John R. David

22.0k total citations · 6 hit papers
243 papers, 17.9k citations indexed

About

John R. David is a scholar working on Immunology, Public Health, Environmental and Occupational Health and Parasitology. According to data from OpenAlex, John R. David has authored 243 papers receiving a total of 17.9k indexed citations (citations by other indexed papers that have themselves been cited), including 125 papers in Immunology, 59 papers in Public Health, Environmental and Occupational Health and 40 papers in Parasitology. Recurrent topics in John R. David's work include Macrophage Migration Inhibitory Factor (95 papers), Research on Leishmaniasis Studies (53 papers) and Parasites and Host Interactions (38 papers). John R. David is often cited by papers focused on Macrophage Migration Inhibitory Factor (95 papers), Research on Leishmaniasis Studies (53 papers) and Parasites and Host Interactions (38 papers). John R. David collaborates with scholars based in United States, Brazil and United Kingdom. John R. David's co-authors include Barry R. Bloom, Ross E. Rocklin, Heinz G. Remold, A Butterworth, Abhay R. Satoskar, H. Sherwood Lawrence, L. Thomas, David E. Trentham, Carl Nathan and Salah Al‐Askari and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

John R. David

238 papers receiving 16.0k citations

Hit Papers

In vitro methods in cell-mediated and tumor immunity 1964 2026 1984 2005 1976 1966 2001 1964 1979 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John R. David United States 72 10.3k 3.4k 2.7k 2.2k 1.9k 243 17.9k
P Vassalli Switzerland 76 10.2k 1.0× 2.8k 0.8× 2.5k 1.0× 691 0.3× 6.1k 3.3× 209 22.4k
Martin Röllinghoff Germany 65 8.1k 0.8× 3.6k 1.1× 3.8k 1.4× 1.4k 0.6× 2.4k 1.3× 249 15.0k
Abhay R. Satoskar United States 61 6.4k 0.6× 3.6k 1.1× 3.0k 1.1× 1.7k 0.8× 1.9k 1.0× 272 13.5k
Kevin W. Moore United States 51 11.4k 1.1× 988 0.3× 3.2k 1.2× 612 0.3× 3.7k 2.0× 98 20.0k
Bernhard Fleischer Germany 59 5.2k 0.5× 1.6k 0.5× 2.8k 1.0× 1.5k 0.7× 1.9k 1.0× 299 12.0k
A Capron France 73 5.5k 0.5× 3.5k 1.0× 3.3k 1.2× 9.3k 4.2× 3.3k 1.8× 598 21.4k
David M. Mosser United States 58 10.3k 1.0× 3.6k 1.1× 4.0k 1.5× 964 0.4× 5.2k 2.8× 132 21.0k
Timothy R. Mosmann Canada 37 10.8k 1.1× 1.8k 0.5× 2.9k 1.1× 1.0k 0.5× 2.2k 1.2× 64 17.1k
Manfred Köpf Switzerland 91 14.6k 1.4× 1.3k 0.4× 3.7k 1.4× 1.1k 0.5× 5.6k 3.0× 234 25.3k
Hans J. Müller‐Eberhard United States 89 15.6k 1.5× 1.1k 0.3× 1.7k 0.6× 715 0.3× 5.7k 3.1× 283 25.1k

Countries citing papers authored by John R. David

Since Specialization
Citations

This map shows the geographic impact of John R. David's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John R. David with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John R. David more than expected).

Fields of papers citing papers by John R. David

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John R. David. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John R. David. The network helps show where John R. David may publish in the future.

