Jonathan L. Blank

4.9k total citations · 1 hit paper
40 papers, 4.1k citations indexed

About

Jonathan L. Blank is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Jonathan L. Blank has authored 40 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 11 papers in Oncology and 9 papers in Cell Biology. Recurrent topics in Jonathan L. Blank's work include Protein Kinase Regulation and GTPase Signaling (20 papers), Ubiquitin and proteasome pathways (14 papers) and Receptor Mechanisms and Signaling (9 papers). Jonathan L. Blank is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (20 papers), Ubiquitin and proteasome pathways (14 papers) and Receptor Mechanisms and Signaling (9 papers). Jonathan L. Blank collaborates with scholars based in United States, United Kingdom and Netherlands. Jonathan L. Blank's co-authors include John H. Exton, Karl Deacon, Carsten Hagemann, Elliott M. Ross, Gabriel Berstein, Johannes L. Bos, Miranda van Triest, Frank Schwede, Stein Ove Døskeland and Anne Elisabeth Christensen and has published in prestigious journals such as Science, Cell and Journal of Biological Chemistry.

In The Last Decade

Jonathan L. Blank

40 papers receiving 4.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A novel Epac-specific cAMP analogue demonstrates independent regulation of Rap1 and ERK

2002 611 citations Jorrit M. Enserink, Anne Elisabeth Christensen et al. Nature Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jonathan L. Blank United States	29	3.4k	790	765	558	308	40	4.1k
Hilary McLauchlan United Kingdom	13	3.2k 0.9×	537 0.7×	775 1.0×	821 1.5×	168 0.5×	16	4.6k
Thierry Grand‐Perret France	14	2.2k 0.6×	455 0.6×	742 1.0×	529 0.9×	125 0.4×	20	3.5k
C. James Hastie United Kingdom	26	3.0k 0.9×	440 0.6×	649 0.8×	650 1.2×	115 0.4×	37	4.2k
Keejung Yoon South Korea	29	3.1k 0.9×	492 0.6×	420 0.5×	653 1.2×	234 0.8×	74	4.5k
Robert Amson France	27	2.0k 0.6×	571 0.7×	762 1.0×	234 0.4×	167 0.5×	40	3.2k
Adriano Marchese United States	33	2.8k 0.8×	919 1.2×	1.0k 1.3×	752 1.3×	156 0.5×	62	4.2k
Steven H. Nye United States	15	2.2k 0.6×	1.2k 1.5×	567 0.7×	349 0.6×	135 0.4×	22	3.8k
William J. Pitts United States	20	2.4k 0.7×	385 0.5×	673 0.9×	304 0.5×	130 0.4×	34	4.4k
Michiaki Kohno Japan	35	3.1k 0.9×	340 0.4×	846 1.1×	523 0.9×	122 0.4×	92	4.5k
Jonathan R. Whitfield United Kingdom	19	3.0k 0.9×	936 1.2×	897 1.2×	302 0.5×	102 0.3×	34	4.0k

Countries citing papers authored by Jonathan L. Blank

Since Specialization

Citations

This map shows the geographic impact of Jonathan L. Blank's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan L. Blank with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan L. Blank more than expected).

Fields of papers citing papers by Jonathan L. Blank

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan L. Blank. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan L. Blank. The network helps show where Jonathan L. Blank may publish in the future.

Co-authorship network of co-authors of Jonathan L. Blank

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan L. Blank. A scholar is included among the top collaborators of Jonathan L. Blank based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan L. Blank. Jonathan L. Blank is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Garcia, Khristofer, Jonathan L. Blank, David C. Bouck, et al.. (2014). Nedd8-Activating Enzyme Inhibitor MLN4924 Provides Synergy with Mitomycin C through Interactions with ATR, BRCA1/BRCA2, and Chromatin Dynamics Pathways. Molecular Cancer Therapeutics. 13(6). 1625–1635. 45 indexed citations

Niewerth, Denise, Gertjan J.L. Kaspers, Yehuda G. Assaraf, et al.. (2014). Interferon-γ-induced upregulation of immunoproteasome subunit assembly overcomes bortezomib resistance in human hematological cell lines. Journal of Hematology & Oncology. 7(1). 7–7. 65 indexed citations

Blank, Jonathan L., Katherine Cosmopoulos, David C. Bouck, et al.. (2012). Novel DNA Damage Checkpoints Mediating Cell Death Induced by the NEDD8-Activating Enzyme Inhibitor MLN4924. Cancer Research. 73(1). 225–234. 85 indexed citations

Kupperman, Erik, Edmund C. Lee, Yueying Cao, et al.. (2010). Evaluation of the Proteasome Inhibitor MLN9708 in Preclinical Models of Human Cancer. Cancer Research. 70(5). 1970–1980. 409 indexed citations

