Katja Prystaz

505 total citations
9 papers, 364 citations indexed

About

Katja Prystaz is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Katja Prystaz has authored 9 papers receiving a total of 364 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 2 papers in Oncology and 2 papers in Epidemiology. Recurrent topics in Katja Prystaz's work include Bone Metabolism and Diseases (4 papers), S100 Proteins and Annexins (3 papers) and Bone fractures and treatments (2 papers). Katja Prystaz is often cited by papers focused on Bone Metabolism and Diseases (4 papers), S100 Proteins and Annexins (3 papers) and Bone fractures and treatments (2 papers). Katja Prystaz collaborates with scholars based in Germany and United States. Katja Prystaz's co-authors include Anita Ignatius, Melanie Haffner‐Luntzer, Verena Fischer, Astrid Liedert, Georg H. Waetzig, Stefan Rose‐John, Gudrun Strauß, Anna Kovtun, Anna E. Rapp and Michael Amling and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Scientific Reports.

In The Last Decade

Katja Prystaz

8 papers receiving 352 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Katja Prystaz Germany	7	149	86	79	70	62	9	364
Nada Tomanović Serbia	13	76 0.5×	40 0.5×	102 1.3×	30 0.4×	126 2.0×	43	472
K. L. Schmidt Germany	11	61 0.4×	51 0.6×	65 0.8×	110 1.6×	68 1.1×	22	469
Tadashi Moriwake Japan	12	166 1.1×	90 1.0×	24 0.3×	75 1.1×	87 1.4×	24	758
Elisabet Bucht Sweden	12	119 0.8×	66 0.8×	29 0.4×	13 0.2×	80 1.3×	21	420
Zheng Lv China	10	85 0.6×	12 0.1×	72 0.9×	60 0.9×	89 1.4×	17	496
Yoshinori Nezu Japan	14	62 0.4×	42 0.5×	44 0.6×	32 0.5×	182 2.9×	42	511
Giuseppa Patti Italy	16	206 1.4×	25 0.3×	47 0.6×	26 0.4×	97 1.6×	51	609
Elisabeth Karsten Australia	9	66 0.4×	12 0.1×	54 0.7×	54 0.8×	63 1.0×	13	427
Maxine Daniels United Kingdom	6	119 0.8×	221 2.6×	15 0.2×	43 0.6×	89 1.4×	9	500
Xin Pan China	11	65 0.4×	20 0.2×	34 0.4×	50 0.7×	136 2.2×	34	366

Countries citing papers authored by Katja Prystaz

Since Specialization

Citations

This map shows the geographic impact of Katja Prystaz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katja Prystaz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katja Prystaz more than expected).

Fields of papers citing papers by Katja Prystaz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katja Prystaz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katja Prystaz. The network helps show where Katja Prystaz may publish in the future.

Co-authorship network of co-authors of Katja Prystaz

This figure shows the co-authorship network connecting the top 25 collaborators of Katja Prystaz. A scholar is included among the top collaborators of Katja Prystaz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katja Prystaz. Katja Prystaz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Haffner‐Luntzer, Melanie, Sandra Foertsch, Verena Fischer, et al.. (2019). Chronic psychosocial stress compromises the immune response and endochondral ossification during bone fracture healing via β-AR signaling. Proceedings of the National Academy of Sciences. 116(17). 8615–8622. 66 indexed citations

2.

Prystaz, Katja, Melanie Haffner‐Luntzer, Stephanie Bergdolt, et al.. (2018). Pharmacological inhibition of IL-6 trans-signaling improves compromised fracture healing after severe trauma. Naunyn-Schmiedeberg s Archives of Pharmacology. 391(5). 523–536. 54 indexed citations

3.

Kovtun, Anna, Katja Prystaz, Melanie Haffner‐Luntzer, et al.. (2018). INHIBITION OF INTERLEUKIN-6 TRANS-SIGNALLING IMPROVES FRACTURE HEALING AFTER TRAUMA. 1 indexed citations

4.

Prystaz, Katja, Anna Kovtun, Melanie Haffner‐Luntzer, et al.. (2017). Distinct Effects of IL-6 Classic and Trans -Signaling in Bone Fracture Healing. American Journal Of Pathology. 188(2). 474–490. 99 indexed citations

5.

Fischer, Verena, Melanie Haffner‐Luntzer, Katja Prystaz, et al.. (2017). Calcium and vitamin-D deficiency marginally impairs fracture healing but aggravates posttraumatic bone loss in osteoporotic mice. Scientific Reports. 7(1). 7223–7223. 52 indexed citations

6.

Haffner‐Luntzer, Melanie, Verena Fischer, Katja Prystaz, Astrid Liedert, & Anita Ignatius. (2017). The inflammatory phase of fracture healing is influenced by oestrogen status in mice. European journal of medical research. 22(1). 23–23. 50 indexed citations

7.

Haffner‐Luntzer, Melanie, Anna Kovtun, Verena Fischer, et al.. (2017). Loss of p53 compensates osteopenia in murine Mysml deficiency. The FASEB Journal. 32(4). 1957–1968. 21 indexed citations

8.

Haffner‐Luntzer, Melanie, Katja Prystaz, Thorsten Schinke, et al.. (2016). Inhibition of Midkine Augments Osteoporotic Fracture Healing. PLoS ONE. 11(7). e0159278–e0159278. 21 indexed citations

9.

Haffner‐Luntzer, Melanie, et al.. (2016). Calcium and vitamin-D supplementation post-trauma improves bone healing and decreases posttraumatic bone resorption in an osteoporotic mouse model. Bone Abstracts.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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