Judit Gal Toth

436 total citations
11 papers, 341 citations indexed

About

Judit Gal Toth is a scholar working on Social Psychology, Behavioral Neuroscience and Immunology. According to data from OpenAlex, Judit Gal Toth has authored 11 papers receiving a total of 341 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Social Psychology, 5 papers in Behavioral Neuroscience and 5 papers in Immunology. Recurrent topics in Judit Gal Toth's work include Stress Responses and Cortisol (5 papers), Neuroendocrine regulation and behavior (5 papers) and Reproductive System and Pregnancy (3 papers). Judit Gal Toth is often cited by papers focused on Stress Responses and Cortisol (5 papers), Neuroendocrine regulation and behavior (5 papers) and Reproductive System and Pregnancy (3 papers). Judit Gal Toth collaborates with scholars based in United States and Spain. Judit Gal Toth's co-authors include Bruce S. McEwen, Kevin G. Bath, Ana Marı́a Magariños, Dapeng Jing, Miklós Tóth, Bingfang Liu, Bojana Zupan, Shifra Klein, Changsoo Kwak and Karen Bulloch and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Nature Neuroscience.

In The Last Decade

Judit Gal Toth

10 papers receiving 340 citations

Peers

Judit Gal Toth
Anna Schroeder Australia
Udani Ratnayake Australia
Pamela W. L. Yeh United States
Rong‐Jun Ni China
Mathilde S. Henry Canada
M. C. Morsink Netherlands
Andrew M. Swanson United States
Changhyeon Ryu South Korea
Jung-A Hwang South Korea
Daniel Peterlik Germany
Anna Schroeder Australia
Judit Gal Toth
Citations per year, relative to Judit Gal Toth Judit Gal Toth (= 1×) peers Anna Schroeder

Countries citing papers authored by Judit Gal Toth

Since Specialization
Citations

This map shows the geographic impact of Judit Gal Toth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Judit Gal Toth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Judit Gal Toth more than expected).

Fields of papers citing papers by Judit Gal Toth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Judit Gal Toth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Judit Gal Toth. The network helps show where Judit Gal Toth may publish in the future.

Co-authorship network of co-authors of Judit Gal Toth

This figure shows the co-authorship network connecting the top 25 collaborators of Judit Gal Toth. A scholar is included among the top collaborators of Judit Gal Toth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Judit Gal Toth. Judit Gal Toth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
2.
Toth, Judit Gal, et al.. (2024). The chemokine XCL1 functions as a pregnancy hormone to program offspring innate anxiety. Brain Behavior and Immunity. 118. 178–189. 1 indexed citations
3.
Toth, Judit Gal, et al.. (2022). Serotonin-1A receptor, a psychiatric disease risk factor, influences offspring immunity via sex-dependent genetic nurture. iScience. 25(12). 105595–105595. 3 indexed citations
4.
Taki, Faten, Katherine Lopez, Bojana Zupan, et al.. (2020). Maternal Programming of Social Dominance via Milk Cytokines. iScience. 23(8). 101357–101357. 8 indexed citations
5.
Taki, Faten, Shifra Klein, Mary Jane Skelly, et al.. (2020). Epigenomically Bistable Regions across Neuron-Specific Genes Govern Neuron Eligibility to a Coding Ensemble in the Hippocampus. Cell Reports. 31(12). 107789–107789. 9 indexed citations
6.
Zupan, Bojana, Bingfang Liu, Faten Taki, Judit Gal Toth, & Miklós Tóth. (2017). Maternal Brain TNF-α Programs Innate Fear in the Offspring. Current Biology. 27(24). 3859–3863.e3. 11 indexed citations
7.
Klein, Shifra, Kimon V. Argyropoulos, Ali Sharma, et al.. (2016). Behavioural traits propagate across generations via segregated iterative-somatic and gametic epigenetic mechanisms. Nature Communications. 7(1). 11492–11492. 28 indexed citations
8.
Liu, Bingfang, et al.. (2013). Maternal hematopoietic TNF, via milk chemokines, programs hippocampal development and memory. Nature Neuroscience. 17(1). 97–105. 48 indexed citations
9.
D’Agostino, Paul M., Changsoo Kwak, Haley A. Vecchiarelli, et al.. (2012). Viral induced encephalitis initiates distinct and functional CD103+ CD11b+ brain dendritic cell populations within the olfactory bulb. (168.8). The Journal of Immunology. 188(1_Supplement). 168.8–168.8. 1 indexed citations
10.
D’Agostino, Paul M., Changsoo Kwak, Haley A. Vecchiarelli, et al.. (2012). Viral-induced encephalitis initiates distinct and functional CD103 + CD11b + brain dendritic cell populations within the olfactory bulb. Proceedings of the National Academy of Sciences. 109(16). 6175–6180. 37 indexed citations
11.
Magariños, Ana Marı́a, et al.. (2010). Effect of brain‐derived neurotrophic factor haploinsufficiency on stress‐induced remodeling of hippocampal neurons. Hippocampus. 21(3). 253–264. 195 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026