John M. Quayle

6.3k total citations · 3 hit papers
37 papers, 5.4k citations indexed

About

John M. Quayle is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, John M. Quayle has authored 37 papers receiving a total of 5.4k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 15 papers in Cardiology and Cardiovascular Medicine and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in John M. Quayle's work include Ion channel regulation and function (24 papers), Cardiac electrophysiology and arrhythmias (14 papers) and Neuroscience and Neuropharmacology Research (11 papers). John M. Quayle is often cited by papers focused on Ion channel regulation and function (24 papers), Cardiac electrophysiology and arrhythmias (14 papers) and Neuroscience and Neuropharmacology Research (11 papers). John M. Quayle collaborates with scholars based in United Kingdom, United States and Netherlands. John M. Quayle's co-authors include Mark T. Nelson, N. B. Standen, Joseph E. Brayden, Nicholas B. Standen, Yü Huang, Noel W. Davies, Tomoko Kamishima, John G. McCarron, Caroline Dart and Adrian D. Bonev and has published in prestigious journals such as Science, Journal of Biological Chemistry and Physiological Reviews.

In The Last Decade

John M. Quayle

37 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Physiological roles and properties of potassium channels in arterial smooth muscle

1995 1.9k citations Mark T. Nelson, John M. Quayle American Journal of Physiology-Cell Physiology profile →
Hyperpolarizing Vasodilators Activate ATP-sensitive K ⁺ Channels in Arterial Smooth Muscle

1989 1.0k citations Nicholas B. Standen, John M. Quayle et al. profile →
ATP-sensitive and inwardly rectifying potassium channels in smooth muscle

1997 667 citations John M. Quayle, Mark T. Nelson et al. profile →

Peers

John M. Quayle

PHYSI

CCM

PFM

CMN

Junhui Sun United States

Joseph E. Brayden United States

Nicholas B. Standen United Kingdom

S. Jamal Mustafa United States

T. B. Bolton United Kingdom

Nancy J. Rusch United States

Norio Taira Japan

A.H. Weston United Kingdom

Jonathan H. Jaggar United States

Fangyi Zhang United States

Junhui Sun United States

John M. Quayle

3.4k ×1.1

2.0k ×1.0

PHYSI

1.2k ×0.7

CCM

925 ×0.6

PFM

450 ×0.3

CMN

Citations per year, relative to John M. Quayle John M. Quayle (= 1×) peers Junhui Sun

Countries citing papers authored by John M. Quayle

Since Specialization

Citations

This map shows the geographic impact of John M. Quayle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John M. Quayle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John M. Quayle more than expected).

Fields of papers citing papers by John M. Quayle

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John M. Quayle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John M. Quayle. The network helps show where John M. Quayle may publish in the future.

Co-authorship network of co-authors of John M. Quayle

This figure shows the co-authorship network connecting the top 25 collaborators of John M. Quayle. A scholar is included among the top collaborators of John M. Quayle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John M. Quayle. John M. Quayle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Yang, Mingming, Amy E. Chadwick, Caroline Dart, Tomoko Kamishima, & John M. Quayle. (2017). Bioenergetic profile of human coronary artery smooth muscle cells and effect of metabolic intervention. PLoS ONE. 12(5). e0177951–e0177951. 26 indexed citations

Prendergast, C, John M. Quayle, Theodor Burdyga, & Susan Wray. (2014). Atherosclerosis differentially affects calcium signalling in endothelial cells from aortic arch and thoracic aorta in Apolipoprotein E knockout mice. Physiological Reports. 2(10). e12171–e12171. 13 indexed citations

Kamishima, Tomoko, et al.. (2013). Exchange protein activated by cAMP (Epac) induces vascular relaxation by activating Ca²⁺‐sensitive K⁺ channels in rat mesenteric artery. The Journal of Physiology. 591(20). 5107–5123. 40 indexed citations

Quayle, John M., et al.. (2010). TREK‐1 K⁺Channels in the Cardiovascular System: Their Significance and Potential as a Therapeutic Target. Cardiovascular Therapeutics. 30(1). e23–9. 39 indexed citations

