Kai‐Kai Chang
- Immunology top 5%
- Reproductive Medicine top 0.5%
- Obstetrics and Gynecology top 1%
- Molecular Biology
- Cancer Research top 10%
- Topics
- Endometriosis Research and Treatment (25 papers)Reproductive System and Pregnancy (21 papers)Endometrial and Cervical Cancer Treatments (8 papers)
- Partner nations
- ChinaUnited StatesSingapore
In The Last Decade
Kai‐Kai Chang
42 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 91
- Immunology 902
- Reproductive Medicine 815
- Obstetrics and Gynecology 624
- Molecular Biology 309
- Cancer Research 177
Countries citing papers authored by Kai‐Kai Chang
This map shows the geographic impact of Kai‐Kai Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kai‐Kai Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kai‐Kai Chang more than expected).
Fields of papers citing papers by Kai‐Kai Chang
This network shows the impact of papers produced by Kai‐Kai Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kai‐Kai Chang. The network helps show where Kai‐Kai Chang may publish in the future.
Co-authorship network of co-authors of Kai‐Kai Chang
This figure shows the co-authorship network connecting the top 25 collaborators of Kai‐Kai Chang. A scholar is included among the top collaborators of Kai‐Kai Chang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kai‐Kai Chang. Kai‐Kai Chang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 13 | |
| 4 | 39 | |
| 5 | 26 | |
| 6 | 28 | |
| 7 | 35 | |
| 8 | 102 | |
| 9 | 35 | |
| 10 | 27 | |
| 11 | 115 | |
| 12 | 21 | |
| 13 | 97 | |
| 14 | 66 | |
| 15 | Autophagy in endometriosis. | 55 |
| 16 | 81 | |
| 17 | TSLP induced by estrogen stimulates secretion of MCP-1 and IL-8 and growth of human endometrial stromal cells through JNK and NF-κB signal pathways. | 23 |
| 18 | 43 | |
| 19 | 14 | |
| 20 | IL-22 in the endometriotic milieu promotes the proliferation of endometrial stromal cells via stimulating the secretion of CCL2 and IL-8. | 33 |
About Kai‐Kai Chang
Kai‐Kai Chang is a scholar working on Reproductive Medicine, Obstetrics and Gynecology and Immunology, having authored 43 papers that have together received 1.5k indexed citations. Recurring topics across this work include Endometriosis Research and Treatment (25 papers), Reproductive System and Pregnancy (21 papers) and Endometrial and Cervical Cancer Treatments (8 papers). The work is most often cited by research in Reproductive Medicine (815 citations), Obstetrics and Gynecology (624 citations) and Immunology (902 citations). Kai‐Kai Chang has collaborated with scholars based in China, United States and Singapore. Frequent co-authors include Ming‐Qing Li, Jie Mei, Da‐Jin Li, Wen‐Jie Zhou, Hui‐Li Yang, Jun Shao, Libing Liu, Yuhan Meng, Feng Xie and Xiaofang Yi. Their work appears in journals such as Science Advances, Cellular and Molecular Life Sciences and Frontiers in Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.