
In The Last Decade
Katsuichi Shuto
84 papers receiving 574 citations
Peers
Comparison fields: 5 of 90
- Molecular Biology 246
- Organic Chemistry 128
- Physiology 119
- Cellular and Molecular Neuroscience 100
- Pharmacology 78
Countries citing papers authored by Katsuichi Shuto
This map shows the geographic impact of Katsuichi Shuto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katsuichi Shuto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katsuichi Shuto more than expected).
Fields of papers citing papers by Katsuichi Shuto
This network shows the impact of papers produced by Katsuichi Shuto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katsuichi Shuto. The network helps show where Katsuichi Shuto may publish in the future.
Co-authorship network of co-authors of Katsuichi Shuto
This figure shows the co-authorship network connecting the top 25 collaborators of Katsuichi Shuto. A scholar is included among the top collaborators of Katsuichi Shuto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katsuichi Shuto. Katsuichi Shuto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 42 | |
| 3 | 1 | |
| 4 | 14 | |
| 5 | 2 | |
| 6 | 29 | |
| 7 | 10 | |
| 8 | 15 | |
| 9 | 2 | |
| 10 | Vasodilating effects of the new calcium antagonist benidipine hydrochloride in anesthetized dogs and cats. | 7 |
| 11 | 1 | |
| 12 | 2 | |
| 13 | 1 | |
| 14 | 7 | |
| 15 | 2 | |
| 16 | 8 | |
| 17 | 4 | |
| 18 | 3 | |
| 19 | 6 | |
| 20 | 7 |
About Katsuichi Shuto
Katsuichi Shuto is a scholar working on Pharmacology, Pharmacology and Gastroenterology, having authored 88 papers that have together received 639 indexed citations. Recurring topics across this work include Phenothiazines and Benzothiazines Synthesis and Activities (10 papers), Synthesis and biological activity (9 papers) and Pharmacological Effects of Natural Compounds (9 papers). The work is most often cited by research in Gastroenterology (41 citations), Cellular and Molecular Neuroscience (100 citations) and Pharmacology (45 citations). Katsuichi Shuto has collaborated with scholars based in Japan, Singapore and United States. Frequent co-authors include Nobuhiro Nakamizo, Akira Karasawa, Kazuhiro Kubo, Akio Ishii, Shunji Ichikawa, Takio Kitazawa, H. OBASE, Kenji Ohmori, Masamichi Tsuboi and Katsuyoshi Kubo. Their work appears in journals such as Journal of Medicinal Chemistry, British Journal of Pharmacology and Bioorganic & Medicinal Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.