John T. Sladky
- Molecular Biology top 5%
- Neurology top 1%
- Cellular and Molecular Neuroscience top 2%
- Physiology top 5%
- Genetics top 5%
- Co-authors
- Mitsuo NarusawaHansell H. StedmanH. Lee SweeneyRA PanettieriA. M. KellyJoseph B. ShragerBJ PetrofMark J. Brown
- Topics
- Peripheral Neuropathies and Disorders (13 papers)Hereditary Neurological Disorders (12 papers)Mitochondrial Function and Pathology (7 papers)
- Partner nations
- United StatesCanadaItaly
In The Last Decade
John T. Sladky
58 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 113
- Molecular Biology 1.5k
- Neurology 908
- Cellular and Molecular Neuroscience 856
- Physiology 416
- Genetics 334
Countries citing papers authored by John T. Sladky
This map shows the geographic impact of John T. Sladky's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John T. Sladky with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John T. Sladky more than expected).
Fields of papers citing papers by John T. Sladky
This network shows the impact of papers produced by John T. Sladky. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John T. Sladky. The network helps show where John T. Sladky may publish in the future.
Co-authorship network of co-authors of John T. Sladky
This figure shows the co-authorship network connecting the top 25 collaborators of John T. Sladky. A scholar is included among the top collaborators of John T. Sladky based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John T. Sladky. John T. Sladky is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 3 | |
| 3 | 23 | |
| 4 | 128 | |
| 5 | 50 | |
| 6 | 166 | |
| 7 | 28 | |
| 8 | 1 | |
| 9 | 6 | |
| 10 | 18 | |
| 11 | 47 | |
| 12 | 10 | |
| 13 | 206 | |
| 14 | The mdx mouse diaphragm reproduces the degenerative changes of Duchenne muscular dystrophybreakdown → | 773 |
| 15 | 7 | |
| 16 | 16 | |
| 17 | 1 | |
| 18 | 34 | |
| 19 | Diagnosis and management of diseases affecting the motor unit in infancy. | 4 |
| 20 | 12 |
About John T. Sladky
John T. Sladky is a scholar working on Neurology, Cellular and Molecular Neuroscience and Neurology, having authored 59 papers that have together received 3.0k indexed citations. Recurring topics across this work include Peripheral Neuropathies and Disorders (13 papers), Hereditary Neurological Disorders (12 papers) and Mitochondrial Function and Pathology (7 papers). The work is most often cited by research in Neurology (908 citations), Cellular and Molecular Neuroscience (856 citations) and Rehabilitation (217 citations). John T. Sladky has collaborated with scholars based in United States, Canada and Italy. Frequent co-authors include Mitsuo Narusawa, Hansell H. Stedman, H. Lee Sweeney, RA Panettieri, A. M. Kelly, Joseph B. Shrager, BJ Petrof, Mark J. Brown, Joel Greenberg and Richard I. Kelley. Their work appears in journals such as Nature, Neuron and SHILAP Revista de lepidopterología.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.