Kate Sleeth

567 total citations
9 papers, 440 citations indexed

About

Kate Sleeth is a scholar working on Molecular Biology, Oncology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Kate Sleeth has authored 9 papers receiving a total of 440 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Oncology and 1 paper in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Kate Sleeth's work include DNA Repair Mechanisms (7 papers), PARP inhibition in cancer therapy (3 papers) and Cancer therapeutics and mechanisms (2 papers). Kate Sleeth is often cited by papers focused on DNA Repair Mechanisms (7 papers), PARP inhibition in cancer therapy (3 papers) and Cancer therapeutics and mechanisms (2 papers). Kate Sleeth collaborates with scholars based in United Kingdom, Sweden and Netherlands. Kate Sleeth's co-authors include Thomas Helleday, Natalia Issaeva, Grigory L. Dianov, Ponnari Gottipati, Ricky A. Sharma, Tatjana Djureinovic, Joyce Solomons, Barbara Vischioni, Niklas Schultz and Helen E. Bryant and has published in prestigious journals such as Journal of Molecular Biology, Biochemistry and Cancer Research.

In The Last Decade

Kate Sleeth

9 papers receiving 438 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kate Sleeth United Kingdom 6 372 265 68 54 32 9 440
Cecilia Ström Sweden 10 418 1.1× 315 1.2× 42 0.6× 44 0.8× 24 0.8× 11 512
Ann Christin Parplys Germany 11 343 0.9× 167 0.6× 81 1.2× 59 1.1× 31 1.0× 18 427
Brianna Monroe United States 4 519 1.4× 176 0.7× 135 2.0× 36 0.7× 26 0.8× 4 606
Niraj Joshi Canada 6 621 1.7× 271 1.0× 100 1.5× 123 2.3× 22 0.7× 7 695
Camille Gelot France 8 442 1.2× 212 0.8× 56 0.8× 44 0.8× 16 0.5× 12 515
Jan Tkáč Canada 3 751 2.0× 338 1.3× 102 1.5× 84 1.6× 47 1.5× 3 818
Marco Barazas Netherlands 6 270 0.7× 213 0.8× 47 0.7× 33 0.6× 27 0.8× 7 361
Natsuko Suwaki United Kingdom 6 280 0.8× 117 0.4× 56 0.8× 38 0.7× 15 0.5× 7 347
Marie K. Classon Canada 2 295 0.8× 238 0.9× 93 1.4× 36 0.7× 36 1.1× 2 401
Benjamin Primack United States 5 654 1.8× 387 1.5× 97 1.4× 97 1.8× 28 0.9× 10 748

Countries citing papers authored by Kate Sleeth

Since Specialization
Citations

This map shows the geographic impact of Kate Sleeth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate Sleeth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate Sleeth more than expected).

Fields of papers citing papers by Kate Sleeth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate Sleeth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate Sleeth. The network helps show where Kate Sleeth may publish in the future.

Co-authorship network of co-authors of Kate Sleeth

This figure shows the co-authorship network connecting the top 25 collaborators of Kate Sleeth. A scholar is included among the top collaborators of Kate Sleeth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kate Sleeth. Kate Sleeth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Cornelissen, Bart, Veerle Kersemans, James Thompson, et al.. (2011). Imaging DNA Damage In Vivo Using γH2AX-Targeted Immunoconjugates. Cancer Research. 71(13). 4539–4549. 57 indexed citations
2.
Issaeva, Natalia, Huw D. Thomas, Janneke E. Jaspers, et al.. (2010). 6-Thioguanine Selectively Kills BRCA2-Defective Tumors and Overcomes PARP Inhibitor Resistance. Cancer Research. 70(15). 6268–6276. 80 indexed citations
3.
Gottipati, Ponnari, Barbara Vischioni, Niklas Schultz, et al.. (2010). Poly(ADP-Ribose) Polymerase Is Hyperactivated in Homologous Recombination–Defective Cells. Cancer Research. 70(13). 5389–5398. 167 indexed citations
4.
Cornelissen, Bart, et al.. (2009). Amplification of IR-induced DNA damage by Auger electron treatment with TAT-radioimmunoconjugates. 50. 638. 2 indexed citations
5.
Cornelissen, Bart, et al.. (2009). Imaging of DNA double strand breaks in vivo using fluorophore-labelled TAT-immunoconjugates. 50. 1015–1015. 1 indexed citations
6.
Sleeth, Kate, Claus Storgaard Sørensen, Natalia Issaeva, et al.. (2007). RPA Mediates Recombination Repair During Replication Stress and Is Displaced from DNA by Checkpoint Signalling in Human Cells. Journal of Molecular Biology. 373(1). 38–47. 55 indexed citations
7.
Sleeth, Kate, Robert L. Robson, & Grigory L. Dianov. (2004). Exchangeability of Mammalian DNA Ligases between Base Excision Repair Pathways. Biochemistry. 43(40). 12924–12930. 46 indexed citations
8.
Allinson, Sarah L., et al.. (2003). Orchestration of base excision repair by controlling the rates of enzymatic activities. DNA repair. 3(1). 23–31. 31 indexed citations
9.
Dianov, Grigory L., Sarah L. Allinson, Helen Budworth, & Kate Sleeth. (2003). Mammalian base excision repair.. Lancaster EPrints (Lancaster University). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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