A Glässner

977 total citations
25 papers, 751 citations indexed

About

A Glässner is a scholar working on Immunology, Hepatology and Epidemiology. According to data from OpenAlex, A Glässner has authored 25 papers receiving a total of 751 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Immunology, 11 papers in Hepatology and 8 papers in Epidemiology. Recurrent topics in A Glässner's work include Immune Cell Function and Interaction (14 papers), Hepatitis C virus research (8 papers) and Liver Disease Diagnosis and Treatment (7 papers). A Glässner is often cited by papers focused on Immune Cell Function and Interaction (14 papers), Hepatitis C virus research (8 papers) and Liver Disease Diagnosis and Treatment (7 papers). A Glässner collaborates with scholars based in Germany, Spain and United Kingdom. A Glässner's co-authors include Benjamin Krämer, Ulrich Spengler, Jacob Nattermann, Tilman Sauerbruch, Marianne Eisenhardt, Hans Dieter Nischalke, Christian Körner, P Kokordelis, Philipp Lutz and F Wolter and has published in prestigious journals such as PLoS ONE, Hepatology and Journal of Hepatology.

In The Last Decade

A Glässner

24 papers receiving 745 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
A Glässner Germany 13 414 344 299 97 58 25 751
Gidon Ellis United Kingdom 7 293 0.7× 207 0.6× 291 1.0× 50 0.5× 57 1.0× 12 584
J Eisenburg Germany 16 249 0.6× 383 1.1× 454 1.5× 57 0.6× 55 0.9× 37 737
Sk. Md. Fazle Akbar Japan 18 486 1.2× 318 0.9× 419 1.4× 59 0.6× 71 1.2× 27 864
Hideko Nuriya Japan 11 163 0.4× 151 0.4× 177 0.6× 19 0.2× 25 0.4× 14 423
Karl‐H. Meyer zum Büschenfelde Germany 9 317 0.8× 250 0.7× 261 0.9× 20 0.2× 42 0.7× 14 676
Katja Klugewitz Germany 13 555 1.3× 310 0.9× 244 0.8× 102 1.1× 27 0.5× 22 928
Constantine G. Marousis United States 7 191 0.5× 634 1.8× 463 1.5× 42 0.4× 34 0.6× 8 784
Huifan Ji China 11 121 0.3× 96 0.3× 127 0.4× 70 0.7× 28 0.5× 21 330
Arjan Boltjes Netherlands 8 320 0.8× 163 0.5× 191 0.6× 27 0.3× 31 0.5× 20 537
Evelyn Rodrigo United States 13 447 1.1× 155 0.5× 187 0.6× 183 1.9× 43 0.7× 17 818

Countries citing papers authored by A Glässner

Since Specialization
Citations

This map shows the geographic impact of A Glässner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A Glässner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A Glässner more than expected).

Fields of papers citing papers by A Glässner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A Glässner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A Glässner. The network helps show where A Glässner may publish in the future.