Co-authorship network of co-authors of John R. David

This figure shows the co-authorship network connecting the top 25 collaborators of John R. David. A scholar is included among the top collaborators of John R. David based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John R. David. John R. David is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pereira, Hernane Borges de Barros, Fúlvio A. Scorza, Antônio-Carlos G. de Almeida, et al.. (2022). Retrospective Cohort Study of COVID-19 in Patients of the Brazilian Public Health System with SARS-CoV-2 Omicron Variant Infection. Vaccines. 10(9). 1504–1504. 3 indexed citations
2.
McDevitt, Michael A., Jianlin Xie, Shanmugasundaram Ganapathy‐Kanniappan, et al.. (2006). A critical role for the host mediator macrophage migration inhibitory factor in the pathogenesis of malarial anemia. The Journal of Experimental Medicine. 203(5). 1185–1196. 108 indexed citations
3.
Jose, Matthew, John R. David, Robert C. Atkins, & Steven J. Chadban. (2003). Blockade of Macrophage Migration Inhibitory Factor Does Not Prevent Acute Renal Allograft Rejection. American Journal of Transplantation. 3(9). 1099–1106. 19 indexed citations
4.
Rodríguez‐Sosa, Miriam, John R. David, Rafael Bojalil, Abhay R. Satoskar, & Luis I. Terrazas. (2002). Cutting Edge: Susceptibility to the Larval Stage of the Helminth Parasite Taenia crassiceps Is Mediated by Th2 Response Induced Via STAT6 Signaling. The Journal of Immunology. 168(7). 3135–3139. 63 indexed citations
5.
Shoemaker, Charles B., et al.. (2001). Sandfly Maxadilan Exacerbates Infection with Leishmania major and Vaccinating Against It Protects Against L. major Infection. The Journal of Immunology. 167(9). 5226–5230. 176 indexed citations
6.
Satoskar, Abhay R., Scott J. Rodig, Sam R. Telford, et al.. (2000). IL-12 gene-deficient C57BL / 6 mice are susceptible toLeishmania donovani but have diminished hepatic immunopathology. European Journal of Immunology. 30(3). 834–839. 53 indexed citations
7.
Satoskar, Abhay R., et al.. (2000). IL-12 gene-deficient C57BL / 6 mice are susceptible to Leishmaniadonovani but have diminished hepatic immunopathology. European Journal of Immunology. 30(3). 834–839. 4 indexed citations
8.
Takeda, Kohsuke, et al.. (2000). Genetically Resistant Mice Lacking IL-18 Gene Develop Th1 Response and Control Cutaneous Leishmania major Infection. The Journal of Immunology. 164(11). 5890–5893. 73 indexed citations
9.
Stamm, Luisa M., et al.. (1999). STAT-4 mediated IL-12 signaling pathway is critical for the development of protective immunity in cutaneous leishmaniasis. European Journal of Immunology. 29(8). 2524–2529. 57 indexed citations
10.
Stamm, Luisa M., Anne Räisänen‐Sokolowski, Mitsuhiro Okano, et al.. (1998). Mice with STAT6-Targeted Gene Disruption Develop a Th1 Response and Control Cutaneous Leishmaniasis. The Journal of Immunology. 161(11). 6180–6188. 74 indexed citations
11.
David, John R.. (1985). Hyperglucagonaemia and treatment with danazol for systemic lupus erythematosus.. BMJ. 291(6503). 1170–1171. 5 indexed citations
12.
Huang, Paul L., Bryan E. Roberts, D M Pratt, John R. David, & Jacqueline S. Miller. (1984). Structure and Arrangement of the β-Tubulin Genes of Leishmania tropica. Molecular and Cellular Biology. 4(7). 1372–1383. 9 indexed citations
13.
Bloom, Barry R. & John R. David. (1976). In vitro methods in cell-mediated and tumor immunity. Academic Press eBooks. 1152 indexed citations breakdown →
14.
Tadakuma, Takushi, et al.. (1976). Biological expressions of lymphocyte activation. V. Characterization of a soluble immune response suppressor (SIRS) produced by concanavalin A-activated spleen cells.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 117(1). 323–30. 71 indexed citations
15.
Daniels, Jerry C., Kenneth S. Warren, & John R. David. (1975). Studies on the mechanism of suppression of delayed hypersensitivity by the antischistosomal compund niridazole.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 115(5). 1414–21. 25 indexed citations
16.
Stubbs, Marion, et al.. (1973). METABOLIC AND FUNCTIONAL STUDIES ON ACTIVATED MOUSE MACROPHAGES. The Journal of Experimental Medicine. 137(2). 537–542. 70 indexed citations
17.
David, John R., et al.. (1967). Satire : a critical anthology.
18.
David, John R., Salah Al‐Askari, H. Sherwood Lawrence, & L. Thomas. (1964). Delayed Hypersensitivity ire vitro. I. The Specificity of Inhibition of Cell Migration by Antigens. II. Effect of Sensitive Cells on Normal Cells in the Presence of Antigen.. Immunology. 93(2). 1 indexed citations
19.
David, John R., Salah Al‐Askari, H. Sherwood Lawrence, & L. Thomas. (1964). Delayed Hypersensitivity ire vitro. I. The Specificity of Inhibition of Cell Migration by Antigens. II. Effect of Sensitive Cells on Normal Cells in the Presence of Antigen. III. The Specificity of Hapten-Protein Conjugates in the Inhibition of Cell Migration.. Immunology. 93(2).
20.
David, John R., Salah Al‐Askari, H. Sherwood Lawrence, & L. Thomas. (1964). Delayed Hypersensitivity in vitro. I. The Specificity of Inhibition of Cell Migration by Antigens.. Immunology. 93(2). 387 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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