Jonathan E. Campbell

4.8k total citations · 1 hit paper
40 papers, 3.3k citations indexed

About

Jonathan E. Campbell is a scholar working on Endocrinology, Diabetes and Metabolism, Physiology and Surgery. According to data from OpenAlex, Jonathan E. Campbell has authored 40 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Endocrinology, Diabetes and Metabolism, 18 papers in Physiology and 14 papers in Surgery. Recurrent topics in Jonathan E. Campbell's work include Adipose Tissue and Metabolism (17 papers), Pancreatic function and diabetes (13 papers) and Diabetes Treatment and Management (12 papers). Jonathan E. Campbell is often cited by papers focused on Adipose Tissue and Metabolism (17 papers), Pancreatic function and diabetes (13 papers) and Diabetes Treatment and Management (12 papers). Jonathan E. Campbell collaborates with scholars based in Canada, United States and United Kingdom. Jonathan E. Campbell's co-authors include Daniel J. Drucker, Michael C. Riddell, Laurie L. Baggio, Erin E. Mulvihill, Sergiu Fediuc, Anna M. D'souza, Xiemin Cao, Thomas J. Hawke, John R. Ussher and Ashley J. Peckett and has published in prestigious journals such as Nature Medicine, PLoS ONE and Cell Metabolism.

In The Last Decade

Jonathan E. Campbell

39 papers receiving 3.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Pharmacology, Physiology, and Mechanisms of Incretin Hormone Action

2013 1.1k citations Jonathan E. Campbell, Daniel J. Drucker Cell Metabolism profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jonathan E. Campbell Canada	26	1.9k	1.2k	1.0k	909	383	40	3.3k
Maria Sörhede Winzell Sweden	28	1.2k 0.6×	1.3k 1.1×	1.1k 1.0×	956 1.1×	325 0.8×	54	3.0k
Brian Finan United States	32	1.9k 1.0×	1.0k 0.8×	1.4k 1.3×	1.1k 1.2×	498 1.3×	57	3.7k
Kenju Shimomura Japan	32	909 0.5×	1.2k 1.0×	1.3k 1.3×	601 0.7×	227 0.6×	122	3.5k
Anita M. Hennige Germany	36	1.0k 0.5×	874 0.7×	1.4k 1.3×	1.3k 1.4×	207 0.5×	78	3.8k
Doss W. Neal United States	37	2.3k 1.2×	2.1k 1.7×	1.2k 1.2×	1.5k 1.7×	187 0.5×	134	4.4k
Julio E. Ayala United States	31	894 0.5×	972 0.8×	1.6k 1.6×	1.6k 1.8×	157 0.4×	65	3.7k
Phillip E. Williams United States	36	1.1k 0.6×	1.2k 1.0×	777 0.7×	1.4k 1.5×	172 0.4×	110	3.3k
Dominic P. Behan United States	31	1.6k 0.8×	598 0.5×	1.1k 1.1×	686 0.8×	823 2.1×	60	4.4k
Mladen Vranić Canada	37	2.0k 1.0×	1.5k 1.2×	1.3k 1.3×	1.6k 1.8×	138 0.4×	96	4.4k
Carla Roberta de Oliveira Carvalho Brazil	33	1.1k 0.6×	685 0.6×	1.3k 1.2×	1.5k 1.6×	136 0.4×	117	3.9k

Countries citing papers authored by Jonathan E. Campbell

Since Specialization

Citations

This map shows the geographic impact of Jonathan E. Campbell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan E. Campbell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan E. Campbell more than expected).

Fields of papers citing papers by Jonathan E. Campbell

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan E. Campbell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan E. Campbell. The network helps show where Jonathan E. Campbell may publish in the future.

Co-authorship network of co-authors of Jonathan E. Campbell

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan E. Campbell. A scholar is included among the top collaborators of Jonathan E. Campbell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan E. Campbell. Jonathan E. Campbell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Feldman, Marissa A., et al.. (2023). Competencies in Telepsychology: A Developmental Framework for Psychology Training and Professional Preparation. Journal of Technology in Behavioral Science. 9(1). 20–25.

Rajagopal, Deepa, Linhua Tian, Shiqiu Xiong, et al.. (2020). An accurate and rapid single step protocol for enumeration of cytokine positive T lymphocytes. PubMed. 9. 100032–100032. 2 indexed citations

Beaudry, Jacqueline L., Kiran Deep Kaur, Elodie M. Varin, et al.. (2019). The brown adipose tissue glucagon receptor is functional but not essential for control of energy homeostasis in mice. Molecular Metabolism. 22. 37–48. 68 indexed citations

Beaudry, Jacqueline L., Kiran Deep Kaur, Elodie M. Varin, et al.. (2019). Physiological roles of the GIP receptor in murine brown adipose tissue. Molecular Metabolism. 28. 14–25. 50 indexed citations

