Joseph M. Rutkowski

6.8k total citations · 3 hit papers
83 papers, 5.3k citations indexed

About

Joseph M. Rutkowski is a scholar working on Oncology, Physiology and Surgery. According to data from OpenAlex, Joseph M. Rutkowski has authored 83 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Oncology, 26 papers in Physiology and 17 papers in Surgery. Recurrent topics in Joseph M. Rutkowski's work include Lymphatic System and Diseases (40 papers), Adipokines, Inflammation, and Metabolic Diseases (13 papers) and Angiogenesis and VEGF in Cancer (10 papers). Joseph M. Rutkowski is often cited by papers focused on Lymphatic System and Diseases (40 papers), Adipokines, Inflammation, and Metabolic Diseases (13 papers) and Angiogenesis and VEGF in Cancer (10 papers). Joseph M. Rutkowski collaborates with scholars based in United States, Switzerland and Finland. Joseph M. Rutkowski's co-authors include Philipp E. Scherer, Jennifer H. Stern, Melody A. Swartz, Kathryn E. Davis, Zhao V. Wang, Jeremy Goldman, Kai Sun, Jacqueline D. Shields, William L. Holland and Nils Halberg and has published in prestigious journals such as Cell, Circulation and Nature Medicine.

In The Last Decade

Joseph M. Rutkowski

78 papers receiving 5.2k citations

Hit Papers

Adiponectin, Leptin, and Fatty Acids in the Maintenanc... 2010 2026 2015 2020 2016 2010 2015 250 500 750

Peers

Joseph M. Rutkowski
Joseph M. Rutkowski
Citations per year, relative to Joseph M. Rutkowski Joseph M. Rutkowski (= 1×) peers Yutaka Takahashi

Countries citing papers authored by Joseph M. Rutkowski

Since Specialization
Citations

This map shows the geographic impact of Joseph M. Rutkowski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph M. Rutkowski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph M. Rutkowski more than expected).

Fields of papers citing papers by Joseph M. Rutkowski

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph M. Rutkowski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph M. Rutkowski. The network helps show where Joseph M. Rutkowski may publish in the future.

Co-authorship network of co-authors of Joseph M. Rutkowski

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph M. Rutkowski. A scholar is included among the top collaborators of Joseph M. Rutkowski based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph M. Rutkowski. Joseph M. Rutkowski is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rutkowski, Joseph M., et al.. (2025). Tissue‐Engineered Therapeutics for Lymphatic Regeneration: Solutions for Myocardial Infarction and Secondary Lymphedema. Advanced Healthcare Materials. 14(6). e2403551–e2403551. 2 indexed citations
2.
Phan, T., et al.. (2024). Therapeutically Induced Lymphangiogenesis Is Ineffective in Resolving Established Kidney Disease in Mice. Kidney360. 6(4). 509–520. 1 indexed citations
3.
Godefroy, David, et al.. (2024). Single-Cell RNA Sequencing Identifies Response of Renal Lymphatic Endothelial Cells to Acute Kidney Injury. Journal of the American Society of Nephrology. 35(5). 549–565. 7 indexed citations
4.
Verissimo, Thomas, Anna Faivre, Soline Bourgeois, et al.. (2023). PCK1 is a key regulator of metabolic and mitochondrial functions in renal tubular cells. American Journal of Physiology-Renal Physiology. 324(6). F532–F543. 28 indexed citations
5.
Baranwal, Gaurav, et al.. (2023). Indole Propionic Acid Increases T Regulatory Cells and Decreases T Helper 17 Cells and Blood Pressure in Mice with Salt-Sensitive Hypertension. International Journal of Molecular Sciences. 24(11). 9192–9192. 14 indexed citations
6.
Phan, T., et al.. (2023). Increased adipose tissue lymphatic vessel density inhibits thermogenesis through elevated neurotensin levels. Frontiers in Cell and Developmental Biology. 11. 1100788–1100788. 3 indexed citations
7.
Reyna, Andrea, et al.. (2022). Evaluation of the Cardiometabolic Disorders after Spinal Cord Injury in Mice. Biology. 11(4). 495–495. 4 indexed citations
8.
Phan, T., et al.. (2022). Current Mechanistic Understandings of Lymphedema and Lipedema: Tales of Fluid, Fat, and Fibrosis. International Journal of Molecular Sciences. 23(12). 6621–6621. 46 indexed citations
9.
Ferguson, Megan, Rachel Cohen, David J. Orlicky, et al.. (2022). Oxidized low density lipoprotein in the liver causes decreased permeability of liver lymphatic- but not liver sinusoidal-endothelial cells via VEGFR-3 regulation of VE-Cadherin. Frontiers in Physiology. 13. 1021038–1021038. 4 indexed citations
10.
Baranwal, Gaurav, et al.. (2021). Expanded renal lymphatics improve recovery following kidney injury. Physiological Reports. 9(22). e15094–e15094. 12 indexed citations
11.
Rutkowski, Joseph M., et al.. (2021). Emerging roles for lymphatics in acute kidney injury: Beneficial or maleficent?. Experimental Biology and Medicine. 246(7). 845–850. 10 indexed citations
12.
Morley, Thomas S., et al.. (2020). Characterizing Lymphangiogenesis and Concurrent Inflammation in Adipose Tissue in Response to VEGF-D. Frontiers in Physiology. 11. 363–363. 11 indexed citations
13.
Reyna, Andrea, et al.. (2019). Vascular Endothelial Growth Factor–D (VEGF-D) Overexpression and Lymphatic Expansion in Murine Adipose Tissue Improves Metabolism in Obesity. American Journal Of Pathology. 189(4). 924–939. 47 indexed citations
14.
Asterholm, Ingrid Wernstedt, Joseph M. Rutkowski, Teppei Fujikawa, et al.. (2014). Elevated resistin levels induce central leptin resistance and increased atherosclerotic progression in mice. Diabetologia. 57(6). 1209–1218. 41 indexed citations
15.
Rutkowski, Joseph M. & Philipp E. Scherer. (2014). Isolation and Quantitation of Adiponectin Higher Order Complexes. Methods in enzymology on CD-ROM/Methods in enzymology. 537. 243–259. 11 indexed citations
16.
Rutkowski, Joseph M., Nils Halberg, Qiong Wang, et al.. (2014). Differential transendothelial transport of adiponectin complexes. Cardiovascular Diabetology. 13(1). 47–47. 18 indexed citations
17.
Asterholm, Ingrid Wernstedt, John McDonald, Pierre-Gilles Blanchard, et al.. (2012). Lack of “immunological fitness” during fasting in metabolically challenged animals. Journal of Lipid Research. 53(7). 1254–1267. 38 indexed citations
18.
Lim, Hwee Ying, Joseph M. Rutkowski, Julie Helft, et al.. (2009). Hypercholesterolemic Mice Exhibit Lymphatic Vessel Dysfunction and Degeneration. American Journal Of Pathology. 175(3). 1328–1337. 137 indexed citations
19.
Thomas, Susan N., Sachiko Hirosue, Joseph M. Rutkowski, Kari Alitalo, & Melody A. Swartz. (2009). Altered immune responses in mice without initial dermal lymphatics. Journal of Vascular Research. 46(3). 46. 1 indexed citations
20.
Mazurek, W., et al.. (2004). Electric field influence on the condensation process in dielectric heat pipe operating with R365/227. 40. 262–266. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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