K. Naito
- Co-authors
- Akihiko MishimaTomoyuki InoueT. TOSHIOKAKensuke HayashiMamoru SanoHisashi HidakaTakashi ChijiwaMasatoshi Hagiwara
- Topics
- Photoreceptor and optogenetics research (2 papers)Cholinesterase and Neurodegenerative Diseases (2 papers)Planarian Biology and Electrostimulation (2 papers)
In The Last Decade
K. Naito
13 papers receiving 1.5k citations
Hit Papers
Peers
Comparison fields: 5 of 107
- Molecular Biology 980
- Cellular and Molecular Neuroscience 446
- Physiology 186
- Immunology 145
- Oncology 123
Countries citing papers authored by K. Naito
This map shows the geographic impact of K. Naito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K. Naito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K. Naito more than expected).
Fields of papers citing papers by K. Naito
This network shows the impact of papers produced by K. Naito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K. Naito. The network helps show where K. Naito may publish in the future.
Co-authorship network of co-authors of K. Naito
This figure shows the co-authorship network connecting the top 25 collaborators of K. Naito. A scholar is included among the top collaborators of K. Naito based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K. Naito. K. Naito is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 1 | |
| 4 | 4 | |
| 5 | 7 | |
| 6 | 10 | |
| 7 | Inhibition of forskolin-induced neurite outgrowth and protein phosphorylation by a newly synthesized selective inhibitor of cyclic AMP-dependent protein kinase, N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinolinesulfonamide (H-89), of PC12D pheochromocytoma cells.breakdown → | 1408 |
| 8 | Problems of denture wearers regarding denture cleaning. | 3 |
| 9 | [Extrarenal nephroblastoma: report of a case and review of the literature]. | 3 |
| 10 | 20 | |
| 11 | 48 | |
| 12 | 5 | |
| 13 | 2 | |
| 14 | The excretion of vitamin C in the urine during and after the heating of human body. | 1 |
About K. Naito
K. Naito is a scholar working on Otorhinolaryngology, Cellular and Molecular Neuroscience and Paleontology, having authored 14 papers that have together received 1.5k indexed citations. Recurring topics across this work include Photoreceptor and optogenetics research (2 papers), Cholinesterase and Neurodegenerative Diseases (2 papers) and Planarian Biology and Electrostimulation (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (446 citations), Physiology (76 citations) and Molecular Biology (980 citations). K. Naito has collaborated with scholars based in Japan and Pakistan. Frequent co-authors include Akihiko Mishima, Tomoyuki Inoue, T. TOSHIOKA, Kensuke Hayashi, Mamoru Sano, Hisashi Hidaka, Takashi Chijiwa, Masatoshi Hagiwara, Ichiro Tanaka and K. Kuriyama. Their work appears in journals such as Nature, Journal of Biological Chemistry and The Journal of Physiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.