John P. Boyle

3.0k total citations
63 papers, 2.4k citations indexed

About

John P. Boyle is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, John P. Boyle has authored 63 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 29 papers in Physiology and 20 papers in Cellular and Molecular Neuroscience. Recurrent topics in John P. Boyle's work include Alzheimer's disease research and treatments (17 papers), Neuroscience and Neuropharmacology Research (16 papers) and Ion channel regulation and function (16 papers). John P. Boyle is often cited by papers focused on Alzheimer's disease research and treatments (17 papers), Neuroscience and Neuropharmacology Research (16 papers) and Ion channel regulation and function (16 papers). John P. Boyle collaborates with scholars based in United Kingdom, United States and Canada. John P. Boyle's co-authors include Chris Peers, Hugh A. Pearson, Jason L. Scragg, Mark Dallas, Leigh D. Plant, Ian F. Smith, Hannah E. Boycott, Nishani T. Hettiarachchi, Moza M. Al‐Owais and Claudia Bauer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

John P. Boyle

62 papers receiving 2.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John P. Boyle United Kingdom 29 1.2k 1.0k 536 282 260 63 2.4k
Andrzej Szutowicz Poland 28 1.0k 0.9× 734 0.7× 603 1.1× 265 0.9× 190 0.7× 124 2.6k
Ana I. Duarte Portugal 31 1.2k 1.1× 1.1k 1.0× 567 1.1× 104 0.4× 363 1.4× 59 3.0k
Nariman Panahian United States 20 1.3k 1.1× 1.0k 1.0× 612 1.1× 192 0.7× 714 2.7× 24 3.0k
Ana Patricia Fernández Spain 30 1.3k 1.1× 1.3k 1.3× 1.2k 2.2× 190 0.7× 770 3.0× 72 3.9k
Swapnil K. Sonkusare United States 25 1.1k 0.9× 986 0.9× 425 0.8× 96 0.3× 200 0.8× 65 2.7k
Mireia Coma Spain 22 921 0.8× 1.4k 1.3× 505 0.9× 74 0.3× 416 1.6× 39 2.7k
Prasad V. G. Katakam United States 35 1.1k 0.9× 970 0.9× 307 0.6× 294 1.0× 336 1.3× 96 2.8k
Sandra Moreno Italy 35 1.9k 1.6× 1.0k 1.0× 845 1.6× 275 1.0× 436 1.7× 80 3.6k
Helena Parfenova United States 33 1.7k 1.4× 666 0.6× 310 0.6× 529 1.9× 328 1.3× 84 3.2k
Cora O’Neill Ireland 28 1.6k 1.4× 1.5k 1.5× 958 1.8× 118 0.4× 366 1.4× 65 3.2k

Countries citing papers authored by John P. Boyle

Since Specialization
Citations

This map shows the geographic impact of John P. Boyle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John P. Boyle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John P. Boyle more than expected).

Fields of papers citing papers by John P. Boyle

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John P. Boyle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John P. Boyle. The network helps show where John P. Boyle may publish in the future.

