Kai Y. Soo

7.4k total citations · 1 hit paper
15 papers, 1.1k citations indexed

About

Kai Y. Soo is a scholar working on Neurology, Genetics and Cell Biology. According to data from OpenAlex, Kai Y. Soo has authored 15 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Neurology, 5 papers in Genetics and 5 papers in Cell Biology. Recurrent topics in Kai Y. Soo's work include Amyotrophic Lateral Sclerosis Research (13 papers), Parkinson's Disease Mechanisms and Treatments (8 papers) and Neurogenetic and Muscular Disorders Research (5 papers). Kai Y. Soo is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (13 papers), Parkinson's Disease Mechanisms and Treatments (8 papers) and Neurogenetic and Muscular Disorders Research (5 papers). Kai Y. Soo collaborates with scholars based in Australia, United States and Qatar. Kai Y. Soo's co-authors include Julie D. Atkin, Manal A. Farg, Vinod Sundaramoorthy, Ian P. Blair, Mark A. Halloran, Anna E. King, Adam K. Walker, Vita Levina, Malcolm Horne and Sonam Parakh and has published in prestigious journals such as PLoS ONE, International Journal of Molecular Sciences and Journal of Neurochemistry.

In The Last Decade

Kai Y. Soo

15 papers receiving 1.1k citations

Hit Papers

C9ORF72, implicated in amytrophic lateral sclerosis and f... 2014 2026 2018 2022 2014 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kai Y. Soo Australia 12 788 463 362 255 192 15 1.1k
Manal A. Farg Australia 13 996 1.3× 548 1.2× 429 1.2× 398 1.6× 243 1.3× 14 1.5k
Elisa Onesto Italy 17 715 0.9× 819 1.8× 411 1.1× 208 0.8× 337 1.8× 18 1.3k
Elizabeth L. Tudor United Kingdom 7 593 0.8× 752 1.6× 311 0.9× 296 1.2× 239 1.2× 7 1.2k
Anna‐Lena Ström Sweden 15 503 0.6× 540 1.2× 172 0.5× 204 0.8× 250 1.3× 21 1.1k
Vinay K. Godena United Kingdom 9 543 0.7× 452 1.0× 244 0.7× 117 0.5× 171 0.9× 9 886
Anna M. Blokhuis Netherlands 7 655 0.8× 451 1.0× 412 1.1× 98 0.4× 151 0.8× 7 929
D. Sau Italy 12 441 0.6× 630 1.4× 227 0.6× 223 0.9× 315 1.6× 15 1.2k
Max Koppers Netherlands 15 921 1.2× 1.0k 2.2× 612 1.7× 293 1.1× 332 1.7× 23 1.8k
Sadaf T. Warraich Australia 12 629 0.8× 375 0.8× 322 0.9× 129 0.5× 131 0.7× 12 825
Nicholas J. Kramer United States 11 688 0.9× 715 1.5× 307 0.8× 161 0.6× 264 1.4× 13 1.2k

Countries citing papers authored by Kai Y. Soo

Since Specialization
Citations

This map shows the geographic impact of Kai Y. Soo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kai Y. Soo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kai Y. Soo more than expected).

Fields of papers citing papers by Kai Y. Soo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kai Y. Soo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kai Y. Soo. The network helps show where Kai Y. Soo may publish in the future.

