Kathryn M Guinta

2.9k total citations
16 papers, 577 citations indexed

About

Kathryn M Guinta is a scholar working on Hematology, Molecular Biology and Oncology. According to data from OpenAlex, Kathryn M Guinta has authored 16 papers receiving a total of 577 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Hematology, 5 papers in Molecular Biology and 4 papers in Oncology. Recurrent topics in Kathryn M Guinta's work include Acute Myeloid Leukemia Research (10 papers), Pancreatic and Hepatic Oncology Research (3 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (3 papers). Kathryn M Guinta is often cited by papers focused on Acute Myeloid Leukemia Research (10 papers), Pancreatic and Hepatic Oncology Research (3 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (3 papers). Kathryn M Guinta collaborates with scholars based in United States, Japan and India. Kathryn M Guinta's co-authors include Jaroslaw P. Maciejewski, Mikkael A. Sekeres, Andrés Jerez, Bartlomiej Przychodzen, Hideki Makishima, Richard A. Padgett, Manuel Afable, Ramón V. Tiu, Sarah Abu Kar and Hirotoshi Sakaguchi and has published in prestigious journals such as Journal of Clinical Oncology, Blood and PLoS ONE.

In The Last Decade

Kathryn M Guinta

15 papers receiving 571 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kathryn M Guinta United States 7 390 370 171 51 51 16 577
Ania Jankowska United States 7 268 0.7× 369 1.0× 160 0.9× 39 0.8× 53 1.0× 10 491
Courtney Prince United States 7 299 0.8× 393 1.1× 232 1.4× 68 1.3× 44 0.9× 16 608
Mandy Brecqueville France 9 321 0.8× 455 1.2× 317 1.9× 35 0.7× 63 1.2× 9 583
M Berends Netherlands 3 416 1.1× 329 0.9× 161 0.9× 33 0.6× 90 1.8× 4 578
Martin Wichmann Germany 6 304 0.8× 502 1.4× 237 1.4× 32 0.6× 82 1.6× 14 589
Costanza Bosi Italy 13 281 0.7× 385 1.0× 210 1.2× 44 0.9× 30 0.6× 25 630
Kim-Hien T. Dao United States 10 309 0.8× 303 0.8× 190 1.1× 50 1.0× 52 1.0× 13 566
Ana Valencia Spain 12 403 1.0× 311 0.8× 79 0.5× 39 0.8× 41 0.8× 21 528
Mar Mallo Spain 11 235 0.6× 313 0.8× 137 0.8× 30 0.6× 112 2.2× 29 505
Thomas McKerrell United Kingdom 7 228 0.6× 417 1.1× 270 1.6× 51 1.0× 135 2.6× 8 532

Countries citing papers authored by Kathryn M Guinta

Since Specialization
Citations

This map shows the geographic impact of Kathryn M Guinta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kathryn M Guinta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kathryn M Guinta more than expected).

Fields of papers citing papers by Kathryn M Guinta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kathryn M Guinta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kathryn M Guinta. The network helps show where Kathryn M Guinta may publish in the future.

