Jun Feng

1.0k total citations · 1 hit paper
24 papers, 716 citations indexed

About

Jun Feng is a scholar working on Molecular Biology, Nephrology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Jun Feng has authored 24 papers receiving a total of 716 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Nephrology and 4 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Jun Feng's work include Mitochondrial Function and Pathology (7 papers), Renal Diseases and Glomerulopathies (6 papers) and Birth, Development, and Health (4 papers). Jun Feng is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), Renal Diseases and Glomerulopathies (6 papers) and Birth, Development, and Health (4 papers). Jun Feng collaborates with scholars based in China and United States. Jun Feng's co-authors include Guohua Ding, Jijia Hu, Zhaowei Chen, Zijing Zhu, Wei Liang, Yiqiong Ma, Qian Yang, Xueyan Yang, Dingping Yang and Yanqin Fan and has published in prestigious journals such as International Journal of Molecular Sciences, Kidney International and Cellular and Molecular Life Sciences.

In The Last Decade

Jun Feng

24 papers receiving 714 citations

Hit Papers

Transition of acute kidney injury to chronic kidney disea... 2022 2026 2023 2024 2022 25 50 75 100

Peers

Jun Feng
Jijia Hu China
Can Wu China
Tomohito Doke United States
Dhanunjay Mukhi United States
Sung-Gyu Cho United States
Jijia Hu China
Jun Feng
Citations per year, relative to Jun Feng Jun Feng (= 1×) peers Jijia Hu

Countries citing papers authored by Jun Feng

Since Specialization
Citations

This map shows the geographic impact of Jun Feng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jun Feng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jun Feng more than expected).

Fields of papers citing papers by Jun Feng

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jun Feng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jun Feng. The network helps show where Jun Feng may publish in the future.

Co-authorship network of co-authors of Jun Feng

This figure shows the co-authorship network connecting the top 25 collaborators of Jun Feng. A scholar is included among the top collaborators of Jun Feng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jun Feng. Jun Feng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Feng, Jun, Feng Ling, Yushan Yan, et al.. (2025). SIRT3 deficiency aggravates mitochondrial metabolic disorder and podocyte injury in DKD via MPC2 acetylation. Cellular Signalling. 135. 112029–112029. 1 indexed citations
2.
Chen, Xinghua, Zijing Zhu, Yiqun Hao, et al.. (2024). STING contributes to lipopolysaccharide-induced tubular cell inflammation and pyroptosis by activating endoplasmic reticulum stress in acute kidney injury. Cell Death and Disease. 15(3). 217–217. 42 indexed citations
3.
Hao, Yiqun, Yanqin Fan, Jun Feng, et al.. (2024). ALCAT1-mediated abnormal cardiolipin remodelling promotes mitochondrial injury in podocytes in diabetic kidney disease. Cell Communication and Signaling. 22(1). 26–26. 21 indexed citations
4.
Chen, Zhaowei, Zijing Zhu, Wei Liang, et al.. (2023). Reduction of anaerobic glycolysis contributes to angiotensin II-induced podocyte injury with foot process effacement. Kidney International. 103(4). 735–748. 38 indexed citations
5.
Yang, Xueyan, Zhaowei Chen, Dingping Yang, et al.. (2023). STING deletion alleviates podocyte injury through suppressing inflammation by targeting NLRP3 in diabetic kidney disease. Cellular Signalling. 109. 110777–110777. 22 indexed citations
6.
Huang, Xiao‐Xiao, Zhaowei Chen, Yiqun Hao, et al.. (2023). PFKFB3 downregulation aggravates Angiotensin II-induced podocyte detachment. Renal Failure. 45(1). 2230318–2230318. 2 indexed citations
7.
Chen, Zhaowei, Wei Liang, Jijia Hu, et al.. (2022). Sirt6 deficiency contributes to mitochondrial fission and oxidative damage in podocytes via ROCK1‐Drp1 signalling pathway. Cell Proliferation. 55(10). e13296–e13296. 21 indexed citations
8.
Chen, Zhaowei, Zijing Zhu, Yiqun Hao, et al.. (2022). Angiotensin II induces podocyte metabolic reprogramming from glycolysis to glycerol-3-phosphate biosynthesis. Cellular Signalling. 99. 110443–110443. 6 indexed citations
9.
Wei, Shanshan, Wenwei Hu, Jun Feng, & Yiting Geng. (2022). Promotion or remission: a role of noncoding RNAs in colorectal cancer resistance to anti-EGFR therapy. Cell Communication and Signaling. 20(1). 150–150. 19 indexed citations
10.
Feng, Jun, Zhaowei Chen, Yiqiong Ma, et al.. (2022). AKAP1 contributes to impaired mtDNA replication and mitochondrial dysfunction in podocytes of diabetic kidney disease. International Journal of Biological Sciences. 18(10). 4026–4042. 36 indexed citations
11.
Zhu, Zijing, Jijia Hu, Zhaowei Chen, et al.. (2022). Transition of acute kidney injury to chronic kidney disease: role of metabolic reprogramming. Metabolism. 131. 155194–155194. 108 indexed citations breakdown →
12.
Zhang, Zongwei, Wei Liang, Qiang Luo, et al.. (2022). PFKP Activation Ameliorates Foot Process Fusion in Podocytes in Diabetic Kidney Disease. Frontiers in Endocrinology. 12. 797025–797025. 17 indexed citations
13.
Yang, Xueyan, Jun Feng, Wei Liang, et al.. (2021). Roles of SIRT6 in kidney disease: a novel therapeutic target. Cellular and Molecular Life Sciences. 79(1). 53–53. 33 indexed citations
14.
Cao, Yun, Jijia Hu, Jun Feng, et al.. (2021). Mfn2 Regulates High Glucose-Induced MAMs Dysfunction and Apoptosis in Podocytes via PERK Pathway. Frontiers in Cell and Developmental Biology. 9. 769213–769213. 66 indexed citations
15.
Fan, Yanqin, Jing Cheng, Qian Yang, et al.. (2021). Sirt6-mediated Nrf2/HO-1 activation alleviates angiotensin II-induced DNA DSBs and apoptosis in podocytes. Food & Function. 12(17). 7867–7882. 32 indexed citations
16.
Ma, Yiqiong, Qian Yang, Jijia Hu, et al.. (2020). AKAP1 mediates high glucose‐induced mitochondrial fission through the phosphorylation of Drp1 in podocytes. Journal of Cellular Physiology. 235(10). 7433–7448. 53 indexed citations
17.
Yang, Qian, Jijia Hu, Yingjie Yang, et al.. (2020). Sirt6 deficiency aggravates angiotensin II-induced cholesterol accumulation and injury in podocytes. Theranostics. 10(16). 7465–7479. 52 indexed citations
18.
Hu, Jijia, Qian Yang, Zhaowei Chen, et al.. (2019). Small GTPase Arf6 regulates diabetes‐induced cholesterol accumulation in podocytes. Journal of Cellular Physiology. 234(12). 23559–23570. 19 indexed citations
19.
Feng, Jun, Yiqiong Ma, Zhaowei Chen, et al.. (2019). Mitochondrial pyruvate carrier 2 mediates mitochondrial dysfunction and apoptosis in high glucose-treated podocytes. Life Sciences. 237. 116941–116941. 30 indexed citations
20.
Feng, Jun, et al.. (2005). Dysfunction of releasing adrenaline in asthma by nerve growth factor. Medical Hypotheses. 65(6). 1043–1046. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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