Juha Isosomppi

780 total citations
20 papers, 610 citations indexed

About

Juha Isosomppi is a scholar working on Molecular Biology, Physiology and Cell Biology. According to data from OpenAlex, Juha Isosomppi has authored 20 papers receiving a total of 610 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 8 papers in Physiology and 6 papers in Cell Biology. Recurrent topics in Juha Isosomppi's work include Lysosomal Storage Disorders Research (8 papers), Hearing, Cochlea, Tinnitus, Genetics (5 papers) and Cellular transport and secretion (5 papers). Juha Isosomppi is often cited by papers focused on Lysosomal Storage Disorders Research (8 papers), Hearing, Cochlea, Tinnitus, Genetics (5 papers) and Cellular transport and secretion (5 papers). Juha Isosomppi collaborates with scholars based in Finland, United States and Israel. Juha Isosomppi's co-authors include Anu Jalanko, Jouni Vesa, John G. Flannery, Esteban C. Dell’Angelica, Mark H. Chin, Leena Peltonen, Leena Peltonen, Eeva‐Marja Sankila, Tuomas Klockars and Scott Geller and has published in prestigious journals such as Journal of Biological Chemistry, Genome Research and Human Molecular Genetics.

In The Last Decade

Juha Isosomppi

19 papers receiving 572 citations

Peers

Juha Isosomppi

PHYSI

Misa Shimuta Japan

Ebru Ercan Germany

Helmuth A. Sánchez Chile

Elena Fdez Spain

Yoshikatsu Aikawa Japan

Minna Savukoski Finland

Pilar Rivero-Ríos Spain

Jia Hao China

Natalia Gustavsson Singapore

Kasturi L. Puranam United States

Misa Shimuta Japan

Juha Isosomppi

483 ×1.5

162 ×0.5

PHYSI

101 ×0.4

77 ×0.7

55 ×0.7

PHYSI

Citations per year, relative to Juha Isosomppi Juha Isosomppi (= 1×) peers Misa Shimuta

Countries citing papers authored by Juha Isosomppi

Since Specialization

Citations

This map shows the geographic impact of Juha Isosomppi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Juha Isosomppi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Juha Isosomppi more than expected).

Fields of papers citing papers by Juha Isosomppi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Juha Isosomppi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Juha Isosomppi. The network helps show where Juha Isosomppi may publish in the future.

Co-authorship network of co-authors of Juha Isosomppi

This figure shows the co-authorship network connecting the top 25 collaborators of Juha Isosomppi. A scholar is included among the top collaborators of Juha Isosomppi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Juha Isosomppi. Juha Isosomppi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Aarnisalo, Antti A., Juha Isosomppi, Heikki Löppönen, et al.. (2011). Speech Recognition and Communication Outcomes With Cochlear Implantation in Usher Syndrome Type 3. Otology & Neurotology. 33(1). 38–41. 23 indexed citations

Dinculescu, Astra, Juha Isosomppi, Scott Geller, et al.. (2010). Alternative splice variants of the USH3A gene Clarin 1 (CLRN1). European Journal of Human Genetics. 19(1). 30–35. 24 indexed citations

Tian, Guilian, Masaru Miyagi, Astra Dinculescu, et al.. (2009). Clarin-1, Encoded by the Usher Syndrome III Causative Gene, Forms a Membranous Microdomain. Journal of Biological Chemistry. 284(28). 18980–18993. 44 indexed citations

Geller, Scott, Karen I. Guerin, Meike Visel, et al.. (2009). CLRN1 Is Nonessential in the Mouse Retina but Is Required for Cochlear Hair Cell Development. PLoS Genetics. 5(8). e1000607–e1000607. 37 indexed citations

Isosomppi, Juha, et al.. (2009). Disease-causing mutations in the CLRN1 gene alter normal CLRN1 protein trafficking to the plasma membrane.. PubMed. 15. 1806–18. 27 indexed citations