Prendergast, C, John M. Quayle, Theodor Burdyga, & Susan Wray. (2009). Cholesterol depletion alters coronary artery myocyte Ca2+ signalling in a stimulus-specific manner. Cell Calcium. 47(1). 84–91. 23 indexed citations

Kamishima, Tomoko, et al.. (2009). Exchange protein activated by cAMP (Epac) mediates cAMP‐dependent but protein kinase A‐insensitive modulation of vascular ATP‐sensitive potassium channels. The Journal of Physiology. 587(14). 3639–3650. 54 indexed citations

Kamishima, Tomoko, Theodor Burdyga, J.A. Gallagher, & John M. Quayle. (2007). Caveolin-1 and caveolin-3 regulate Ca²⁺homeostasis of single smooth muscle cells from rat cerebral resistance arteries. American Journal of Physiology-Heart and Circulatory Physiology. 293(1). H204–H214. 20 indexed citations

Wlodarski, B, Brian Foster, Katherine M. Buckley, et al.. (2005). Human Keratinocytes Release ATP and Utilize Three Mechanisms for Nucleotide Interconversion at the Cell Surface. Journal of Biological Chemistry. 280(33). 29667–29676. 105 indexed citations

Quayle, John M., et al.. (2004). Molecular analysis of the subtype‐selective inhibition of cloned K_ATP channels by PNU‐37883A. British Journal of Pharmacology. 141(5). 867–873. 27 indexed citations

10.

Wellman, George C., et al.. (1999). Inhibition of vascular K_ATP channels by U‐37883A: a comparison with cardiac and skeletal muscle. British Journal of Pharmacology. 128(4). 909–916. 26 indexed citations

11.

Wellman, George C., John M. Quayle, & N. B. Standen. (1998). ATP‐sensitive K⁺ channel activation by calcitonin gene‐related peptide and protein kinase A in pig coronary arterial smooth muscle. The Journal of Physiology. 507(1). 117–129. 150 indexed citations

12.

Stoop, Ron & John M. Quayle. (1998). Fading and rebound of P2X₂ currents at millimolar ATP concentrations caused by low pH. British Journal of Pharmacology. 125(2). 235–237. 11 indexed citations

13.

Kubo, Masahiro, John M. Quayle, & N. B. Standen. (1997). Angiotensin II Inhibition of ATP‐Sensitive K⁺ Currents in Rat Arterial Smooth Muscle Cells Through Protein Kinase C. The Journal of Physiology. 503(3). 489–496. 71 indexed citations

14.

Quayle, John M., Caroline Dart, & N. B. Standen. (1996). The properties and distribution of inward rectifier potassium currents in pig coronary arterial smooth muscle.. The Journal of Physiology. 494(3). 715–726. 125 indexed citations

15.

Wellman, George C., John M. Quayle, & N. B. Standen. (1996). Evidence against the association of the sulphonylurea receptor with endogenous Kir family members other than KATP in coronary vascular smooth muscle. Pflügers Archiv - European Journal of Physiology. 432(2). 355–357. 11 indexed citations

16.

Quayle, John M. & N. B. Standen. (1994). KATP channels in vascular smooth muscle. Cardiovascular Research. 28(6). 797–804. 112 indexed citations

17.

Quayle, John M., John G. McCarron, Joseph E. Brayden, & Mark T. Nelson. (1993). Inward rectifier K+ currents in smooth muscle cells from rat resistance-sized cerebral arteries. American Journal of Physiology-Cell Physiology. 265(5). C1363–C1370. 221 indexed citations

18.

Quayle, John M., et al.. (1993). Single calcium channels in resistance-sized cerebral arteries from rats. American Journal of Physiology-Heart and Circulatory Physiology. 264(2). H470–H478. 26 indexed citations

19.

Brayden, Joseph E., John M. Quayle, Nicholas B. Standen, & Mark T. Nelson. (1991). Role of Potassium Channels in the Vascular Response to Endogenous and Pharmacological Vasodilators. Journal of Vascular Research. 28(1-3). 147–153. 57 indexed citations

20.

Huang, Yü, John M. Quayle, Jennings F. Worley, N. B. Standen, & Mark T. Nelson. (1989). External cadmium and internal calcium block of single calcium channels in smooth muscle cells from rabbit mesenteric artery. Biophysical Journal. 56(5). 1023–1028. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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