Co-authorship network of co-authors of Kai Y. Soo

This figure shows the co-authorship network connecting the top 25 collaborators of Kai Y. Soo. A scholar is included among the top collaborators of Kai Y. Soo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kai Y. Soo. Kai Y. Soo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Ragagnin, Audrey, Sonam Parakh, Kai Y. Soo, et al.. (2024). C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTD. Molecular Neurobiology. 61(12). 10318–10338. 4 indexed citations
2.
Furness, John B., Kara L. Britt, Kai Y. Soo, et al.. (2020). Three-dimensional growth of breast cancer cells potentiates the anti-tumor effects of unacylated ghrelin and AZP-531. eLife. 9. 7 indexed citations
3.
Farg, Manal A., Anna Konopka, Kai Y. Soo, Daisuke Ito, & Julie D. Atkin. (2017). The DNA damage response (DDR) is induced by the C9orf72 repeat expansion in amyotrophic lateral sclerosis. Human Molecular Genetics. 26(15). 2882–2896. 120 indexed citations
4.
Soo, Kai Y. & Julie D. Atkin. (2015). Autophagy dysregulation by mutant fused in sarcoma—implications for amyotrophic lateral sclerosis. Cell Death and Disease. 6(10). e1945–e1945. 6 indexed citations
5.
Soo, Kai Y., Mark A. Halloran, Vinod Sundaramoorthy, et al.. (2015). Rab1-dependent ER–Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS. Acta Neuropathologica. 130(5). 679–697. 89 indexed citations
6.
Farg, Manal A., Vinod Sundaramoorthy, Shu Yang, et al.. (2014). C9ORF72, implicated in amytrophic lateral sclerosis and frontotemporal dementia, regulates endosomal trafficking. Human Molecular Genetics. 23(13). 3579–3595. 377 indexed citations breakdown →
7.
Walker, Adam K., Kai Y. Soo, Vinod Sundaramoorthy, et al.. (2013). ALS-Associated TDP-43 Induces Endoplasmic Reticulum Stress, Which Drives Cytoplasmic TDP-43 Accumulation and Stress Granule Formation. PLoS ONE. 8(11). e81170–e81170. 140 indexed citations
8.
Sundaramoorthy, Vinod, Adam K. Walker, Justin J. Yerbury, et al.. (2013). Extracellular wildtype and mutant SOD1 induces ER–Golgi pathology characteristic of amyotrophic lateral sclerosis in neuronal cells. Cellular and Molecular Life Sciences. 70(21). 4181–4195. 52 indexed citations
9.
Parakh, Sonam, Damian M. S. Spencer, Mark A. Halloran, Kai Y. Soo, & Julie D. Atkin. (2013). Redox Regulation in Amyotrophic Lateral Sclerosis. Oxidative Medicine and Cellular Longevity. 2013. 1–12. 66 indexed citations
10.
Soo, Kai Y., Julie D. Atkin, Manal A. Farg, et al.. (2012). Bim Links ER Stress and Apoptosis in Cells Expressing Mutant SOD1 Associated with Amyotrophic Lateral Sclerosis. PLoS ONE. 7(4). e35413–e35413. 34 indexed citations
11.
Farg, Manal A., Kai Y. Soo, Sadaf T. Warraich, et al.. (2012). Ataxin-2 interacts with FUS and intermediate-length polyglutamine expansions enhance FUS-related pathology in amyotrophic lateral sclerosis. Human Molecular Genetics. 22(4). 717–728. 83 indexed citations
12.
Farg, Manal A., Kai Y. Soo, Adam K. Walker, et al.. (2012). Mutant FUS induces endoplasmic reticulum stress in amyotrophic lateral sclerosis and interacts with protein disulfide-isomerase. Neurobiology of Aging. 33(12). 2855–2868. 88 indexed citations
13.
Walker, Adam K., Kai Y. Soo, Vita Levina, Gert Talbo, & Julie D. Atkin. (2012). N–linked glycosylation modulates dimerization of protein disulfide isomerase family A member 2 (PDIA2). FEBS Journal. 280(1). 233–243. 17 indexed citations
14.
Soo, Kai Y., Manal A. Farg, & Julie D. Atkin. (2011). Molecular Motor Proteins and Amyotrophic Lateral Sclerosis. International Journal of Molecular Sciences. 12(12). 9057–9082. 17 indexed citations
15.
Soo, Kai Y., Julie D. Atkin, Malcolm Horne, & Phillip Nagley. (2008). Recruitment of mitochondria into apoptotic signaling correlates with the presence of inclusions formed by amyotrophic lateral sclerosis‐associated SOD1 mutations. Journal of Neurochemistry. 108(3). 578–590. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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