Co-authorship network of co-authors of John P. Boyle

This figure shows the co-authorship network connecting the top 25 collaborators of John P. Boyle. A scholar is included among the top collaborators of John P. Boyle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John P. Boyle. John P. Boyle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Dallas, Mark, Moza M. Al‐Owais, Nishani T. Hettiarachchi, et al.. (2021). Hydrogen sulfide regulates hippocampal neuron excitability via S-sulfhydration of Kv2.1. Scientific Reports. 11(1). 8194–8194. 14 indexed citations
2.
Daly, Catherine, et al.. (2019). Permeabilised skeletal muscle reveals mitochondrial deficiency in malignant hyperthermia-susceptible individuals. British Journal of Anaesthesia. 122(5). 613–621. 9 indexed citations
3.
Wang, Dongliang, et al.. (2018). Trends in sociodemographic disparities of pediatric cochlear implantation over a 15-year period. International Journal of Pediatric Otorhinolaryngology. 115. 165–170. 23 indexed citations
4.
Hettiarachchi, Nishani T., John P. Boyle, Mark Dallas, et al.. (2017). Heme oxygenase-1 derived carbon monoxide suppresses Aβ1–42 toxicity in astrocytes. Cell Death and Disease. 8(6). e2884–e2884. 35 indexed citations
5.
Al‐Owais, Moza M., Nishani T. Hettiarachchi, John P. Boyle, et al.. (2017). Multiple mechanisms mediating carbon monoxide inhibition of the voltage-gated K+ channel Kv1.5. Cell Death and Disease. 8(11). e3163–e3163. 13 indexed citations
6.
Elíes, Jacobo, Jason L. Scragg, Mark Dallas, et al.. (2015). Inhibition of T-type Ca2+ Channels by Hydrogen Sulfide. Advances in experimental medicine and biology. 860. 353–360. 5 indexed citations
7.
Al‐Owais, Moza M., Mark Dallas, John P. Boyle, Jason L. Scragg, & Chris Peers. (2015). Heme Oxygenase-1 Influences Apoptosis via CO-mediated Inhibition of K+ Channels. Advances in experimental medicine and biology. 860. 343–351. 9 indexed citations
8.
Elíes, Jacobo, Emily L. Johnson, John P. Boyle, Jason L. Scragg, & Chris Peers. (2015). H2S does not regulate proliferation via T-type Ca2+ channels. Biochemical and Biophysical Research Communications. 461(4). 659–664. 5 indexed citations
9.
Duckles, Hayley, Hannah E. Boycott, Moza M. Al‐Owais, et al.. (2014). Heme oxygenase-1 regulates cell proliferation via carbon monoxide-mediated inhibition of T-type Ca2+ channels. Pflügers Archiv - European Journal of Physiology. 467(2). 415–427. 24 indexed citations
10.
Linley, John E., et al.. (2012). Reactive oxygen species are second messengers of neurokinin signaling in peripheral sensory neurons. Proceedings of the National Academy of Sciences. 109(24). E1578–86. 81 indexed citations
11.
Dallas, Mark, Zhaokang Yang, John P. Boyle, et al.. (2012). Carbon Monoxide Induces Cardiac Arrhythmia via Induction of the Late Na+ Current. American Journal of Respiratory and Critical Care Medicine. 186(7). 648–656. 66 indexed citations
12.
Al‐Owais, Moza M., Jason L. Scragg, Mark Dallas, et al.. (2012). Carbon Monoxide Mediates the Anti-apoptotic Effects of Heme Oxygenase-1 in Medulloblastoma DAOY Cells via K+ Channel Inhibition. Journal of Biological Chemistry. 287(29). 24754–24764. 55 indexed citations
13.
Boyle, John P., Nishani T. Hettiarachchi, Hugh A. Pearson, et al.. (2012). Cellular consequences of the expression of Alzheimer's disease-causing presenilin 1 mutations in human neuroblastoma (SH-SY5Y) cells. Brain Research. 1443. 75–88. 8 indexed citations
14.
Peers, Chris, Claudia Bauer, John P. Boyle, Jason L. Scragg, & Mark Dallas. (2011). Modulation of Ion Channels by Hydrogen Sulfide. Antioxidants and Redox Signaling. 17(1). 95–105. 83 indexed citations
15.
Hettiarachchi, Nishani T., Mark Dallas, Hugh A. Pearson, et al.. (2010). Gap junction-mediated spontaneous Ca2+ waves in differentiated cholinergic SN56 cells. Biochemical and Biophysical Research Communications. 397(3). 564–568. 2 indexed citations
16.
Peers, Chris, Mark Dallas, Hannah E. Boycott, et al.. (2009). Hypoxia and Neurodegeneration. Annals of the New York Academy of Sciences. 1177(1). 169–177. 136 indexed citations
17.
Bauer, Claudia, John P. Boyle, Karen E. Porter, & Chris Peers. (2009). Modulation of Ca2+ signalling in human vascular endothelial cells by hydrogen sulfide. Atherosclerosis. 209(2). 374–380. 41 indexed citations
18.
Boycott, Hannah E., Mark Dallas, John P. Boyle, Hugh A. Pearson, & Chris Peers. (2007). Hypoxia suppresses astrocyte glutamate transport independently of amyloid formation. Biochemical and Biophysical Research Communications. 364(1). 100–104. 24 indexed citations
19.
Plant, Leigh D., Nicola J. Webster, John P. Boyle, et al.. (2005). Amyloid β peptide as a physiological modulator of neuronal ‘A’-type K+ current. Neurobiology of Aging. 27(11). 1673–1683. 85 indexed citations
20.
Peers, Chris, Ian F. Smith, John P. Boyle, & Hugh A. Pearson. (2004). Remodelling of Ca2+ homeostasis in type I cortical astrocytes by hypoxia: evidence for association with Alzheimer's disease. Biological Chemistry. 385(3-4). 285–9. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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