Kate E. Brilliant

563 total citations
22 papers, 421 citations indexed

About

Kate E. Brilliant is a scholar working on Molecular Biology, Hepatology and Oncology. According to data from OpenAlex, Kate E. Brilliant has authored 22 papers receiving a total of 421 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 8 papers in Hepatology and 7 papers in Oncology. Recurrent topics in Kate E. Brilliant's work include Liver physiology and pathology (5 papers), Extracellular vesicles in disease (4 papers) and Cancer Mechanisms and Therapy (3 papers). Kate E. Brilliant is often cited by papers focused on Liver physiology and pathology (5 papers), Extracellular vesicles in disease (4 papers) and Cancer Mechanisms and Therapy (3 papers). Kate E. Brilliant collaborates with scholars based in United States, Croatia and Italy. Kate E. Brilliant's co-authors include Douglas C. Hixson, Peter J. Quesenberry, Bharat Ramratnam, Mark S. Dooner, Jason M. Aliotta, Mandy Pereira, Djuro Josić, David Lee, Paul N. McMillan and Kevin W. Johnson and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Oncology and PLoS ONE.

In The Last Decade

Kate E. Brilliant

21 papers receiving 410 citations

Peers

Kate E. Brilliant
Zhihua Li China
Shrabasti Roychoudhury United States
Kehinde Adekola United States
Rakad M. Kh. Al-Jumaily Iraq
Galina Bogatcheva United States
François Béliveau Canada
Amin Yaqubie United States
Ranjan Prasad Devbhandari China
Margit Stimpfl Austria
Zhihua Li China
Kate E. Brilliant
Citations per year, relative to Kate E. Brilliant Kate E. Brilliant (= 1×) peers Zhihua Li

Countries citing papers authored by Kate E. Brilliant

Since Specialization
Citations

This map shows the geographic impact of Kate E. Brilliant's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate E. Brilliant with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate E. Brilliant more than expected).

Fields of papers citing papers by Kate E. Brilliant

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate E. Brilliant. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate E. Brilliant. The network helps show where Kate E. Brilliant may publish in the future.

Co-authorship network of co-authors of Kate E. Brilliant

This figure shows the co-authorship network connecting the top 25 collaborators of Kate E. Brilliant. A scholar is included among the top collaborators of Kate E. Brilliant based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kate E. Brilliant. Kate E. Brilliant is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gajdošik, Martina Šrajer, Douglas C. Hixson, Kate E. Brilliant, et al.. (2018). Soft agar-based selection of spontaneously transformed rat prostate epithelial cells with highly tumorigenic characteristics. Experimental and Molecular Pathology. 105(1). 89–97.
2.
Ahsan, Nagib, Stephanie Post, Kate E. Brilliant, et al.. (2017). Proteomic analysis of laser capture microdissected focal lesions in a rat model of progenitor marker-positive hepatocellular carcinoma. Oncotarget. 8(16). 26041–26056. 10 indexed citations
3.
Brilliant, Kate E., et al.. (2016). Persistent effect of mTOR inhibition on preneoplastic foci progression and gene expression in a rat model of hepatocellular carcinoma. Carcinogenesis. 37(4). 408–419. 8 indexed citations
4.
Perez, Kimberly, Kate E. Brilliant, Evgeny Yakirevich, et al.. (2013). Heterogeneity of colorectal cancer (CRC) in reference to KRAS proto-oncogene utilizing wave technology.. Journal of Clinical Oncology. 31(15_suppl). e14637–e14637. 1 indexed citations
5.
Perez, Kimberly, Kate E. Brilliant, Lelia Noble, et al.. (2013). Heterogeneity of colorectal cancer (CRC) in reference to KRAS proto-oncogene utilizing WAVE technology. Experimental and Molecular Pathology. 95(1). 74–82. 13 indexed citations
6.
Lu, Shaolei, Kimberly Perez, Sara Guevara, et al.. (2013). RKIP phosphorylation and STAT3 activation is inhibited by oxaliplatin and camptothecin and are associated with poor prognosis in stage II colon cancer patients. BMC Cancer. 13(1). 463–463. 46 indexed citations
7.
Walsh, Edward G., et al.. (2013). MRI contrast demonstration of antigen-specific targeting with an iron-based ferritin construct. Journal of Nanoparticle Research. 15(1). 5 indexed citations
8.
Aliotta, Jason M., Mandy Pereira, Ming Li, et al.. (2012). Stable cell fate changes in marrow cells induced by lung‐derived microvesicles. Journal of Extracellular Vesicles. 1(1). 41 indexed citations
9.
Sanders, Jennifer A., et al.. (2012). The inhibitory effect of rapamycin on the oval cell response and development of preneoplastic foci in the rat. Experimental and Molecular Pathology. 93(1). 40–49. 9 indexed citations
10.
Mills, David, et al.. (2012). The cholangiocyte marker, BD. 1, forms a stable complex with CLIP170 and shares an identity with eIF3a, a multifunctional subunit of the eIF3 initiation complex. Experimental and Molecular Pathology. 93(2). 250–260. 2 indexed citations
11.
Brilliant, Kate E., et al.. (2012). The Transmembrane Domain of CEACAM1-4S Is a Determinant of Anchorage Independent Growth and Tumorigenicity. PLoS ONE. 7(1). e29606–e29606. 13 indexed citations
12.
Moen, Erika L., Sicheng Wen, Talha Anwar, et al.. (2012). Regulation of RKIP Function by Helicobacter pylori in Gastric Cancer. PLoS ONE. 7(5). e37819–e37819. 16 indexed citations
13.
Aliotta, Jason M., Mandy Pereira, Kevin W. Johnson, et al.. (2010). Microvesicle entry into marrow cells mediates tissue-specific changes in mRNA by direct delivery of mRNA and induction of transcription. Experimental Hematology. 38(3). 233–245. 173 indexed citations
14.
Brilliant, Kate E., et al.. (2010). Accumulation of neoplastic traits prior to spontaneous in vitro transformation of rat cholangiocytes determines susceptibility to activated ErbB-2/Neu. Experimental and Molecular Pathology. 89(3). 248–259. 6 indexed citations
15.
Clifton, James R, Feilei Huang, Dajana Gašo-Sokač, et al.. (2009). Use of proteomics for validation of the isolation process of clotting factor IX from human plasma. Journal of Proteomics. 73(3). 678–688. 13 indexed citations
16.
17.
Mills, David, Helen Callanan, Donna Flanagan, et al.. (2009). Monoclonal antibody to novel cell surface epitope on Hsc70 promotes morphogenesis of bile ducts in newborn rat liver. Cell Stress and Chaperones. 15(1). 39–53. 9 indexed citations
18.
19.
Laurie, Nikia A., et al.. (2005). Carcinoembryonic Antigen–Related Cell Adhesion Molecule 1a-4L Suppression of Rat Hepatocellular Carcinomas. Cancer Research. 65(23). 11010–11017. 18 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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