Blackburn, Christopher, Jonathan L. Blank, Frank J. Bruzzese, et al.. (2010). Optimization of a series of dipeptides with a P3 threonine residue as non-covalent inhibitors of the chymotrypsin-like activity of the human 20S proteasome. Bioorganic & Medicinal Chemistry Letters. 20(22). 6581–6586. 39 indexed citations

Nathan, Meena, et al.. (2010). The Use of Extracorporeal Membrane Oxygenation, High-Frequency Oscillatory Ventilation, and Surfactant for the Management of Intraoperative Acute Pulmonary Hemorrhage in Tetralogy of Fallot. Journal of Cardiothoracic and Vascular Anesthesia. 25(2). 313–315. 2 indexed citations

Williamson, Mark J., Jonathan L. Blank, Frank J. Bruzzese, et al.. (2006). Comparison of biochemical and biological effects of ML858 (salinosporamide A) and bortezomib. Molecular Cancer Therapeutics. 5(12). 3052–3061. 57 indexed citations

Mistry, Pratibha, Karl Deacon, Sharad Mistry, Jonathan L. Blank, & Rajni V. Patel. (2004). NF-κB Promotes Survival during Mitotic Cell Cycle Arrest. Journal of Biological Chemistry. 279(2). 1482–1490. 46 indexed citations

Deacon, Karl, Pratibha Mistry, Jonathan Chernoff, Jonathan L. Blank, & R. M. Patel. (2003). p38 Mitogen-Activated Protein Kinase Mediates Cell Death and p21-Activated Kinase Mediates Cell Survival during Chemotherapeutic Drug-induced Mitotic Arrest. Molecular Biology of the Cell. 14(5). 2071–2087. 153 indexed citations

10.

Hornigold, David C., et al.. (2003). Evidence for cross‐talk between M₂ and M₃ muscarinic acetylcholine receptors in the regulation of second messenger and extracellular signal‐regulated kinase signalling pathways in Chinese hamster ovary cells. British Journal of Pharmacology. 138(7). 1340–1350. 30 indexed citations

11.

Enserink, Jorrit M., Anne Elisabeth Christensen, Johan de Rooij, et al.. (2002). A novel Epac-specific cAMP analogue demonstrates independent regulation of Rap1 and ERK. Nature Cell Biology. 4(11). 901–906. 611 indexed citations breakdown →

12.

Hagemann, Carsten & Jonathan L. Blank. (2001). The ups and downs of MEK kinase interactions. Cellular Signalling. 13(12). 863–875. 232 indexed citations

13.

Deacon, Karl & Jonathan L. Blank. (1999). MEK Kinase 3 Directly Activates MKK6 and MKK7, Specific Activators of the p38 and c-Jun NH2-terminal Kinases. Journal of Biological Chemistry. 274(23). 16604–16610. 82 indexed citations

14.

Dickenson, John M., Jonathan L. Blank, & Stephen J. Hill. (1998). Human adenosine A₁ receptor and P2Y₂‐purinoceptor‐mediated activation of the mitogen‐activated protein kinase cascade in transfected CHO cells. British Journal of Pharmacology. 124(7). 1491–1499. 67 indexed citations

15.

Deacon, Karl & Jonathan L. Blank. (1997). Characterization of the Mitogen-activated Protein Kinase Kinase 4 (MKK4)/c-Jun NH2-terminal kinase 1 and MKK3/p38 Pathways Regulated by MEK Kinases 2 and 3. Journal of Biological Chemistry. 272(22). 14489–14496. 100 indexed citations

16.

Blank, Jonathan L., et al.. (1996). Molecular Cloning of Mitogen-activated Protein/ERK Kinase Kinases (MEKK) 2 and 3. Journal of Biological Chemistry. 271(10). 5361–5368. 201 indexed citations

17.

Blank, Jonathan L. & John H. Exton. (1994). [19] Purification of 110 kDa phosphoinositide phospholipase C activated by G-protein βλ subunits. Methods in enzymology on CD-ROM/Methods in enzymology. 238. 237–244. 2 indexed citations

18.

Blank, Jonathan L. & John H. Exton. (1994). [14] Purification of activated and heterotrimeric forms of Gq proteins. Methods in enzymology on CD-ROM/Methods in enzymology. 237. 174–181. 5 indexed citations

19.

Berstein, Gabriel, Jonathan L. Blank, Deok-Young Jhon, et al.. (1992). Phospholipase C-β1 is a GTPase-activating protein for Gq/11, its physiologic regulator. Cell. 70(3). 411–418. 355 indexed citations

20.

Blank, Jonathan L., Keith Foster, & J. N. Hawthorne. (1991). Purification, Properties, and Phosphorylation by Protein Kinase C of Two Phosphoinositidase C Isozymes from Rat Brain. Journal of Neurochemistry. 57(1). 15–21. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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