Jonathan R. Friedman
- Molecular Biology top 2%
- Cell Biology top 0.5%
- Epidemiology top 5%
- Clinical Biochemistry top 0.5%
- Physiology top 5%
- Co-authors
- Jodi NunnariGia K. VoeltzMatthew WestLaura L. LacknerDavid N. MastronardeKristen J. VerheyBrant M. WebsterMartin Graef
- Topics
- Mitochondrial Function and Pathology (19 papers)ATP Synthase and ATPases Research (10 papers)Metabolism and Genetic Disorders (7 papers)
- Journals
- NatureScienceNature Communications
- Partner nations
- United StatesGermanyFrance
In The Last Decade
Jonathan R. Friedman
34 papers receiving 5.1k citations
Hit Papers
Peers
Comparison fields: 5 of 135
- Molecular Biology 3.7k
- Cell Biology 1.3k
- Epidemiology 949
- Clinical Biochemistry 764
- Physiology 551
Countries citing papers authored by Jonathan R. Friedman
This map shows the geographic impact of Jonathan R. Friedman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan R. Friedman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan R. Friedman more than expected).
Fields of papers citing papers by Jonathan R. Friedman
This network shows the impact of papers produced by Jonathan R. Friedman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan R. Friedman. The network helps show where Jonathan R. Friedman may publish in the future.
Co-authorship network of co-authors of Jonathan R. Friedman
This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan R. Friedman. A scholar is included among the top collaborators of Jonathan R. Friedman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan R. Friedman. Jonathan R. Friedman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 10 | |
| 3 | 4 | |
| 4 | 14 | |
| 5 | 26 | |
| 6 | 18 | |
| 7 | 5 | |
| 8 | 3 | |
| 9 | 2 | |
| 10 | 8 | |
| 11 | 43 | |
| 12 | 26 | |
| 13 | 37 | |
| 14 | 74 | |
| 15 | Mitochondrial form and functionbreakdown → | 1358 |
| 16 | 287 | |
| 17 | ER Tubules Mark Sites of Mitochondrial Divisionbreakdown → | 1598 |
| 18 | 236 | |
| 19 | 113 | |
| 20 | 15 |
About Jonathan R. Friedman
Jonathan R. Friedman is a scholar working on Clinical Biochemistry, Endocrinology and Physiology, having authored 35 papers that have together received 5.1k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (19 papers), ATP Synthase and ATPases Research (10 papers) and Metabolism and Genetic Disorders (7 papers). The work is most often cited by research in Clinical Biochemistry (764 citations), Cell Biology (1.3k citations) and Molecular Biology (3.7k citations). Jonathan R. Friedman has collaborated with scholars based in United States, Germany and France. Frequent co-authors include Jodi Nunnari, Gia K. Voeltz, Matthew West, Laura L. Lackner, David N. Mastronarde, Kristen J. Verhey, Brant M. Webster, Martin Graef, Mohan Babu and Christopher I. Graham. Their work appears in journals such as Nature, Science and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.