John R. Glossop
- Molecular Biology
- Rheumatology top 5%
- Immunology
- Genetics top 10%
- Hematology top 10%
- Co-authors
- Derek L. MatteyP. T. DawesSarah H. CartmellNicola B NixonWilliam E. FarrellAnthony A. FryerRichard D. EmesJon Packham
- Topics
- Epigenetics and DNA Methylation (5 papers)Rheumatoid Arthritis Research and Therapies (5 papers)Mesenchymal stem cell research (4 papers)
- Cited by
- RheumatologyGeneticsHematology
- Partner nations
- United KingdomUnited StatesAustria
In The Last Decade
John R. Glossop
22 papers receiving 722 citations
Peers
Comparison fields: 5 of 88
- Molecular Biology 281
- Rheumatology 184
- Immunology 141
- Genetics 104
- Hematology 79
Countries citing papers authored by John R. Glossop
This map shows the geographic impact of John R. Glossop's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John R. Glossop with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John R. Glossop more than expected).
Fields of papers citing papers by John R. Glossop
This network shows the impact of papers produced by John R. Glossop. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John R. Glossop. The network helps show where John R. Glossop may publish in the future.
Co-authorship network of co-authors of John R. Glossop
This figure shows the co-authorship network connecting the top 25 collaborators of John R. Glossop. A scholar is included among the top collaborators of John R. Glossop based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John R. Glossop. John R. Glossop is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 50 | |
| 3 | 39 | |
| 4 | 53 | |
| 5 | 36 | |
| 6 | 46 | |
| 7 | 71 | |
| 8 | 26 | |
| 9 | 15 | |
| 10 | 2 | |
| 11 | 11 | |
| 12 | 81 | |
| 13 | 3 | |
| 14 | 35 | |
| 15 | 62 | |
| 16 | 45 | |
| 17 | Anemia in rheumatoid arthritis: association with polymorphism in the tumor necrosis factor receptor I and II genes. | 14 |
| 18 | No association of polymorphisms in the tumor necrosis factor receptor I and receptor II genes with disease severity in rheumatoid arthritis. | 21 |
| 19 | 39 | |
| 20 | 28 |
About John R. Glossop
John R. Glossop is a scholar working on Genetics, Rheumatology and Clinical Biochemistry, having authored 22 papers that have together received 737 indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (5 papers), Rheumatoid Arthritis Research and Therapies (5 papers) and Mesenchymal stem cell research (4 papers). The work is most often cited by research in Rheumatology (184 citations), Genetics (104 citations) and Hematology (79 citations). John R. Glossop has collaborated with scholars based in United Kingdom, United States and Austria. Frequent co-authors include Derek L. Mattey, P. T. Dawes, Sarah H. Cartmell, Nicola B Nixon, William E. Farrell, Anthony A. Fryer, Richard D. Emes, Jon Packham, Kim E. Haworth and I Smith. Their work appears in journals such as Human Molecular Genetics, Archives of Disease in Childhood and Lara D. Veeken.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.