Júlia Canet-Pons

495 total citations
16 papers, 286 citations indexed

About

Júlia Canet-Pons is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Júlia Canet-Pons has authored 16 papers receiving a total of 286 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 5 papers in Neurology. Recurrent topics in Júlia Canet-Pons's work include Mitochondrial Function and Pathology (7 papers), RNA Research and Splicing (4 papers) and Genetic Neurodegenerative Diseases (4 papers). Júlia Canet-Pons is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), RNA Research and Splicing (4 papers) and Genetic Neurodegenerative Diseases (4 papers). Júlia Canet-Pons collaborates with scholars based in Germany, Spain and United States. Júlia Canet-Pons's co-authors include Georg Auburger, Suzana Gispert, Jana Key, Nesli-Ece Şen, David Meierhofer, Sylvia Torres-Odio, Michel Mittelbronn, Melanie V. Halbach, Tuan Trang and Patrick N. Harter and has published in prestigious journals such as Scientific Reports, International Journal of Molecular Sciences and Molecular & Cellular Proteomics.

In The Last Decade

Júlia Canet-Pons

15 papers receiving 284 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Júlia Canet-Pons Germany 10 161 140 99 41 30 16 286
Sissel Ida Schmidt Denmark 8 146 0.9× 109 0.8× 129 1.3× 65 1.6× 26 0.9× 12 317
Lina A. Matschke Germany 10 190 1.2× 153 1.1× 148 1.5× 63 1.5× 21 0.7× 11 378
Fangshuo Zheng China 10 118 0.7× 110 0.8× 36 0.4× 38 0.9× 25 0.8× 15 262
Zhi-hua Yang China 8 179 1.1× 60 0.4× 99 1.0× 50 1.2× 26 0.9× 15 324
Alexander Starr United States 8 234 1.5× 105 0.8× 186 1.9× 53 1.3× 26 0.9× 10 404
Giorgia Puorro Italy 10 238 1.5× 196 1.4× 130 1.3× 24 0.6× 35 1.2× 14 354
Vivian Dambeck Germany 7 98 0.6× 106 0.8× 145 1.5× 85 2.1× 33 1.1× 11 312
Lara Petricca Italy 10 250 1.6× 249 1.8× 99 1.0× 47 1.1× 36 1.2× 13 362
Chikara Yamashita Japan 6 137 0.9× 83 0.6× 160 1.6× 46 1.1× 41 1.4× 9 270
Jian-Nan Zhang China 9 154 1.0× 86 0.6× 36 0.4× 40 1.0× 29 1.0× 12 289

Countries citing papers authored by Júlia Canet-Pons

Since Specialization
Citations

This map shows the geographic impact of Júlia Canet-Pons's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Júlia Canet-Pons with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Júlia Canet-Pons more than expected).

Fields of papers citing papers by Júlia Canet-Pons

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Júlia Canet-Pons. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Júlia Canet-Pons. The network helps show where Júlia Canet-Pons may publish in the future.

