Josef P. Kapfhammer

5.4k total citations · 3 hit papers
80 papers, 4.4k citations indexed

About

Josef P. Kapfhammer is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Developmental Neuroscience. According to data from OpenAlex, Josef P. Kapfhammer has authored 80 papers receiving a total of 4.4k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Molecular Biology, 51 papers in Cellular and Molecular Neuroscience and 17 papers in Developmental Neuroscience. Recurrent topics in Josef P. Kapfhammer's work include Neuroscience and Neuropharmacology Research (22 papers), Neurogenesis and neuroplasticity mechanisms (17 papers) and Nerve injury and regeneration (15 papers). Josef P. Kapfhammer is often cited by papers focused on Neuroscience and Neuropharmacology Research (22 papers), Neurogenesis and neuroplasticity mechanisms (17 papers) and Nerve injury and regeneration (15 papers). Josef P. Kapfhammer collaborates with scholars based in Switzerland, Germany and United States. Josef P. Kapfhammer's co-authors include JA Raper, Martin E. Schwab, C. E. Bandtlow, Martin E. Schwab, Jonathan A. Raper, Friedrich Metzger, Silvia Arber, Florence Botteri, Corinna Schneider and Pico Caroni and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Josef P. Kapfhammer

78 papers receiving 4.4k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Disruption of CREB function in brain leads to neurodegeneration

2002 567 citations Josef P. Kapfhammer et al. profile →
Overexpression of the neural growth-associated protein GAP-43 induces nerve sprouting in the adult nervous system of transgenic mice

1995 535 citations Josef P. Kapfhammer et al. profile →
Inhibitors of Neurite Growth

1993 467 citations Martin E. Schwab, Josef P. Kapfhammer et al. Annual Review of Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Josef P. Kapfhammer Switzerland	31	2.9k	1.9k	1.4k	648	361	80	4.4k
Christine E. Bandtlow Austria	31	3.7k 1.3×	1.8k 1.0×	2.0k 1.4×	938 1.4×	523 1.4×	56	5.3k
Philip Barker Canada	40	3.0k 1.0×	2.3k 1.2×	1.2k 0.9×	423 0.7×	372 1.0×	69	4.5k
Hitoshi Komuro United States	31	2.9k 1.0×	2.0k 1.0×	1.6k 1.1×	450 0.7×	471 1.3×	71	4.6k
Keling Zang United States	23	1.7k 0.6×	1.6k 0.8×	856 0.6×	464 0.7×	266 0.7×	27	3.8k
Markus H. Schwab Germany	31	2.3k 0.8×	2.4k 1.3×	1.8k 1.3×	497 0.8×	735 2.0×	48	5.0k
Jessica A. Gorski United States	19	2.0k 0.7×	2.0k 1.1×	1.1k 0.8×	541 0.8×	256 0.7×	23	4.1k
Ferdinando Rossi Italy	41	2.5k 0.9×	1.7k 0.9×	2.1k 1.5×	584 0.9×	988 2.7×	118	4.6k
Soledad Alcántara Spain	32	2.4k 0.8×	1.5k 0.8×	1.9k 1.4×	411 0.6×	434 1.2×	51	4.2k
Shernaz X. Bamji Canada	32	1.9k 0.7×	2.0k 1.1×	604 0.4×	609 0.9×	255 0.7×	47	3.8k
Barbara A. Barres United States	16	2.6k 0.9×	2.7k 1.4×	1.3k 0.9×	331 0.5×	748 2.1×	22	4.7k

Countries citing papers authored by Josef P. Kapfhammer

Since Specialization

Citations

This map shows the geographic impact of Josef P. Kapfhammer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Josef P. Kapfhammer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Josef P. Kapfhammer more than expected).

Fields of papers citing papers by Josef P. Kapfhammer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Josef P. Kapfhammer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Josef P. Kapfhammer. The network helps show where Josef P. Kapfhammer may publish in the future.

Co-authorship network of co-authors of Josef P. Kapfhammer

This figure shows the co-authorship network connecting the top 25 collaborators of Josef P. Kapfhammer. A scholar is included among the top collaborators of Josef P. Kapfhammer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Josef P. Kapfhammer. Josef P. Kapfhammer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kapfhammer, Josef P., et al.. (2023). Viewpoint: spinocerebellar ataxias as diseases of Purkinje cell dysfunction rather than Purkinje cell loss. Frontiers in Molecular Neuroscience. 16. 1182431–1182431. 10 indexed citations

Kapfhammer, Josef P., et al.. (2022). CRISPR-Cas13-Mediated Knockdown of Regulator of G-Protein Signaling 8 (RGS8) Does Not Affect Purkinje Cell Dendritic Development. Frontiers in Cell and Developmental Biology. 10. 854273–854273. 2 indexed citations

