Kai Schuh

3.9k total citations · 1 hit paper
7 papers, 1.1k citations indexed

About

Kai Schuh is a scholar working on Genetics, Hematology and Epidemiology. According to data from OpenAlex, Kai Schuh has authored 7 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Genetics, 3 papers in Hematology and 2 papers in Epidemiology. Recurrent topics in Kai Schuh's work include Coagulation, Bradykinin, Polyphosphates, and Angioedema (3 papers), Autophagy in Disease and Therapy (2 papers) and Cellular transport and secretion (2 papers). Kai Schuh is often cited by papers focused on Coagulation, Bradykinin, Polyphosphates, and Angioedema (3 papers), Autophagy in Disease and Therapy (2 papers) and Cellular transport and secretion (2 papers). Kai Schuh collaborates with scholars based in Germany, United States and Canada. Kai Schuh's co-authors include Thomas Renné, Bernhard Nieswandt, Peter Burfeind, David Gailani, Hans‐Ulrich Pauer, Sabine Grüner, Miroslava Požgajová, Guido Stoll, Martin Bendszus and Christoph Renné and has published in prestigious journals such as The Journal of Experimental Medicine, Blood and The Journal of Immunology.

In The Last Decade

Kai Schuh

5 papers receiving 1.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Defective thrombus formation in mice lacking coagulation factor XII

2005 537 citations Thomas Renné, Miroslava Požgajová et al. The Journal of Experimental Medicine profile →

Peers

Countries citing papers authored by Kai Schuh

Since Specialization

Citations

This map shows the geographic impact of Kai Schuh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kai Schuh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kai Schuh more than expected).

Fields of papers citing papers by Kai Schuh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kai Schuh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kai Schuh. The network helps show where Kai Schuh may publish in the future.

Co-authorship network of co-authors of Kai Schuh

This figure shows the co-authorship network connecting the top 25 collaborators of Kai Schuh. A scholar is included among the top collaborators of Kai Schuh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kai Schuh. Kai Schuh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

7 of 7 papers shown

#	Work	Indexed citations
1	From Ca2+ dysregulation to heart failure: β-adrenoceptor activation by RKIP postpones molecular damages and subsequent cardiac dysfunction in mice carrying mutant PLNR9C by correction of aberrant Ca2+-handling 2024 ·Pharmacological Research ·Theresa Brand, Birgit Baumgarten, (unknown), Yvonne Reinders, (unknown), (unknown), (unknown), Nilgün Gedik, (unknown), Petra Kleinbongard, Jan Dudek, (unknown), Elen Tolstik, Kai Schuh, Martina Krüger, Dobromir Dobrev, Friederike Cuello, (unknown), Joachim P. Schmitt, Kristina Lorenz	0
2	Quantification of autophagosome/lysosome fusion in cardiomyocytes and brains of biallelic SPRED2-KO mice reveals a defect in autophagy 2023 ·Physiology ·(unknown), (unknown), (unknown), Kai Schuh	0
3	SPRED2 deficiency elicits cardiac arrhythmias and premature death via impaired autophagy 2019 ·Journal of Molecular and Cellular Cardiology ·Melanie Ullrich, (unknown), Anne Marie Augustin, (unknown), Marco Abeßer, (unknown), Franziska Werner, Ralf Erkens, Paula-Anahi Arias-Loza, (unknown), Helga Wagner, Peter M. Benz, Andreas Unger, Wolfgang A. Linke, Stefan Frantz, Hideo A. Baba, Michaela Kühn, Kai Schuh	22
4	Targeting coagulation factor XII provides protection from pathological thrombosis in cerebral ischemia without interfering with hemostasis 2006 ·The Journal of Experimental Medicine ·Christoph Kleinschnitz, Guido Stoll, Martin Bendszus, Kai Schuh, Hans‐Ulrich Pauer, Peter Burfeind, Christoph Renné, David Gailani, Bernhard Nieswandt, Thomas Renné	360
5	PKCδ regulates collagen-induced platelet aggregation through inhibition of VASP-mediated filopodia formation 2006 ·Blood ·Giordano Pula, Kai Schuh, Keiko Nakayama, Keiichi I. Nakayama, Ulrich Walter, Alastair W. Poole	88
6	Defective thrombus formation in mice lacking coagulation factor XII breakdown → 2005 ·The Journal of Experimental Medicine ·Thomas Renné, Miroslava Požgajová, Sabine Grüner, Kai Schuh, Hans‐Ulrich Pauer, Peter Burfeind, David Gailani, Bernhard Nieswandt	537
7	Local Bradykinin Formation Is Controlled by Glycosaminoglycans 2005 ·The Journal of Immunology ·Thomas Renné, Kai Schuh, Werner Müller‐Esterl	78

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact