K Hayashi

435 citations
24 papers · 371 indexed · h-index 11
Topics
Trace Elements in Health (3 papers)Iron Metabolism and Disorders (3 papers)Cell death mechanisms and regulation (3 papers)
Partner nations
JapanChinaUnited Kingdom

In The Last Decade

K Hayashi

23 papers receiving 362 citations

Peers

K Hayashi
Comparison fields: 5 of 72
  • Immunology 95
  • Molecular Biology 93
  • Hematology 92
  • Oncology 72
  • Genetics 67
Replace S.S. Papiha with:
S.S. Papiha United Kingdom
Guosu Wang United Kingdom
Werner Olipitz United States
Lina Tian China
Anna Lollies Germany
Truong‐Minh Dang Singapore
Christiane Faltz Germany
Chantal Humblet Belgium
Dinglei Su China
Nina Pilat Austria
K Hayashi relative to S.S. Papiha United Kingdom S.S. Papiha's profile →
Citations per field
00.5×3.4×
S.S. Papiha · 1×
Citations per year

Countries citing papers authored by K Hayashi

Since Specialization
Citations

This map shows the geographic impact of K Hayashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by K Hayashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites K Hayashi more than expected).

Fields of papers citing papers by K Hayashi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by K Hayashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by K Hayashi. The network helps show where K Hayashi may publish in the future.

Co-authorship network of co-authors of K Hayashi

This figure shows the co-authorship network connecting the top 25 collaborators of K Hayashi. A scholar is included among the top collaborators of K Hayashi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with K Hayashi. K Hayashi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 7
2
Immunohistochemical study of ameloblastoma and malignant ameloblastoma by monoclonal antibody against proliferating cell nuclear antigen
3
3 12
4 35
5 114
6 13
7 3
8 4
9 14
10 2
11 12
12 41
13 9
14 4
15 4
16 2
17 0
18 21
19 3
20
[On the varied histological picture of cancer of the pancreas; on the so-called basal cell carcinoma of undifferentiated type].
1

About K Hayashi

K Hayashi is a scholar working on Hematology, Genetics and Oncology, having authored 24 papers that have together received 371 indexed citations. Recurring topics across this work include Trace Elements in Health (3 papers), Iron Metabolism and Disorders (3 papers) and Cell death mechanisms and regulation (3 papers). The work is most often cited by research in Hematology (92 citations), Genetics (67 citations) and Biological Psychiatry (14 citations). K Hayashi has collaborated with scholars based in Japan, China and United Kingdom. Frequent co-authors include Michiyasu Awai, Jianping Lu, Tadaatsu Akagi, Jianping Lu, Kei Takahashi, Lei Cao, Eisei Kondo, Darryl Shibata, Weiss Lm and Shigeru Okada. Their work appears in journals such as Blood, Cancer and Schizophrenia Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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