Batran, Rami Al, K. Lockhart Jamieson, Ahmed M. Darwesh, et al.. (2019). l‐Citrulline supplementation improves glucose and exercise tolerance in obese male mice. Experimental Physiology. 105(2). 270–281. 14 indexed citations

Batran, Rami Al, Keshav Gopal, Mackenzie D. Martin, et al.. (2018). Skeletal muscle-specific Cre recombinase expression, controlled by the human α-skeletal actin promoter, improves glucose tolerance in mice fed a high-fat diet. Diabetologia. 61(8). 1849–1855. 8 indexed citations

Mulvihill, Erin E., Elodie M. Varin, Jonathan E. Campbell, et al.. (2016). Cellular Sites and Mechanisms Linking Reduction of Dipeptidyl Peptidase-4 Activity to Control of Incretin Hormone Action and Glucose Homeostasis. Cell Metabolism. 25(1). 152–165. 80 indexed citations

Campbell, Jonathan E. & Daniel J. Drucker. (2015). Islet α cells and glucagon—critical regulators of energy homeostasis. Nature Reviews Endocrinology. 11(6). 329–338. 206 indexed citations

Ussher, John R., Laurie L. Baggio, Jonathan E. Campbell, et al.. (2014). Inactivation of the cardiomyocyte glucagon-like peptide-1 receptor (GLP-1R) unmasks cardiomyocyte-independent GLP-1R-mediated cardioprotection. Molecular Metabolism. 3(5). 507–517. 106 indexed citations

10.

Campbell, Jonathan E. & Daniel J. Drucker. (2013). Pharmacology, Physiology, and Mechanisms of Incretin Hormone Action. Cell Metabolism. 17(6). 819–837. 1136 indexed citations breakdown →

11.

Beaudry, Jacqueline L., et al.. (2011). A rodent model of rapid-onset diabetes (ROD) induced by glucocorticoids and high-fat feeding. Disease Models & Mechanisms. 5(5). 671–80. 67 indexed citations

12.

Bates, Holly E., Jonathan E. Campbell, John R. Ussher, et al.. (2011). Gipr Is Essential for Adrenocortical Steroidogenesis; However, Corticosterone Deficiency Does Not Mediate the Favorable Metabolic Phenotype of Gipr−/− Mice. Diabetes. 61(1). 40–48. 36 indexed citations

13.

Campbell, Jonathan E., et al.. (2011). Skeletal muscle protein synthesis and the abundance of the mRNA translation initiation repressor PDCD4 are inversely regulated by fasting and refeeding in rats. American Journal of Physiology-Endocrinology and Metabolism. 300(6). E986–E992. 18 indexed citations

14.

Krause, Matthew P., Jonathan E. Campbell, E. Cafarelli, et al.. (2010). Impaired Growth and Force Production in Skeletal Muscles of Young Partially Pancreatectomized Rats: A Model of Adolescent Type 1 Diabetic Myopathy?. PLoS ONE. 5(11). e14032–e14032. 21 indexed citations

15.

Campbell, Jonathan E., Sergiu Fediuc, Thomas J. Hawke, & Michael C. Riddell. (2009). Endurance exercise training increases adipose tissue glucocorticoid exposure: adaptations that facilitate lipolysis. Metabolism. 58(5). 651–660. 39 indexed citations

16.

Campbell, Jonathan E., et al.. (2008). Voluntary wheel running initially increases adrenal sensitivity to adrenocorticotrophic hormone, which is attenuated with long-term training. Journal of Applied Physiology. 106(1). 66–72. 44 indexed citations

17.

Campbell, Jonathan E., et al.. (2006). Efficacy of Continuous Real-Time Blood Glucose Monitoring During and After Prolonged High-Intensity Cycling Exercise: Spinning with a Continuous Glucose Monitoring System. Diabetes Technology & Therapeutics. 8(6). 627–635. 74 indexed citations

18.

Coutinho, Agnes E., Sergiu Fediuc, Jonathan E. Campbell, & Michael C. Riddell. (2006). Metabolic effects of voluntary wheel running in young and old Syrian golden hamsters. Physiology & Behavior. 87(2). 360–367. 14 indexed citations

19.

Fediuc, Sergiu, Jonathan E. Campbell, & Michael C. Riddell. (2006). Effect of voluntary wheel running on circadian corticosterone release and on HPA axis responsiveness to restraint stress in Sprague-Dawley rats. Journal of Applied Physiology. 100(6). 1867–1875. 112 indexed citations

20.

Coutinho, Agnes E., Jonathan E. Campbell, Sergiu Fediuc, & Michael C. Riddell. (2005). Effect of voluntary exercise on peripheral tissue glucocorticoid receptor content and the expression and activity of 11β-HSD1 in the Syrian hamster. Journal of Applied Physiology. 100(5). 1483–1488. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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