Co-authorship network of co-authors of Kathryn M Guinta

This figure shows the co-authorship network connecting the top 25 collaborators of Kathryn M Guinta. A scholar is included among the top collaborators of Kathryn M Guinta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kathryn M Guinta. Kathryn M Guinta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Padmanabhan, Roshan, Kathryn M Guinta, Tyler Stevens, et al.. (2023). Microbiomic profiles of bile in patients with benign and malignant pancreaticobiliary disease. PLoS ONE. 18(4). e0283021–e0283021. 14 indexed citations
2.
Padmanabhan, Roshan, Kathryn M Guinta, Tyler Stevens, et al.. (2021). Microbiomic profiles of bile in patients with benign and malignant pancreaticobiliary disease.. Journal of Clinical Oncology. 39(3_suppl). 417–417. 2 indexed citations
3.
Padmanabhan, Roshan, Prabhleen Chahal, Madhusudhan R. Sanaka, et al.. (2019). Microbiome signature of bile from pancreatic and biliary tract cancer patients: A pilot study.. Journal of Clinical Oncology. 37(15_suppl). e15744–e15744. 3 indexed citations
4.
Burger, Denis R., Yvonne Parker, Kathryn M Guinta, & Daniel J. Lindner. (2017). PRO 140 Monoclonal Antibody to CCR5 Prevents Acute Xenogeneic Graft-versus-Host Disease in NOD-scid IL-2Rynull Mice. Biology of Blood and Marrow Transplantation. 24(2). 260–266. 12 indexed citations
5.
Thota, Swapna, Paul C. Lakin, Holleh Husseinzadeh, et al.. (2013). Somatic Mutational Screen For Improved Prediction Of The Outcomes Of Epigenetic Therapy In MDS. Blood. 122(21). 659–659. 1 indexed citations
6.
Thota, Swapna, Sarah McMahon, Hideki Makishima, et al.. (2013). Germline Events In GFI1 In Myelodysplastic Syndrome. Blood. 122(21). 1231–1231. 1 indexed citations
7.
Przychodzen, Bartlomiej, Andrés Jerez, Kathryn M Guinta, et al.. (2013). Patterns of missplicing due to somatic U2AF1 mutations in myeloid neoplasms. Blood. 122(6). 999–1006. 117 indexed citations
8.
Sugimoto, Yuka, Mikkael A. Sekeres, Hideki Makishima, et al.. (2012). Cytogenetic and molecular predictors of response in patients with myeloid malignancies without del[5q] treated with lenalidomide. Journal of Hematology & Oncology. 5(1). 4–4. 18 indexed citations
9.
Hosono, Naoko, Hasan Rehman, Bartlomiej Przychodzen, et al.. (2012). Various Germline Congenital Disorder Genes Are Somatically Mutated in Myeloid Malignancies. Blood. 120(21). 1405–1405.
10.
Kar, Sarah Abu, Ania Jankowska, Hirokazu Makishima, et al.. (2012). Spliceosomal gene mutations are frequent events in the diverse mutational spectrum of chronic myelomonocytic leukemia but largely absent in juvenile myelomonocytic leukemia. Haematologica. 98(1). 107–113. 61 indexed citations
11.
Makishima, Hideki, Valeria Visconte, Hirotoshi Sakaguchi, et al.. (2012). Mutations in the spliceosome machinery, a novel and ubiquitous pathway in leukemogenesis. Blood. 119(14). 3203–3210. 279 indexed citations
12.
Negrotto, Soledad, Kwok Peng Ng, Anna Jankowska, et al.. (2011). CpG methylation patterns and decitabine treatment response in acute myeloid leukemia cells and normal hematopoietic precursors. Leukemia. 26(2). 244–254. 64 indexed citations
13.
Makishima, Hideki, Hirotoshi Sakaguchi, Valeria Visconte, et al.. (2011). Acquired Molecular Defects in Spliceosome Machinery: Novel Pathogenetic Pathways in Myeloid Leukemogenesis. Blood. 118(21). 271–271. 1 indexed citations
14.
Sugimoto, Yuka, Mikkael A. Sekeres, Hideki Makishima, et al.. (2010). Cytogenetic Predictors of Response to Lenalidomide In Myeloid Malignancies without Del(5q). Blood. 116(21). 4016–4016. 2 indexed citations
15.
Traina, Fabı́ola, Ania Jankowska, Hideki Makishima, et al.. (2010). New TET2, ASXL1 and CBL Mutations Have Poor Prognostic Impact In Systemic Mastocytosis and Related Disorders. Blood. 116(21). 3076–3076. 1 indexed citations
16.
Makishima, Hideki, Ania Jankowska, Ramón V. Tiu, et al.. (2010). Identification of Oncogenic EZH2 Mutations In Myelodysplastic Syndromes and Related Myeloid Malignancies. Blood. 116(21). 607–607. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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