Co-authorship network of co-authors of Júlia Canet-Pons

This figure shows the co-authorship network connecting the top 25 collaborators of Júlia Canet-Pons. A scholar is included among the top collaborators of Júlia Canet-Pons based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Júlia Canet-Pons. Júlia Canet-Pons is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Almaguer-Mederos, Luis E., Jana Key, Nesli-Ece Şen, et al.. (2025). Multiomics approach identifies SERPINB1 as candidate biomarker for spinocerebellar ataxia type 2. Scientific Reports. 15(1). 42559–42559.
2.
Şen, Nesli-Ece, Júlia Canet-Pons, Matthew P. Stokes, et al.. (2025). Spinal Cord Phosphoproteome of SCA2 Mouse Model Reveals Alteration of ATXN2-N-Term PRM–SH3–Actin Interactome and of Autophagy. Molecular & Cellular Proteomics. 24(11). 101072–101072. 1 indexed citations
3.
Harding, Erika K., et al.. (2024). Expression of GAD2 in excitatory neurons projecting from the ventrolateral periaqueductal gray to the locus coeruleus. iScience. 27(6). 109972–109972. 4 indexed citations
4.
5.
Canet-Pons, Júlia, Nesli-Ece Şen, Melanie V. Halbach, et al.. (2021). Atxn2-CAG100-KnockIn mouse spinal cord shows progressive TDP43 pathology associated with cholesterol biosynthesis suppression. Neurobiology of Disease. 152. 105289–105289. 30 indexed citations
6.
Key, Jana, Ilka Wittig, Suzana Gispert, et al.. (2021). Increased presence of nuclear DNAJA3 and upregulation of cytosolic STAT1 and of nucleic acid sensors trigger innate immunity in the ClpP-null mouse. Neurogenetics. 22(4). 297–312. 12 indexed citations
7.
Kwok, Charlie H.T., et al.. (2020). Spinal interleukin-6 contributes to central sensitisation and persistent pain hypersensitivity in a model of juvenile idiopathic arthritis. Brain Behavior and Immunity. 90. 145–154. 21 indexed citations
8.
Halbach, Melanie V., Júlia Canet-Pons, Claudia Döring, et al.. (2020). Mouse Ataxin-2 Expansion Downregulates CamKII and Other Calcium Signaling Factors, Impairing Granule—Purkinje Neuron Synaptic Strength. International Journal of Molecular Sciences. 21(18). 6673–6673. 20 indexed citations
9.
Şen, Nesli-Ece, David Meierhofer, Susanne Brodesser, et al.. (2019). In Human and Mouse Spino-Cerebellar Tissue, Ataxin-2 Expansion Affects Ceramide-Sphingomyelin Metabolism. International Journal of Molecular Sciences. 20(23). 5854–5854. 20 indexed citations
10.
Şen, Nesli-Ece, Júlia Canet-Pons, Melanie V. Halbach, et al.. (2019). Generation of an Atxn2-CAG100 knock-in mouse reveals N-acetylaspartate production deficit due to early Nat8l dysregulation. Neurobiology of Disease. 132. 104559–104559. 27 indexed citations
11.
Auburger, Georg, Suzana Gispert, Sylvia Torres-Odio, et al.. (2019). SerThr-PhosphoProteome of Brain from Aged PINK1-KO+A53T-SNCA Mice Reveals pT1928-MAP1B and pS3781-ANK2 Deficits, as Hub between Autophagy and Synapse Changes. International Journal of Molecular Sciences. 20(13). 3284–3284. 13 indexed citations
12.
Canet-Pons, Júlia, Ralf Schubert, Matthias Kieslich, et al.. (2018). Ataxia telangiectasia alters the ApoB and reelin pathway. Neurogenetics. 19(4). 237–255. 11 indexed citations
13.
Gámez‐Valero, Ana, Júlia Canet-Pons, Aintzane Urbizu, et al.. (2018). INDEL Length and Haplotypes in the β-Synuclein Gene: A Key to Differentiate Dementia with Lewy Bodies?1. Journal of Alzheimer s Disease. 65(1). 207–219. 6 indexed citations
14.
Torres-Odio, Sylvia, Jana Key, Hans-Hermann Hoepken, et al.. (2017). Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation. Journal of Neuroinflammation. 14(1). 154–154. 60 indexed citations
15.
Muñoz, Ana, Estefanía Moreno, Irene Reyes‐Resina, et al.. (2014). Stronger Dopamine D1 Receptor-Mediated Neurotransmission in Dyskinesia. Molecular Neurobiology. 52(3). 1408–1420. 52 indexed citations
16.
Urbizu, Aintzane, Júlia Canet-Pons, Ana M Muñoz‐Mármol, et al.. (2014). Cystatin C is differentially involved in multiple system atrophy phenotypes. Neuropathology and Applied Neurobiology. 41(4). 507–519. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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