Jonathan E. Pottle
Impact in
- Sensory Systems top 5%
- Ion Channels and Receptors
- Physiology top 10%
- Calcium signaling and nucleotide metabolism
Papers in ⓘ
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- Ion channel regulation and function 5
- Nicotinic Acetylcholine Receptors Study 2
- Pharmacological Receptor Mechanisms and Effects 1
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- Ion Channels and Receptors 4
- Co-authors
- James T. Taylor (3 shared papers)Suresh C. Sikka (2 shared papers)Xiangbin Zeng (2 shared papers)Alun R. Wang (1 shared paper)Ming Li (2 shared papers)Kevin Lee (1 shared paper)Dongsheng Jia (1 shared paper)Asim B. Abdel‐Mageed (1 shared paper)
- Journals
- Cancer Letters (1 paper)Prostate Cancer and Prostatic Diseases (1 paper)World Journal of Gastroenterology (1 paper)Cancer Research (1 paper)Journal of Cancer Therapy (1 paper)
- Partner nations
- United StatesChinaDenmark
In The Last Decade
Jonathan E. Pottle
4 papers receiving 337 citations
Peers
Comparison fields: 5 of 61
- Sensory Systems 128
- Physiology 29
- Toxicology 17
- Molecular Biology 254
- Cellular and Molecular Neuroscience 61
Countries citing papers authored by Jonathan E. Pottle
This map shows the geographic impact of Jonathan E. Pottle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan E. Pottle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan E. Pottle more than expected).
Fields of papers citing papers by Jonathan E. Pottle
This network shows the impact of papers produced by Jonathan E. Pottle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan E. Pottle. The network helps show where Jonathan E. Pottle may publish in the future.
Co-authors
The 20 scholars most cited alongside Jonathan E. Pottle, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 133 | |
| 2 | 2009 | 101 | |
| 3 | 2008 | 88 | |
| 4 | 2013 | 17 | |
| 5 | 2012 | 2 |
About Jonathan E. Pottle
Jonathan E. Pottle is a scholar working on Molecular Biology, Sensory Systems, Cellular and Molecular Neuroscience, Immunology and Infectious Diseases, having authored 5 papers that have together received 341 indexed citations. Recurring topics across this work include Ion channel regulation and function (5 papers), Ion Channels and Receptors (4 papers), Nicotinic Acetylcholine Receptors Study (2 papers), Pharmacological Receptor Mechanisms and Effects (1 paper), Toxin Mechanisms and Immunotoxins (1 paper), Neuroscience and Neural Engineering (1 paper) and Neuroscience and Neuropharmacology Research (1 paper). The work is most often cited by research in Sensory Systems (128 citations), Physiology (29 citations), Toxicology (17 citations), Molecular Biology (254 citations) and Cellular and Molecular Neuroscience (61 citations). Jonathan E. Pottle has collaborated with scholars based in United States, China and Denmark. Frequent co-authors include James T. Taylor, Suresh C. Sikka, Xiangbin Zeng, Alun R. Wang, Ming Li, Kevin Lee, Dongsheng Jia, Asim B. Abdel‐Mageed, Chunyan Xu and Linglin Huang. Their work appears in journals such as Cancer Letters, Prostate Cancer and Prostatic Diseases, World Journal of Gastroenterology, Cancer Research and Journal of Cancer Therapy.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.