Co-authorship network of co-authors of A Glässner

This figure shows the co-authorship network connecting the top 25 collaborators of A Glässner. A scholar is included among the top collaborators of A Glässner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A Glässner. A Glässner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Glässner, A, Michael Steffens, Gerda Wurpts, et al.. (2025). Analysis of differential gene expression of PBMC for the in vitro detection of drug sensitization. Allergology International. 74(3). 414–423.
2.
Glässner, A, Gerda Wurpts, Amir S. Yazdi, et al.. (2023). IFN-γ secretion of PBMC from non-drug-allergic control persons: Considerations for the validity of a positive lymphocyte transformation test. Journal of Immunological Methods. 519. 113515–113515. 1 indexed citations
3.
Glässner, A, et al.. (2022). Lymphocyte transformation test for drug allergy detection. Annals of Allergy Asthma & Immunology. 129(4). 497–506.e3. 11 indexed citations
4.
Sachs, Bernhardt, et al.. (2021). Lymphocyte transformation test: History and current approaches. Journal of Immunological Methods. 493. 113036–113036. 44 indexed citations
5.
Glässner, A, et al.. (2021). Future perspectives on in-vitro diagnosis of drug allergy by the lymphocyte transformation test. Journal of Immunological Methods. 495. 113072–113072. 7 indexed citations
6.
Kaczmarek, Dominik J., P Kokordelis, Benjamin Krämer, et al.. (2017). Alterations of the NK cell pool in HIV/HCV co-infection. PLoS ONE. 12(4). e0174465–e0174465. 12 indexed citations
7.
Krämer, Benjamin, Felix Goeser, Philipp Lutz, et al.. (2017). Compartment-specific distribution of human intestinal innate lymphoid cells is altered in HIV patients under effective therapy. PLoS Pathogens. 13(5). e1006373–e1006373. 88 indexed citations
8.
Krämer, Benjamin, Beatriz Sastre, Philipp Lutz, et al.. (2016). IL-28B Genetic Variants Determine the Extent of Monocyte-Induced Activation of NK Cells in Hepatitis C. PLoS ONE. 11(9). e0162068–e0162068. 4 indexed citations
9.
Goeser, Felix, A Glässner, P Kokordelis, et al.. (2015). HIV mono-infection is associated with an impaired anti-HCV activity of NK cells. AIDS. 1–1. 6 indexed citations
10.
Kokordelis, P, Benjamin Krämer, Christoph Boesecke, et al.. (2015). CD3(+)CD56(+) Natural Killer-Like T Cells Display Anti-HCV Activity but Are Functionally Impaired in HIV(+) Patients With Acute Hepatitis C. JAIDS Journal of Acquired Immune Deficiency Syndromes. 70(4). 338–346. 14 indexed citations
11.
Wolter, F, A Glässner, Benjamin Krämer, et al.. (2015). Hypoxia impairs anti-viral activity of natural killer (NK) cells but has little effect on anti-fibrotic NK cell functions in hepatitis C virus infection. Journal of Hepatology. 63(6). 1334–1344. 10 indexed citations
12.
Eisenhardt, Marianne, A Glässner, F Wolter, et al.. (2014). CD27(+)CD56Bright natural killer cells may be involved in spontaneous clearance of acute hepatitis C in HIV-positive patients. AIDS. 28(13). 1879–1884. 7 indexed citations
13.
14.
Langhans, Bettina, Benjamin Krämer, A Glässner, et al.. (2014). Regulatory CD4+ T cells modulate the interaction between NK cells and hepatic stellate cells by acting on either cell type. Journal of Hepatology. 62(2). 398–404. 61 indexed citations
15.
Glässner, A, Marianne Eisenhardt, P Kokordelis, et al.. (2013). Impaired CD4+ T cell stimulation of NK cell anti-fibrotic activity may contribute to accelerated liver fibrosis progression in HIV/HCV patients. Journal of Hepatology. 59(3). 427–433. 66 indexed citations
16.
Nischalke, Hans Dieter, Cordula Berger, Philipp Lutz, et al.. (2013). Influence of the CXCL1 rs4074 A Allele on Alcohol Induced Cirrhosis and HCC in Patients of European Descent. PLoS ONE. 8(11). e80848–e80848. 18 indexed citations
17.
Eisenhardt, Marianne, A Glässner, Benjamin Krämer, et al.. (2012). The CXCR3(+)CD56Bright Phenotype Characterizes a Distinct NK Cell Subset with Anti-Fibrotic Potential That Shows Dys-Regulated Activity in Hepatitis C. PLoS ONE. 7(7). e38846–e38846. 35 indexed citations
18.
Körner, Christian, Katarina Riesner, Benjamin Krämer, et al.. (2012). TRAIL receptor I (DR4) polymorphisms C626G and A683C are associated with an increased risk for hepatocellular carcinoma (HCC) in HCV-infected patients. BMC Cancer. 12(1). 85–85. 19 indexed citations
19.
Glässner, A, Marianne Eisenhardt, Benjamin Krämer, et al.. (2012). NK cells from HCV-infected patients effectively induce apoptosis of activated primary human hepatic stellate cells in a TRAIL-, FasL- and NKG2D-dependent manner. Laboratory Investigation. 92(7). 967–977. 126 indexed citations
20.
Coenen, Martin, Hans Dieter Nischalke, Benjamin Krämer, et al.. (2011). Hepatitis C virus core protein induces fibrogenic actions of hepatic stellate cells via toll-like receptor 2. Laboratory Investigation. 91(9). 1375–1382. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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