Jon Weil

2.2k total citations
52 papers, 1.7k citations indexed

About

Jon Weil is a scholar working on Genetics, Molecular Biology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Jon Weil has authored 52 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Genetics, 18 papers in Molecular Biology and 17 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Jon Weil's work include Bacteriophages and microbial interactions (13 papers), Bacterial Genetics and Biotechnology (11 papers) and BRCA gene mutations in cancer (8 papers). Jon Weil is often cited by papers focused on Bacteriophages and microbial interactions (13 papers), Bacterial Genetics and Biotechnology (11 papers) and BRCA gene mutations in cancer (8 papers). Jon Weil collaborates with scholars based in United States, Australia and New Zealand. Jon Weil's co-authors include Ethan R. Signer, David Henderson, Lois B. Epstein, Charles J. Epstein, Theodore Homyk, J. J. Sedmak, Sidney E. Grossberg, J L Sabran, C. J. Epstein and Betty E. Terzaghi and has published in prestigious journals such as Nature, SHILAP Revista de lepidopterología and Journal of Molecular Biology.

In The Last Decade

Jon Weil

51 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jon Weil United States 23 836 786 557 244 166 52 1.7k
Elizabeth M. Tucker Slovakia 26 715 0.9× 527 0.7× 49 0.1× 130 0.5× 175 1.1× 119 2.3k
Kevin M. Sullivan United States 30 1.2k 1.4× 909 1.2× 150 0.3× 133 0.5× 236 1.4× 72 3.3k
Sérgio D. J. Pena Brazil 18 577 0.7× 872 1.1× 126 0.2× 125 0.5× 236 1.4× 39 2.5k
Michael D. Edge United States 29 1.2k 1.4× 778 1.0× 129 0.2× 29 0.1× 229 1.4× 77 2.7k
Barbara J. Harris United States 26 784 0.9× 201 0.3× 91 0.2× 109 0.4× 53 0.3× 77 2.8k
Sigrún Sigurðardóttir Iceland 21 990 1.2× 1.3k 1.7× 49 0.1× 197 0.8× 410 2.5× 36 2.9k
Emilio García García Spain 25 1.2k 1.4× 1.0k 1.3× 316 0.6× 51 0.2× 87 0.5× 183 2.4k
Michael Clinton United Kingdom 33 1.4k 1.6× 1.1k 1.4× 88 0.2× 122 0.5× 122 0.7× 94 3.0k
Sarah Williams‐Blangero United States 22 204 0.2× 469 0.6× 403 0.7× 160 0.7× 67 0.4× 67 1.7k
P. L. Chambers United Kingdom 26 410 0.5× 403 0.5× 155 0.3× 24 0.1× 132 0.8× 78 2.1k

Countries citing papers authored by Jon Weil

Since Specialization
Citations

This map shows the geographic impact of Jon Weil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon Weil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon Weil more than expected).

Fields of papers citing papers by Jon Weil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon Weil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon Weil. The network helps show where Jon Weil may publish in the future.

Co-authorship network of co-authors of Jon Weil

This figure shows the co-authorship network connecting the top 25 collaborators of Jon Weil. A scholar is included among the top collaborators of Jon Weil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon Weil. Jon Weil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
McEwen, Alison, Allyson Mary Davys, & Jon Weil. (2025). Supervision for genetic counselors: The role of career‐long supervision to develop resilient practitioners. Journal of Genetic Counseling. 34(1). e70014–e70014. 3 indexed citations
2.
Abrams, Liane, et al.. (2024). Experiences of individuals receiving “Not Parent Expected” results through direct‐to‐consumer genetic testing. Journal of Genetic Counseling. 34(3). e1977–e1977. 1 indexed citations
3.
Weil, Jon, et al.. (2024). Promoting international, locally focused, and patient-oriented genetic counseling. SHILAP Revista de lepidopterología. 2(Suppl 2). 101880–101880. 1 indexed citations
4.
Ormond, Kelly E., et al.. (2023). International genetic counseling: What do genetic counselors actually do?. Journal of Genetic Counseling. 33(2). 382–391. 10 indexed citations
5.
Weil, Jon, Kelly E. Ormond, June A. Peters, et al.. (2006). The Relationship of Nondirectiveness to Genetic Counseling: Report of a Workshop at the 2003 NSGC Annual Education Conference. Journal of Genetic Counseling. 15(2). 85–93. 44 indexed citations
6.
Weil, Jon. (2003). Psychosocial Genetic Counseling in the Post‐Nondirective Era: A Point of View. Journal of Genetic Counseling. 12(3). 199–211. 101 indexed citations
7.
Habermann, CR, et al.. (2002). MRT in der postoperativen Diagnostik bei funktionell univentrikulärem Herz: Korrelation zu Echokardiographie und Kardangiographie. RöFo - Fortschritte auf dem Gebiet der Röntgenstrahlen und der bildgebenden Verfahren. 174(12). 1537–1543. 3 indexed citations
8.
Weil, Jon. (2001). Multicultural education and genetic counseling. Clinical Genetics. 59(3). 143–149. 44 indexed citations
9.
Weil, Jon. (2000). Introduction. Journal of Genetic Counseling. 9(5). 375–378. 12 indexed citations
10.
Weil, Jon, et al.. (1995). [Cutaneous pulpar temperature and cold test. Predictive specificity and sensitivity in pharmaco-clinical studies].. PubMed. 20(1). 38–44.
11.
Weil, Jon, Lois B. Epstein, & Charles J. Epstein. (1980). Synthesis of Interferon-Induced Polypeptides in Normal and Chromosome 21-Aneuploid Human Fibroblasts: Relationship to Relative Sensitivities in Antiviral Assays. Journal of Interferon Research. 1(1). 111–124. 36 indexed citations
12.
Henderson, David & Jon Weil. (1977). Morphogenesis of bacteriophage lambda deletion mutants. Journal of Molecular Biology. 113(1). 43–66. 8 indexed citations
13.
Henderson, David & Jon Weil. (1977). Morphogenesis of bacteriophage lambda deletion mutants. Journal of Molecular Biology. 113(1). 67–87. 1 indexed citations
14.
Bardos, P., et al.. (1976). [Glycoproteins in maternal and fetal blood and amniotic fluid. Variations in normal and pathologic pregnancies].. PubMed. 5(5). 633–6. 1 indexed citations
15.
Henderson, David & Jon Weil. (1975). The nature and origin of a class of essential gene substitutions in bacteriophage λ. Virology. 67(1). 124–135. 25 indexed citations
16.
Weil, Jon, et al.. (1975). Morphogenesis of λ with genomes containing excess DNA: Functional particles containing 12 and 15% excess DNA. Virology. 63(2). 352–366. 9 indexed citations
17.
Weil, Jon. (1969). Reciprocal and non-reciprocal recombination in bacteriophage λ. Journal of Molecular Biology. 43(2). 351–355. 21 indexed citations
18.
Weil, Jon & Ethan R. Signer. (1968). Recombination in bacteriophage λ. Journal of Molecular Biology. 34(2). 273–279. 97 indexed citations
19.
Echols, Harrison, et al.. (1968). The General Recombination System of Bacteriophage  . Cold Spring Harbor Symposia on Quantitative Biology. 33(0). 711–714. 37 indexed citations
20.
Signer, Ethan R. & Jon Weil. (1968). Recombination in bacteriophage λ. Journal of Molecular Biology. 34(2). 261–271. 251 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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