Jon Fernández‐Rueda

441 total citations
8 papers, 339 citations indexed

About

Jon Fernández‐Rueda is a scholar working on Oncology, Molecular Biology and Genetics. According to data from OpenAlex, Jon Fernández‐Rueda has authored 8 papers receiving a total of 339 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Oncology, 4 papers in Molecular Biology and 3 papers in Genetics. Recurrent topics in Jon Fernández‐Rueda's work include Mesenchymal stem cell research (3 papers), Tissue Engineering and Regenerative Medicine (2 papers) and Immune Response and Inflammation (2 papers). Jon Fernández‐Rueda is often cited by papers focused on Mesenchymal stem cell research (3 papers), Tissue Engineering and Regenerative Medicine (2 papers) and Immune Response and Inflammation (2 papers). Jon Fernández‐Rueda collaborates with scholars based in Spain, United Kingdom and China. Jon Fernández‐Rueda's co-authors include César Trigueros, Akaitz Dorronsoro, Valérie Lang, Pilar Sepúlveda, Emma Jakobsson, Karoline Fechter, Ana M. Zubiaga, Seth J. Field, Ivan Carcamo‐Orive and Jesús Delgado‐Calle and has published in prestigious journals such as Oncogene, Scientific Reports and Journal of Bone and Mineral Research.

In The Last Decade

Jon Fernández‐Rueda

8 papers receiving 335 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jon Fernández‐Rueda Spain 8 159 139 82 74 64 8 339
Rafael Contreras‐López France 8 112 0.7× 176 1.3× 51 0.6× 95 1.3× 51 0.8× 12 325
Shawn A. Hallett United States 11 283 1.8× 108 0.8× 85 1.0× 35 0.5× 63 1.0× 16 506
Menno ter Huurne Netherlands 10 271 1.7× 207 1.5× 49 0.6× 54 0.7× 65 1.0× 15 605
Monique Fangradt Germany 8 140 0.9× 98 0.7× 45 0.5× 123 1.7× 124 1.9× 9 451
Antonietta Fazio Italy 10 216 1.4× 139 1.0× 43 0.5× 35 0.5× 48 0.8× 23 458
Bart Vaes Netherlands 11 225 1.4× 161 1.2× 52 0.6× 36 0.5× 30 0.5× 17 493
Kevin Leclerc United States 8 145 0.9× 91 0.7× 36 0.4× 39 0.5× 44 0.7× 12 318
Karolina Kania United Kingdom 6 163 1.0× 109 0.8× 40 0.5× 37 0.5× 57 0.9× 8 412
Motokazu Tsuneto Japan 13 309 1.9× 75 0.5× 85 1.0× 138 1.9× 50 0.8× 37 530

Countries citing papers authored by Jon Fernández‐Rueda

Since Specialization
Citations

This map shows the geographic impact of Jon Fernández‐Rueda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon Fernández‐Rueda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon Fernández‐Rueda more than expected).

Fields of papers citing papers by Jon Fernández‐Rueda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon Fernández‐Rueda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon Fernández‐Rueda. The network helps show where Jon Fernández‐Rueda may publish in the future.

Co-authorship network of co-authors of Jon Fernández‐Rueda

This figure shows the co-authorship network connecting the top 25 collaborators of Jon Fernández‐Rueda. A scholar is included among the top collaborators of Jon Fernández‐Rueda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon Fernández‐Rueda. Jon Fernández‐Rueda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Dorronsoro, Akaitz, et al.. (2020). Intracellular role of IL-6 in mesenchymal stromal cell immunosuppression and proliferation. Scientific Reports. 10(1). 21853–21853. 60 indexed citations
2.
Dorronsoro, Akaitz, et al.. (2013). Human Mesenchymal Stromal Cell-Mediated Immunoregulation: Mechanisms of Action and Clinical Applications. PubMed. 2013. 1–8. 33 indexed citations
3.
Dorronsoro, Akaitz, et al.. (2013). Identification of the NF-κB inhibitor A20 as a key regulator for human adipogenesis. Cell Death and Disease. 4(12). e972–e972. 16 indexed citations
4.
Dorronsoro, Akaitz, Emma Jakobsson, Jon Fernández‐Rueda, et al.. (2013). Human mesenchymal stromal cells modulate T‐cell responses through TNF‐α‐mediated activation of NF‐κB. European Journal of Immunology. 44(2). 480–488. 74 indexed citations
5.
Fernández‐Rueda, Jon, et al.. (2012). Histone Deacetylase 3 Modulates the Expansion of Human Hematopoietic Stem Cells. Stem Cells and Development. 21(14). 2581–2591. 16 indexed citations
6.
Iglesias–Ara, Ainhoa, Olatz Zenarruzabeitia, Jon Fernández‐Rueda, et al.. (2010). Accelerated DNA replication in E2F1- and E2F2-deficient macrophages leads to induction of the DNA damage response and p21CIP1-dependent senescence. Oncogene. 29(41). 5579–5590. 22 indexed citations
7.
Carcamo‐Orive, Ivan, Jesús Delgado‐Calle, Akaitz Dorronsoro, et al.. (2010). Regulation of human bone marrow stromal cell proliferation and differentiation capacity by glucocorticoid receptor and AP-1 crosstalk. Journal of Bone and Mineral Research. 25(10). 2115–2125. 64 indexed citations
8.
Infante, Arantza, Usua Laresgoiti, Jon Fernández‐Rueda, et al.. (2008). E2F2 represses cell cycle regulators to maintain quiescence. Cell Cycle. 7(24). 3915–3927. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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