Kapfhammer, Josef P., et al.. (2021). Serine/threonine kinase 17b (STK17B) signalling regulates Purkinje cell dendritic development and is altered in multiple spinocerebellar ataxias. European Journal of Neuroscience. 54(7). 6673–6684. 13 indexed citations

Kapfhammer, Josef P., et al.. (2020). The CRISPR-Cas13a system interferes with Purkinje cell dendritic development. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1867(7). 118710–118710. 9 indexed citations

Jäger, Levy, et al.. (2018). Reduced Purkinje cell size is compatible with near normal morphology and function of the cerebellar cortex in a mouse model of spinocerebellar ataxia. Experimental Neurology. 311. 205–212. 13 indexed citations

Kapfhammer, Josef P., et al.. (2017). Increased biological activity of protein Kinase C gamma is not required in Spinocerebellar ataxia 14. Molecular Brain. 10(1). 34–34. 18 indexed citations

Chip, Sophorn, Xinzhou Zhu, & Josef P. Kapfhammer. (2014). The Analysis of Neurovascular Remodeling in Entorhino-hippocampal Organotypic Slice Cultures. Journal of Visualized Experiments. 3 indexed citations

Radojevic, Vesna, et al.. (2011). The somatostatinergic system in the mammalian cochlea. BMC Neuroscience. 12(1). 89–89. 8 indexed citations

Gajendran, Nadesan, Josef P. Kapfhammer, Marco Canepari, et al.. (2009). Neuregulin Signaling Is Dispensable for NMDA- and GABA_A-Receptor Expression in the CerebellumIn Vivo. Journal of Neuroscience. 29(8). 2404–2413. 19 indexed citations

10.

Kapfhammer, Josef P., et al.. (2009). Reduced size of the dendritic tree does not protect Purkinje cells from excitotoxic death. Journal of Neuroscience Research. 88(4). 774–783. 4 indexed citations

11.

Kapfhammer, Josef P., et al.. (2008). Spontaneous regeneration of intrinsic spinal cord axons in a novel spinal cord slice culture model. European Journal of Neuroscience. 27(10). 2483–2492. 30 indexed citations

12.

Bendfeldt, Kerstin, Vesna Radojevic, Josef P. Kapfhammer, & Cordula Nitsch. (2007). Basic Fibroblast Growth Factor Modulates Density of Blood Vessels and Preserves Tight Junctions in Organotypic Cortical Cultures of Mice: A NewIn VitroModel of the Blood–Brain Barrier. Journal of Neuroscience. 27(12). 3260–3267. 58 indexed citations

13.

Ferrer, Isidró, Josef P. Kapfhammer, C. Hindelang, et al.. (2005). Inactivation of the peroxisomal ABCD2 transporter in the mouse leads to late-onset ataxia involving mitochondria, Golgi and endoplasmic reticulum damage. Human Molecular Genetics. 14(23). 3565–3577. 86 indexed citations

14.

Kapfhammer, Josef P.. (2004). Cellular and molecular control of dendritic growth and development of cerebellar Purkinje cells. Progress in Histochemistry and Cytochemistry. 39(3). 131–182. 92 indexed citations

15.

Kapfhammer, Josef P., et al.. (2002). Altered dendritic development of cerebellar Purkinje cells in slice cultures from protein kinase Cγ-deficient mice. Neuroscience. 110(4). 675–689. 77 indexed citations

16.

Prang, Peter, Domenico Del Turco, & Josef P. Kapfhammer. (2001). Regeneration of Entorhinal Fibers in Mouse Slice Cultures Is Age Dependent and Can Be Stimulated by NT-4, GDNF, and Modulators of G-Proteins and Protein Kinase C. Experimental Neurology. 169(1). 135–147. 44 indexed citations

17.

Thallmair, Michaela, et al.. (1998). Increased lesion‐induced sprouting of corticospinal fibres in the myelin‐free rat spinal cord. European Journal of Neuroscience. 10(1). 45–56. 46 indexed citations

18.

Kapfhammer, Josef P. & Martin E. Schwab. (1994). Inverse patterns of myelination and GAP‐43 expression in the adult CNS: Neurite growth inhibitors as regulators of neuronal plasticity?. The Journal of Comparative Neurology. 340(2). 194–206. 169 indexed citations

19.

Kapfhammer, Josef P., Franziska Christ, & Martin E. Schwab. (1994). The expression of GAP-43 and synaptophysin in the developing rat retina. Developmental Brain Research. 80(1-2). 251–260. 43 indexed citations

20.

Schwab, Martin E., Josef P. Kapfhammer, & C. E. Bandtlow. (1993). Inhibitors of Neurite Growth. Annual Review of Neuroscience. 16(1). 565–595. 467 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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