Aarnisalo, Antti A., Jaana Joensuu, Juha Isosomppi, et al.. (2007). Anti-clarin-1 AAV-delivered ribozyme induced apoptosis in the mouse cochlea. Hearing Research. 230(1-2). 9–16. 9 indexed citations

Isosomppi, Juha, et al.. (2006). Two Finnish USH1B patients with three novel mutations in myosin VIIA.. PubMed. 12. 1093–7. 10 indexed citations

Geller, Scott, et al.. (2004). Vision Loss in Usher Syndrome Type III is Caused by Mutations in Clarin–1, an Inner Retinal Protein.. Investigative Ophthalmology & Visual Science. 45(13). 5123–5123. 1 indexed citations

Holmberg, Ville, Anu Jalanko, Juha Isosomppi, et al.. (2004). The mouse ortholog of the neuronal ceroid lipofuscinosis CLN5 gene encodes a soluble lysosomal glycoprotein expressed in the developing brain. Neurobiology of Disease. 16(1). 29–40. 52 indexed citations

10.

Isosomppi, Juha. (2003). MOLECULAR AND CELL BIOLOGY OF INFANTILE (CLN1) AND VARIANT LATE INFANTILE (CLN5) NEURONAL CEROID LIPOFUSCINOSES. Työväentutkimus Vuosikirja. 3 indexed citations

11.

Isosomppi, Juha. (2002). Lysosomal localization of the neuronal ceroid lipofuscinosis CLN5 protein. Human Molecular Genetics. 11(8). 885–891. 100 indexed citations

12.

Vesa, Jouni, Mark H. Chin, Juha Isosomppi, et al.. (2002). Neuronal Ceroid Lipofuscinoses Are Connected at Molecular Level: Interaction of CLN5 Protein with CLN2 and CLN3. Molecular Biology of the Cell. 13(7). 2410–2420. 94 indexed citations

13.

Klockars, Tuomas, et al.. (1999). Positional Cloning of the CLN5 Gene Defective in the Finnish Variant of the LINCL. Molecular Genetics and Metabolism. 66(4). 324–328. 6 indexed citations

14.

Isosomppi, Juha, Outi Heinonen, Jukka O. Hiltunen, et al.. (1999). Developmental expression of palmitoyl protein thioesterase in normal mice. Developmental Brain Research. 118(1-2). 1–11. 47 indexed citations

15.

Rapola, Juhani, Jaana Lähdetie, Juha Isosomppi, et al.. (1999). Prenatal diagnosis of variant late infantile neuronal ceroid lipofuscinosis (vLINCLFinnish; CLN5). Prenatal Diagnosis. 19(7). 685–688. 14 indexed citations

16.

Nikali, Kaisu, et al.. (1997). Toward Cloning of a Novel Ataxia Gene: Refined Assignment and Physical Map of the IOSCA Locus (SCA8) on 10q24. Genomics. 39(2). 185–191. 40 indexed citations

17.

Klockars, Tuomas, et al.. (1997). The Visual Assignment of Genes by Fiber-Fish: BTF3 Protein Homologue Gene (BTF3) and a Novel Pseudogene of Human RNA Helicase A (DDX9P) on 13q22. Genomics. 44(3). 355–357. 8 indexed citations

18.

Isosomppi, Juha, et al.. (1996). A Physical Map of the 6q14–q15 Region Harboring the Locus for the Lysosomal Membrane Sialic Acid Transport Defect. Genomics. 37(1). 62–67. 17 indexed citations

19.

Klockars, Tuomas, Minna Savukoski, Juha Isosomppi, et al.. (1996). Efficient Construction of a Physical Map by Fiber-Fish of the CLN5 Region: Refined Assignment and Long-Range Contig Covering the Critical Region on 13q22. Genomics. 35(1). 71–78. 48 indexed citations

20.

Laan, Maris, Juha Isosomppi, Tuomas Klockars, Laura‐Maria Peltonen, & Aarno Palotie. (1996). Utilization of FISH in positional cloning: an example on 13q22.. Genome Research. 6(10). 1002–1012. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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