Junko Matsuda
- Physiology top 2%
- Molecular Biology
- Endocrine and Autonomic Systems top 1%
- Epidemiology top 10%
- Cell Biology top 5%
- Co-authors
- Ichiro YokotaEtsuo NaitoKunihiko SuzukiMichinori ItoYasuhiro KurodaKinuko SuzukiKenji ShimaYuichi Kuroda
- Topics
- Lysosomal Storage Disorders Research (21 papers)Sphingolipid Metabolism and Signaling (15 papers)Glycosylation and Glycoproteins Research (10 papers)
- Journals
- Journal of Biological ChemistryGenes & DevelopmentThe Journal of Clinical Endocrinology & Metabolism
- Partner nations
- JapanUnited StatesFrance
In The Last Decade
Junko Matsuda
70 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 83
- Physiology 928
- Molecular Biology 678
- Endocrine and Autonomic Systems 444
- Epidemiology 356
- Cell Biology 282
Countries citing papers authored by Junko Matsuda
This map shows the geographic impact of Junko Matsuda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Junko Matsuda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Junko Matsuda more than expected).
Fields of papers citing papers by Junko Matsuda
This network shows the impact of papers produced by Junko Matsuda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Junko Matsuda. The network helps show where Junko Matsuda may publish in the future.
Co-authorship network of co-authors of Junko Matsuda
This figure shows the co-authorship network connecting the top 25 collaborators of Junko Matsuda. A scholar is included among the top collaborators of Junko Matsuda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Junko Matsuda. Junko Matsuda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 4 | |
| 3 | 9 | |
| 4 | 8 | |
| 5 | 16 | |
| 6 | 13 | |
| 7 | 34 | |
| 8 | 1 | |
| 9 | 1 | |
| 10 | 28 | |
| 11 | 19 | |
| 12 | 6 | |
| 13 | 23 | |
| 14 | 7 | |
| 15 | 17 | |
| 16 | 36 | |
| 17 | 51 | |
| 18 | [Pyruvate dehydrogenase phosphatase deficiency]. | 2 |
| 19 | 21 | |
| 20 | 19 |
About Junko Matsuda
Junko Matsuda is a scholar working on Biochemistry, Clinical Biochemistry and Physiology, having authored 71 papers that have together received 1.9k indexed citations. Recurring topics across this work include Lysosomal Storage Disorders Research (21 papers), Sphingolipid Metabolism and Signaling (15 papers) and Glycosylation and Glycoproteins Research (10 papers). The work is most often cited by research in Endocrine and Autonomic Systems (444 citations), Physiology (928 citations) and Clinical Biochemistry (146 citations). Junko Matsuda has collaborated with scholars based in Japan, United States and France. Frequent co-authors include Ichiro Yokota, Etsuo Naito, Kunihiko Suzuki, Michinori Ito, Yasuhiro Kuroda, Kinuko Suzuki, Kenji Shima, Yuichi Kuroda, Azusa Yoneshige and Takashi Murakami. Their work appears in journals such as Journal of Biological Chemistry, Genes & Development and The Journal of Clinical Endocrinology & Metabolism.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.